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51篇 您的检索式:作者名="Diep N"
    题名 作者 年代 出处 被引量
1Expression of cell cycle proteins in blood vessels of angiotensin Ⅱ-infused rats:role of AT(1) receptors显示文摘DIEP Q N EL-MABROUK M TOUYZ R M 2001Hypertension2001,37,22:1
2Docosahexaenoic acid,a peraxisome proliferator-activated receptor-alpha ligand,induces apoptosis in vascular smooth muscle cells by stimulation of p38 mitogen-activated protein kinase显示文摘Diep Q N Touyz R M Schiffrin E L 2000Hypertension2000,,36:1
3Longterm effects of Pioglitazone on cardiac hypertrophy,inflammation and fibrosis in strokeprone spontaneously hypertensive rats显示文摘Diep Q N Benkirane K Schiffrin EL 2002Hypertenstion2002,40,:1
4Efect of AT( 1 ) receptor blockade on cardiac apoptosis in angiotensin Ⅱ-induced hypertension显示文摘DIEP Q N EI MABROUK M YUE P 2002Am J Physiol Heart Circ Physiol2002,282,5:1
5Effect of AT(1) recep tor blockade on cardiac apoptosis in angiotensin Ⅱ-induced hypertension显示文摘Diep Q N Elmabrouk M Yue P 2002American Journal of Physiology-Heart & Circula tory Physiology2002,282,5:1
6Characterisation of the Wnt antagonists and their response to conditionally activated Wnt signalling in the developing mouse forebrain 显示文摘DIEP D B HOEN N BACKMAN M 2004Brain Res Dev BrainRes2004,153,2:1
7Structure,endothelial function,cell growth,and inflammation in blood vessels of angiotensin Ⅱ-infused rats:role of peroxisome proliferator activated receptor-gamma显示文摘DIEP Q N El MABROUK M COHN J S 2002Circulation2002,105,19:1
8PPAR alpha activator fenofibrate inhibits myocardial inflammation and fibrosis in angiotensin Ⅱ infused rats显示文摘DIEP Q N BENKIRANE K AMIRI F 2004J Mol Cell Cardiol2004,36,:1
9Morbidity associated with primary HIFU and redo-HIFU for the treatment of localized prostate cancer显示文摘Berge V Dickinson L McCartan N Hindley RG Diep LM Emberton M Ahmed HU 2013The Journal of Urology2013,,:1
10Isolation of Pseudomonas stutzeri in wastewater of catfish fish-ponds in the Mekong Delta and its application for wastewater treatment 显示文摘Diep C N Cam P M Vung N H 2009Bioresource Technol2009,100,:1
11Structure, endothelial function, cell growth and inflammation in blood vessels of angiotensin II -infused rats: role of peroxisome proliferator -activated receptor- gamma显示文摘Diep Q N El Mabrouk M Cohn JS 2002Circulation2002,105,19:1
12Characterisation of the Wnt antagonists and their response to conditionally activated Wnt signalling in the developing mouse forebrain显示文摘Diep DB Hoen N Backman M 2004Brain Res Dev Brain Res2004,153,:1
13Characterisation of the Wnt antagonists and their response to conditionally activated Wnt signalling in the developing mouse forebrain显示文摘Diep DB Hoen N Backman M 2004Brain Res Dev Brain Res2004,153,2:1
14Estimation of the potential of rice straw for ethanol production and the optimum faeility size for different regions ha Vietnam 显示文摘Diep N Q 2012Applied Energy2012,93,:1
15Increased expression of peroxisome proliferator- activated receptor- alpha and - gamma in blood 显示文摘Diep Q N Schiffrin E L 2001Hypertension2001,38,2:1
16Widespread skin and soft- tissue infections due to two methicillin- resistant Staphylococcus aureus strains harboring the genes for panton- valentine leucocidin 显示文摘Diep B A Sensabaugh G F Sombcona N S 2004J Clin Microbiol2004,42,5:1
17Can short-term fasting protect against doxorubicin-induced cardiotoxicity?显示文摘Doxorubicin(Dox) is one of the most effective chemotherapeutic agents used in the treatment of several types of cancer. However the use is limited by cardiotoxicity. Despite extensive investigation into the mechanisms of toxicity and preventative strategies, Dox-induced cardiotoxicity still remains a major cause of morbidity and mortality in cancer survivors. Thus, continued research into preventative strategies is vital. Short-term fasting has proven to be cardioprotective against a variety of insults. Despite the potential, only a few studies have been conducted investigating its ability to prevent Dox-induced cardiotoxicity. However, all show proof-of-principle that short-term fasting is cardioprotective against Dox. Fasting affects a plethora of cellular processes making it difficult to discern the mechanism(s) translating fasting to cardioprotection, but may involve suppression of insulin and insulin-like growth factor-1 signaling with stimulated autophagy. It is likely that additional mechanisms also contribute. Importantly, the literature suggests that fasting may enhance the antitumor activity of Dox. Thus, fasting is a regimen that warrants further investigation as a potential strategy to prevent Dox-induced cardiotoxicity. Future research should aim to determine the optimal regimen of fasting, confirmation that this regimen does not interfere with the antitumor properties of Dox, as well as the underlying mechanisms exerting the cardioprotective effects.Amie J Dirks-Naylor Samir A Kouzi Sendra Yang Ngan TK Tran Joseph D Bero Raean Mabolo Diep T Phan Stephanie D Whitt Heather N Taylor 2014World Journal of Biological Chemistry2014,5,3:1
18Structure endothelial function cell growth and inflammation in blood vessel of Ang-Ⅱ -infused rats:Role of peroxisome proliferator-ac tivated receptor-γ 显示文摘Diep QN M abrouk ME Coh n JS 2002Circulation2002,105,:1
19Effect of AT( 1 ) receptor blockade on cardiac apoptosis in angiutensin I1 -induced hype,len- sion 显示文摘Diep Q N EI Mabrouk M Yue P 2002Am J Physiol Heal1 Circ Physiol2002,282,5:1
20Advances in the profiling of DNA modifications: cytosine methylation and beyond显示文摘Plongthongkum N Diep DH Zhang K 2014Nat Rev Genet2014,15,10:1
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