SARS-CoV-2 promote autophagy to suppress type I interferon response

查看全文 作　　者：Xianfeng [1]Hui;Linliang [2]Zhang;Lei [1]Cao;Kun [1]Huang;Ya [1]Zhao;Yufei [1]Zhang;Xi [1]Chen;Xian [3]Lin;Mingzhou [2]Chen;Meilin [1]Jin

机构地区：[1]State Key Laboratory of Agricultural Microbiology,Huazhong Agricultural University,Wuhan,China;[2]State Key Laboratory of Virology and Modern Virology Research Center,College of Life Sciences,Wuhan University,Wuhan,China;[3]CAS Key Laboratory of Special Pathogens and Biosafety,Wuhan Institute of Virology,Wuhan,China

出　　处：《Signal Transduction and Targeted Therapy》索引2021年第6卷第6期,共3页

基　　金：Emergency Science and Technology Project of China's Ministry of Science and Technology(2020YFC0845600);Emergency Science and Technology Project of Hubei Province(2020FCA046);the Fundamental Research Funds for the Central University(2042021 kf0049);the Special Fund for COVID-19 Research of Wuhan University,National Key R&D Program of China(2017YFA0505801);the National Natural Science Foundation of China(81825015,81871650,and 31630086);National Science and Technology Major Project(2018ZX10101004);the Natural Science Foundation of Hubei Province Innovation Group(2017CFA022). 摘　　要：Dear Editor,The outbreak of SARS-CoV-2 leads global epidemic with high morbidity and mortality.However,the pathophysiology of this deadly virus is complex and largely unknown.Autophagy is a highly conserved homeostatic process that allows cells to recycle their components.Several studies provided evidence that human coronavirus infections are closely related to various cellular aspects associated with autophagy.1 Autophagy may play a crucial role in the SARS-CoV-2 viral lifecycle. 关键词：MORTALITY INTERFERON MORBIDITY

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SARS-CoV-2 promote autophagy to suppress type I interferon response

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