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| 1 | Resveratrol prevents ammonia toxicity in astroglial cells显示文摘 | Bobermin LD Quincozes-Santos A Guerra MC | 2012 | PLoS One2012，7，52： | 1 |
| 2 | Lipoic acid increases glutamate uptake, glutamine synthetase activity and glutathione content in C6 astrocyte cell line显示文摘 | Kleinkauf-Rocha J Bobermin LD Machado PM | 2013 | Int J Dev Neurosci2013，31，3： | 1 |
| 3 | Secretion of S100B, an astrocyte - derived neurotrophic protein, is stimulated by flu- oxetine via a mechanism independent of serotonin 显示文摘 | Tramontina A C Tramontina F Bobermin L D | 2008 | Prog Neuropsy- chopharmacol Biol Psychiatry2008，32，6： | 1 |
| 4 | Secretion of S100B, an astrocyte-derived neurotrophic protein, is stimulated by fluoxetine via a mechanism independent of serotonin显示文摘 | Tranmntina AC Tramontina F Bobermin LD | 2008 | Prog Nan- ropsychopharmacol Biol Psychiatry2008，32，6： | 1 |
| 5 | Atypical neuroleptic risperidone modulates glial functions in C6 astroglial cells显示文摘 | André Quincozes-Santos Larissa Daniele Bobermin Juliana Kleinkauf-Rocha Diogo Onofre Souza Rudimar Riesgo Carlos-Alberto Gon?alves Carmem Gottfried | 2008 | Progress in Neuropsychopharmacology & Biological Psychiatry2008，，1： | 1 |
| 6 | Lipoic acid and N-acetylcysteine prevent ammonia-induced inflammatory response in C6 astroglial cells: The putative role of ERK and HO1 signaling pathways 显示文摘 | SANTOS C L BOBERMIN L D SOUZA D G | 2015 | Toxicology in Vitro2015，29，7： | 1 |
| 7 | Secre- tion of S100B, an astrocyte--derived neurotrophic protein, is stimulated by fluoxetine via a mechanism independent of sero- tonin显示文摘 | Tramontina AC Tramontina F Bobermin LD | 2008 | Prog Neuropsychopharmacol Biol Psychiatry2008，32，6： | 1 |
| 8 | Secre tion of S100B, an astrocyte-derived neurotrophic protein, is stimulated by fluoxetine via a mechanism independent of sero- tonin 显示文摘 | Tramontina AC Tramontina F Bobermin LD | 2008 | Prog Neuropsychopharmacol Biol Psychiatry2008，32，6： | 1 |
| 9 | Lipoic acid increases glutamate uptake, glutamine synthetase activity and glutathiane content in C6 astrocyte cell line显示文摘 | Kleinkauf-Rocha J Bobermin LD Machado Pde M | 2013 | Int J Dev Neuresci2013，31，3： | 1 |
| 10 | Resveratml protects C6 astrocyte cell line against hydrogen peroxide-induced ox- idative stress through heine oxygenase 1显示文摘 | Quincozes-Santos A Bobermin LD Latini A | 2013 | PLoS One2013，8，64： | 1 |
| 11 | Oxida- tive stress mediated by NMDA, AMPA/KA channels in acute hipp- ocampal slices : neuroprotective effect of resveratrol显示文摘 | Quineozes-Santos A Bobermin LD Tramontina AC | 2014 | Toxicol in Vitro2014，28，： | 1 |
| 12 | Gliopreventive effects of guanosine against glucose deprivation in vitro显示文摘 | André Quincozes-Santos Larissa Daniele Bobermin Débora Guerini Souza Bruna Bellaver Carlos-Alberto Gon?alves Diogo Onofre Souza | 2013 | Purinergic Signalling2013，，4： | 1 |
| 13 | Impact of SARS-CoV-2 infection during pregnancy on postnatal brain development:The potential role of glial cells显示文摘Glial cells are crucial for maintaining central nervous system(CNS)homeostasis.They actively participate in immune responses,as well as form functional barriers,such as blood-brain barrier(BBB),which restrict the entry of pathogens and inflammatory mediators into the CNS.In general,viral infections during the gestational period can alter the embryonic and fetal environment,and the related inflammatory response may affect neurodevelopment and lead to behavioral dysfunction during later stage of life,as highlighted by our group for Zika virus infection.Severe acute respiratory syndrome coronavirus-2(SARS-CoV-2)induces a cytokine storm and,during pregnancy,may be related to a more severe form of the coronavirus disease-19(COVID-19)and also to higher preterm birth rates.SARS-CoV-2 can also affect the CNS by inducing neurochemical remodeling in neural cells,which can compromise neuronal plasticity and synaptic function.However,the impact of SARS-CoV-2 infection during pregnancy on postnatal CNS,including brain development during childhood and adulthood,remains undetermined.Our group has recently highlighted the impact of COVID-19 on the expression of molecular markers associated with neuropsychiatric disorders,which are strongly related to the inflammatory response.Thus,based on these relationships,we discussed the impact of SARS-CoV-2 infection either during pregnancy or in critical periods of neurodevelopment as a risk factor for neurological consequences in the offspring later in life,focusing on the potential role of glial cells.Thus,it is important to consider future and long-term public health concerns associated with SARS-CoV-2 infection during pregnancy. | LARISSA DANIELE BOBERMIN LARA SCOPEL MEDEIROS FERNANDA WEBER GIANCARLO TOMAZZONI DE OLIVEIRA LUCÉLIA SANTI WALTER ORLANDO BEYS-DA-SILVA CARLOS-ALBERTO GONÇALVES ANDRÉQUINCOZES-SANTOS | 2022 | BIOCELL2022，46，12： | 0 |
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