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| 1 | Transcription-coupled nucleotide excision repair in mammalian cells: molecular mechanisms and biological effects显示文摘有脱氧核糖核酸损害的 RNA II (RNAPIIo ) 有的伸长的相遇为这个事件为 P53 依赖的 apoptosis 提供一个强壮的信号的房间和房间的严重后果骑车拘捕。为了抵抗,延长了抄写的阻塞,房间由联合抄写的修理(TC-NER ) 移开堵住 RNAPIIo 脱氧核糖核酸损害,核苷酸切除修理(NER ) 的一条专业化潜水艇小径。到 UVB 光或化学药品的老鼠的暴露阐明了那 TC-NER 是免于 genotoxic 暴露的尖锐有毒、长期的效果(癌症) 的一条批评幸存小径。在 TC-NER 的缺乏在产生稀罕人的混乱安乐乡症候群(CS ) 的 CSA 和 CSB 基因与变化被联系。最近的数据建议 CSA 和 CSB 在哺乳动物的 TC-NER 起微分作用:是的 CSB 联合因素吸引 NER 蛋白质,染色质 remodellers 和对阻止的 RNAPIIo 复杂的 CSA- E3-ubiquitin 连接酶的一个修理。CSA 为 NER 蛋白质的吸引力是非必需的,还在有 CSB 的合作被要求到成员 XAB2, nucleosomal 绑定蛋白质 HMGN1 和 TFIIS。TC-NER 的新兴的图画是复杂的:没有脱氧核糖核酸拖延损坏的 RNAPIIo 的排水量,堵住抄写的损害的修理发生,并且要求至少二个必要集会因素(CSA 和 CSB ) ,核心 NER 因素(除了 XPC-RAD23B ) ,并且 TC-NER 特定的因素。这些和还未辩别出的蛋白质将完成堵住抄写的损害的有效修理不仅,但是也是可能的贡献脱氧核糖核酸损坏发信号事件。 | Mafia Fousteri Leon HF Mullenders | 2008 | Cell Research2008，18，1： | 10 |
| 2 | Nasal cardiac myosinpeptide treatment and ox40 blockade protect mice fromacute and chronic virally-induced myocarditis 显示文摘 | Fousteri G Dave A Morin B | 2011 | JAutoimmun2011，36，34： | 1 |
| 3 | Penetration of colistin into cerebro- spinal fluid 显示文摘 | Markmltonis SL Markou N Fousteri M | 2009 | Antimicrob Agents Che- mother2009，53，11： | 1 |
| 4 | Excretion of ropivacaine in breast milk during patient-controlled epidural analgesia after cesarean delivery显示文摘 | Matsota P K Markantonis S L Fousteri M Z | 2009 | Reg Anesth Pain Med2009，34，2： | 1 |
| 5 | Water- promoted iodocyclisation of 2 - allylphenols 显示文摘 | Fousteris M Chevrin C Bras J L | 2006 | Green Chemistry2006，8，： | 1 |
| 6 | Optimization of the allylic oxidation in the synthesis of 7-keto-△5-steroidal substrates显示文摘 | ARSENOU E S KOUTSOUREA A I FOUSTERIS M A | 2003 | Steroids2003，68，： | 1 |
| 7 | Excretion of ropiaeaine in breast milk during patient-continUed epidural analgesia after cesarean delivery 显示文摘 | Matsota P K Markantonis S L Fousteri M Z | 2009 | Reg Anesth Pain Med2009，34，2： | 1 |
| 8 | Synthetic approaches for the synthesis of a cytostatic steroidal B - D bilactam 显示文摘 | Koutsourea A I Arsenou E S Fousteris M A | 2003 | Steroids2003，68，： | 1 |
| 9 | Nasal cardiac myosin peptide treatment and OX40 blockade protect mice from acute and chron- ic virally-induced myocarditis 显示文摘 | Fousteri G Dave A Morin B | 2011 | J Autoimmun2011，36，34： | 1 |
| 10 | Toll/interleukin-1 receptor member ST2 exhibits higher soluble levels in type 2 diabe- tes,especially when accompanied with left ventricular diastolic dys- function 显示文摘 | Fousteris E Melidonis A Panoutsopoulos G | 2011 | Cardiovasc Diabeto12011，10，： | 1 |
| 11 | Head-to-head comparison of 2 inflammatory biomarkers for the long-term prediction of left ventricular diastolic dysfunction in type 2 diabetes patients: soluble ST2 versus hs-CRP 显示文摘 | Fousteris E Theodosis-Georgilas A Chantanis S | 2014 | Int J Cardiol2014，174，3： | 1 |
| 12 | Toll/interleukin-1 receptor member ST2 exhibits higher soluble levels in type 2diabetes,especially when accompanied with left ventricular diastolic dysfunction显示文摘 | Fousteris E Melidonis A Panoutsopoulos G | 2011 | Cardiovasc Diabetol2011，10，： | 1 |
| 13 | Synergistic cytogenetic and antineoplastic effects by the combined action of esteric steroidal derivatives of nitrogen mustards 显示文摘 | Constantinos Mourelatos Sotiris Nikolaropoulos Manolis Fousteris | 2012 | Genetic Testing and Molecular Biomarkers2012，16，6： | 1 |
| 14 | Penetration of colistin in- to cerebrospinal fluid 显示文摘 | Markantonis SL Markou N Fousteri M | 2009 | Antimicrob Agents Chemother2009，53，11： | 1 |
| 15 | Excretion of ropivacaine inbreast milk during patient-controlled epidural analgesia after cesarean delivery 显示文摘 | Matsota PK Markantonis SL Fousteri MZ | 2009 | Reg Anesth PainMed2009，34，2： | 1 |
| 16 | Excretion of ropivacaine in breast milk during patient-controlled epidural analgesia after cesarean delivery 显示文摘 | MATSOTA P K MARKANTONIS S L FOUSTERI M Z | 2009 | Reg Anesth Pain Med2009，34，2： | 1 |
| 17 | Penetration of colistin into cerebrospinal fluid 显示文摘 | MARKANTONIS SL MARKOU N FOUSTERI M et ol | 2009 | Antimicrob Agents Chemother2009，53，11： | 1 |
| 18 | PTPN22 and islet-specific autoimmunity:What have the mouse models taught us?显示文摘An allelic variant of the protein tyrosin phosphatase non-receptor 22(PTPN22) gene, PTPN22 R620 W, constitutes the strongest non-HLA genetic risk factor for the development of type 1 diabetes(T1D). A numberstudies using mouse models have addressed how PTPN22 predisposes to T1D. PTPN22 downmodulation, overexpression or expression of the variant gene in genetically manipulated mice has generated controversial results. These discrepancies probably derive from the fact that PTPN22 has differential effects on innate and adaptive immune responses. Moreover, the effects of PTPN22 are dependent on other genetic variables. Here we discuss these findings and try to explain the discrepancies. Exploring the mechanism by which PTPN22 contributes to islet-specific autoimmunity could help us understand its role in T1D pathogenesis and exploit it as a potential therapeutic target to prevent the disease. | Giuseppe Galvani Georgia Fousteri | 2017 | World Journal of Diabetes2017，8，7： | 1 |
| 19 | Synthetic approaches for the synthesis of a cytostatic steroidal B-D bilactam 显示文摘 | Koutsourea AI Arsenou ES Fousteris MA | 2003 | Steroids2003，68，78： | 1 |
| 20 | A novel SMC protein complex in Schizosaccharomyces pombe contains the Rad 18 DNA repair protein 显示文摘 | Fousteri MI Lehmann AR | 2000 | EMBO J2000，19，7： | 1 |
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