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| 1 | Predicting freshwater fish distributions using landscape-level variables 显示文摘 | Argent D G Bishop J A Stauffer J R Jr | 2003 | Fisheries Research2003，60，1： | 1 |
| 2 | TGF-βsignaling is activated in patients with chronic HBV infection and repressed by SMAD7 overexpression after successful antiviral treatment显示文摘 | Argentou N Germanidis G Hytiroglou P | 2016 | Inflamm Res2016，65，5： | 1 |
| 3 | The wild bananas of Papua New Guinea 显示文摘 | Argent G C G | 1976 | Notes from the Royal Botanic Garden Edinburgh1976，35，： | 1 |
| 4 | Two interesting wild Musa species (Musaceae) from Sabah, Malaysia 显示文摘 | Argent G | 2000 | Gardens'' bulletin Singapore2000，52，： | 1 |
| 5 | A successful,sustainable and low cost control-programme for bovine hypodermosis in France显示文摘 | BOULARD C ALVINERIE M ARGENTE G | 2008 | Vet Parasitol2008，158，12： | 1 |
| 6 | The diversity of 15 ha of lowland mixed dipterocarp forest, Central Kalimantan 显示文摘 | Wilkie P Argent G Cambell E | 2004 | Biodiversity and Conservation2004，13，： | 1 |
| 7 | Chromosome number of some tropical Rhododendrons (section Vireya)显示文摘 | Atkinson R Jong K Argent G 2000 | 2000 | EDBG J Bot2000，57，1： | 1 |
| 8 | Proton Conduction in aPhosphonate-Based Metal-Organic Framework Mediated by Intrinsic “FreeDiffusion inside a Sphere”显示文摘 | PiliS Argent S P Morris C G | 2016 | +J Am Chem Soc2016，138，20： | 1 |
| 9 | Liver Foxp3 and PD1/PDL1 expression is down - regulated in chronic HBV hepatitis on maintained remission related to the degree of inflammation 显示文摘 | Germanidis G Argentou N Hytiroglou P | 2013 | Front Immunol2013，，4： | 1 |
| 10 | Does adherence to treat- ment targets in children with severe traumatic brain injury avoid brain hypoxia A brain tissue oxygenation study显示文摘 | Figaji A A Fieggen A G Argent A C et aI | 2008 | Neurosurgery2008，63，1： | 1 |
| 11 | Assessment of the validity of the section in Musa (Musaceae) using AFLP显示文摘 | Wong C Kiew R Argent G | 2002 | Ann Bot2002，，90： | 1 |
| 12 | Pediatric intensive care in South Africa: an account of making optimum use of limited resources at the Red Cross War Memorial Children's Hospital显示文摘 | Andrew C Argent Johann Ahrens Brenda M Morrow Louis G Reynolds Mark Hatherill Shamiel Salie Solomon R Benatar | 2014 | Pediatric critical care medicine:a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies2014，15，1： | 1 |
| 13 | Stream fish community response to a gradient of specific conductance 显示文摘 | KIMMEL W G ARGENT D G | 2009 | Water Air & Soil Pollution2009，206，： | 1 |
| 14 | Rapid detection of MYD88-L265P mutation by PCR-RFLP in B-cell lymphoproliferative disorders显示文摘 | Argentou N Vassilopoulos G Ioannou M | 2014 | Leukemia2014，28，2： | 1 |
| 15 | Effect of herpesvirus infection on pancreatic duct cell secretion显示文摘AIM: To examine the effect of acute infection caused by herpesvirus (pseudorabies virus, PRV) on pancreatic ductal secretion.METHODS: The virulent Ba-DupGreen (BDG) and nonvirulent Ka-RREpOlacgfp (KEG) genetically modified strains of PRV were used in this study and both of them contain the gene for green fluorescent protein (GFP). Small intra/interlobular ducts were infected with BDG virus (107 PFU/mL for 6 h) or with KEG virus (1010 PFU/mL for 6 h), while non-infected ducts were incubated only with the culture media. The ducts were then cultured for a further 18 h.The rate of HCO3- secretion [base efflux -J(B-)] was determined from the buffering capacity of the cells and the initial rate of intracellular acidification (1) after sudden blockage of basolateral base loaders with dihydro-4,4,-diisothiocyanatostilbene-2,2,-disulfonic acid (500 μmol/L)and amiloride (200 μmol/L), and (2) after alkali loading the ducts by exposure to NH4Cl. All the experiments were performed in HCO3--buffered Ringer solution at 37 ℃ (n = 5ducts for each experimental condition). Viral structural proteins were visualized by immunohistochemistry. Virallyencoded GFP and immunofluorescence signals were recorded by a confocal laser scanning microscope.RESULTS: The BDG virus infected the majority of accessible cells of the duct as judged by the appearance of GFP and viral antigens in the ductal cells. KEG virus caused a similarly high efficiency of infection. After blockage of basolateral base loaders, BDG infection significantly elevated -J(B-) 24 h after the infection, compared to the non-infected group. However, KEG infection did not modify -J(B-). After alkali loading the ducts, -J(B-) was significantly elevated in the BDG group compared to the control group 24 h after the infection. As we found with the inhibitor stop method, no change was observed in the group KEG compared to the non-infected group.CONCLUSION: Incubation with the BDG or KEG strains of PRV results in an effective infection of ductal epithelial cells. The BDG strain of PRV, which is able to initiate a lytic viral cycle, stimulates HcO3- secretion in guinea pig pancreatic duct by about four- to fivefold, 24 h after the infection. However, the KEG strain of PRV, which can infect,but fails to replicate, has no effect on HCO3- secretion.We suggest that this response of pancreatic ducts to virulent PRV infection may represent a defense mechanism against invasive pathogens to avoid pancreatic injury. | Péter Hegyi Balázs rdg Zoltán Rakonczai Jr Tamás Takács János Lonovics Annamária Szabolcs Réka Sári András Tóth Julius G Papp András Varró Mária K Kovács Mike A Gray Barry E Argent Zsolt Boldogki | 2005 | World Journal of Gastroenterology2005，11，38： | 0 |
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