TRIM9 short isoform preferentially promotes DNA and RNA virus-induced production of type I interferon by recruiting GSK3β to TBK1

查看全文 作　　者：Yunfei [1]Qin;Qingxiang [1]Liu;Shuo [1]Tian;Weihong [2]Xie;Jun [1,3]Cui;Rong-Fu [4,5,6]Wang

机构地区：[1]Key Laboratory of Gene Engineering of the Ministry of Education, State Key Laboratory of Biocontrol, School of Life Sciences,;[2]Zhongshan School of Medicine, Guangzhou, Guangdong, China;[3]Collaborative Innovation Center of Cancer Medicine, Sun Yat-sen University, Guangzhou, Guangdong, China;[4]Houston Methodist Research Institute, Houston, TX 77030, USA;[5]Department of Microbiology and Immu- nology, Weill Cornell Medical College, Cornell University, New York, NY 10065, USA;[6]Institute of Biosciences and Technology, College of Medicine, Texas A & M University, Houston, TX 77030, USA

出　　处：《Cell Research》索引2016年第26卷第5期,共16页

关键词：RNA病毒病毒DNA 干扰素抗病毒免疫诱导信号转导炎性细胞因子病毒感染

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TRIM9 short isoform preferentially promotes DNA and RNA virus-induced production of type I interferon by recruiting GSK3β to TBK1

参考文献(44)

引证文献(4)

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