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26篇 您的检索式:作者名="Baracca A"
    题名 作者 年代 出处 被引量
1Rhodamine 123 as a probe of mitoehondrial membrane potential: Evaluation of proton flux through F(0) during ATP synthesis显示文摘Baracca A Sgarbi G Solaini G 2003Bioehim Biophys Acta2003,1606,13:1
2Mitoehondrial Complex I : structure, function, and implications in neurodegeneration 显示文摘LENAZ G BARACCA A FATO R 2006Ital J Biochem2006,55,34:1
3New insights into structure and function of mitochondria and their role in ageing and disease显示文摘Lenaz G Baracca A Fato R 2006Antioxid Redox Signal2006,8,:1
4Rhodamine 123 as a probe of mitoehondrial membrane potential: evaluation of proton flux through F(0) during ATP synthesis显示文摘Baracca A Sgarbi G Solaini G 2003Eiochim Biophys Acta2003,1606,13:1
5Rhodamine 125 as a probe of mitochondrial membrane potential: evaluation of proton flux through f(O) during ATP synthesis显示文摘Baracca A Sgarbi G Solaini G 2003Bioch Biophys Acta2003,1606,:1
6Rhodamine 123 as a probe of mitochondrial membrane potential: evaluation of proton flux through F (0) during ATP synthesis 显示文摘BARACCA A SGARBI G SOLAINI G 2003Biochim Biophys Acta2003,1606,13:1
7Biochemical phenotypes associated with the mitochondrial ATP 6 gene mutations at nt8993 显示文摘Baracca A Sgarbi G Mattiazzi M 2007Biochem Biophys Acta2007,1767,7:1
8Rhodamine 123 as a probe of mitochondrial membrane potential:evaluation of proton flux through F(0) during ATP synthesis显示文摘BARACCA A SGARBI G SOLAINI G 2003Biochim Biophys Acta2003,1606,:1
9Rhodamine 123 as a probe of mitochondrial membrane potential:evaluation of proton flux through F (O) during ATP synthesis?显示文摘Baracca A Sgarbi G Solaini G 2003Biochim Biophys Acta2003,1606,13:1
10Rhodamine 123 as a probe of mitochondrial membrane potential: evaluation of proton flux through F (0) during ATP synthesis显示文摘Baracca A Sgarbi G Solaini G 2003Biochim Biophya Aeta2003,1606,13:1
11Hypoxia and mi-tochondrial oxidative metabolism 显示文摘Solaini G Baracca A Lenaz G 2010Biochim Biophys Acta2010,1797,67:1
12Mitochondrial complex I: structure, function, and implications in neurodegeneration 显示文摘Lenaz G Baracca A Fato R 2006ItalJ Biochem2006,55,34:1
13inefficient coupling between proton transport and ATP synthesis may be the pathogenic mechanism for NARP and Leigh syndrome resulting from the T8993G mutation in mtDNA显示文摘Sgarbi G Baracca A Lenaz G Valentino LM Carelli V Solaini G 2006Biochem J2006,,:1
14Bioenergetics of mitochondrial diseases associated with mtDNA mutations 显示文摘Lenaz G Baracca A Carelli V D'Aurelio M Sgarbi G Solaini G 2004Biochim Biophys Acta2004,1658,12:1
15New insights intostructure and function of mitochondria and their role in agingand disease显示文摘LENAZ G BARACCA A FATO R 2006Antioxid Redox Signal2006,8,34:1
16Biochemical phenotypes associated with the mitochondrial ATP6 gene mutations at nt8993显示文摘Baracca A Sgarbi G Mattiazzi M 2007Biochim Biophys Acta2007,1767,7:1
17Mitochondrial Complex I : structure, function, and implications in neurodegene ra-tion 显示文摘Lenaz G Baracca A Fato R 2006Ital J Biochem2006,55,34:1
18Rhodamine 123 as a probe of mitochondrial membrane potential:evaluation of proton flux through F(O) during ATP synthesis显示文摘Baracca A Sqarbi G Solaini G 0,,:1
19Mitochondrial Complex I: structure, function, and implications in ncurodegeneration 显示文摘Lenaz G Baracca A Fato R ct al 2006Ital J Biochem2006,55,34:1
20M itochondrial Complex I:structure, function,and implications in neuro-degeneration显示文摘Lenaz G Baracca A Fato R 2006Ital Journal of Biochem2006,55,34:1
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