|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | Enamel microabrasion: An overview of clinical and scientific considerations显示文摘Superficial stains and irregularities of the enamelare generally what prompt patients to seek dental intervention to improve their smile. These stains or defects may be due to hypoplasia, amelogenesis imperfecta, mineralized white spots, or fluorosis, for which enamel microabrasion is primarily indicated. Enamel microabrasion involves the use of acidic and abrasive agents, such as with 37% phosphoric acid and pumice or 6% hydrochloric acid and silica, applied to the altered enamel surface with mechanical pressure from a rubber cup coupled to a rotatory mandrel of a lowrotation micromotor. If necessary, this treatment can be safely combined with bleaching for better esthetic results. Recent studies show that microabrasion is a conservative treatment when the enamel wear is minimal and clinically imperceptible. The most important factor contributing to the success of enamel microabrasion is the depth of the defect, as deeper, opaque stains, such as those resulting from hypoplasia, cannot be resolved with microabrasion, and require a restorative approach. Surface enamel alterations that result from microabrasion, such as roughness and microhardness, are easily restored by saliva. Clinical studies support the efficacy and longevity of this safe and minimally invasive treatment. The present article presents the clinical and scientific aspects concerning the microabrasion technique, and discusses the indications for and effects of the treatment, including recent works describing microscopic and clinical evaluations. | Núbia Inocencya Pavesi Pini Daniel Sundfeld-Neto Flavio Henrique Baggio Aguiar Renato Herman Sundfeld Luis Roberto Marcondes Martins José Roberto Lovadino Débora Alves Nunes Leite Lima | 2015 | World Journal of Clinical Cases2015，3，1： | 6 |
| 2 | Gitelman s variant of Bartter s syndrome, inherited hypokalaemie alkalosis, is caused by mutations in the hiazide-sensitive Na-C1 eotransporter显示文摘 | Simon D B Nelson-Williams C Bia M J | 1996 | Nat Genet1996，12，： | 1 |
| 3 | Development and func- tion of myeloid-derived suppressor cells generated from mouse embryonic and hematopoietic stem cells 显示文摘 | ZI-IOU Z FRENCH D L biA G | 2010 | Stem Cells2010，28，3： | 1 |
| 4 | Gitelman syn- drome revisited an evaluat ion of symptoms and health-related quality of life显示文摘 | Cruz D N S haer A J Bia M J | 2001 | Kidney Int2001，59，： | 1 |
| 5 | Feasibility of a transient elastography technique for in vitro arterial elasticity assessment 显示文摘 | Brum J Balay G Bia D | 2010 | Conf Proc IEEE Eng Med Biol Soc2010，2010，： | 1 |
| 6 | Gitelman's variant of Bartter's syndrome, inherited hypokalemic alkalosis, is caused by mutations in the thiazide-sensitive Na-C1 cotransporter显示文摘 | Simon D B Nelson-Williams C Bia M J | 1996 | Nat Genet1996，12，1： | 1 |
| 7 | Changes in wall viscosity and filtering as determinant of carotid and femoral atherosclerotic plaque vulnerability: theoretical analysis 显示文摘 | Bia D Brum J Pessana F | 2010 | Conf Proc IEEE Eng Med Biol Soc2010，2010，： | 1 |
| 8 | Pulse wave velocity as marker of preclinical arterial disease: reference levels in a uruguayan popula- tion considering wave detection algorithms, path lengths, aging, and blood pressure显示文摘 | Farro I Bia D Zocalo Y | 2012 | Int J Hypertens2012，2012，16： | 1 |
| 9 | Arsenic-bearing phases in SouthAndean volcanic ashes:Implications for As mobility in aquatic en-vironments显示文摘 | Bia G Borgnino L Gaiero D | 2015 | Chemical Geology2015，，393394： | 1 |
| 10 | Non-invasive assessment of atherosclerotic plaques effects on the segment-to-segment human carotid visco-elasticity and filtering显示文摘 | Bia D Pessana F Forcada P | 2010 | Conf Proc IEEE Eng Med Biol Soc2010，20，10： | 1 |
| 11 | Pulse wave velocity as marker of preclinical arterial disease:reference levels in a uruguayan population considering wave detection algorithms,path lengths, aging,and blood pressure显示文摘 | Farro I Bia D Zócalo Y | | 0，，： | 1 |
| 12 | Gitelman's variant of Bartter's syndrome, inherited hypokalaemic alkalosis,is caused by mutations in the thiazide sensitive Na-Cl cotransporter显示文摘 | Simon D B Nelson-Williams C Bia M J | 1996 | Nat Genet1996，12，1： | 1 |
| 13 | Effect of Deformation Parameters on the Non-Reerystallization Temperature in Nb-Bearing Steels 显示文摘 | Bia D Q | 1993 | Metallurgical Transactions A1993，24，： | 1 |
| 14 | β - adrenergic control of extrarenal potassium disposal; A β 2- adrenergic phenomenon显示文摘 | Lu D Tyler K | 1986 | Nephron1986，43，： | 1 |
| 15 | Gitelman's variant of Bartter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-C1 cotransporter显示文摘 | Simon DB Nelson-Wilhams C Bia M J E11ison D Karet FE Molina AM | 1996 | Nat Genct1996，12，1： | 1 |
您所在的机构暂未开通此部分的功能，请联系管理员