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20篇 您的检索式:作者名="Brieger K"
    题名 作者 年代 出处 被引量
1Blocking capability of planar devices with field limiting rings显示文摘 Gerlach W Pelka J 1983Solid-State Electronics1983,26,8:1
2Heparin-induced thrombocytopenia显示文摘Brieger DB Mak KH Kottke-Marchant K 1998J Am Coil Cardiol1998,31,7:1
3The contour of an optimal field plate-an analytical approach显示文摘BRIEGER K P FLACK E GERLACH W 1988IEEE Trans Electron Devices1988,35,2:1
4Heparin-induced thrombocytopenia显示文摘Brieger DB Mak KH Kottke-Marchant K 1998Am Coll Cardiol1998,31,:1
5Acute Coronary Syndromes without Chest Pain, an Underdiagnosed and Un- dertreated High-risk Group: Insights from the Global Reg- istry of Acute Coronary Events显示文摘Brieger D Eagle K A Goodman S G 2004Chest2004,126,2:1
6Angiogenesis‐related prognosis in patients with oral squamous cell carcinoma—role of the VEGF +936 C/T polymorphism显示文摘Peer W. K?mmerer Bilal Al‐Nawas Sasa Kalkan Jan Liese Kai Fruth Bernhard Frerich Jürgen Brieger 2015J Oral Pathol Med2015,,:1
7Acute coronary syndromes without chest pain,anunderdiagnosed and undertreated high-risk group:insights from the global registry of acute coronaryevents显示文摘BRIEGER D EAGLE K A GOODMAN S G 2004Chest2004,126,:1
8Blocking Capability of Planar Devices with Field Limiting Rings显示文摘Brieger K P Gerlach W Pelka J 1983Solid-State Electronics1983,26,8:1
9Blocking capability of planar devices with field limiting rings显示文摘Brieger K P Gerlach W Pelka J 1983Solidstate Electron1983,26,8:1
10Blocking capability of planar devices with field limiting rings显示文摘Brieger K P Gerlach W Pelka J 1983Solid-State Electron1983,26,8:1
11Reactive oxygen species:from health to disease 显示文摘Brieger K Schiavone S Miller F J Jr Krause K H 2012Swiss Med Wkly2012,142,13:1
12The expression of the Wilms' tumor gene in acute myelocytic leukemias as a possible marker for leukemia blast cells显示文摘Brieger J Weidmann E Fenchel K 1994Leukemia1994,8,:1
13The contour of an optimal field plate: An analytical approach显示文摘BRIEGER K P FALCK E GERLACH W 1988IEEE Transactions on Electron Devices1988,35,5:1
14Acute coronary syn- dromes without chest pain, an underdiagnosed and undertreated high--risk group: insights from the Global Registry of Acute Coro- nary Events显示文摘Brieger D Eagle K A Goodman S G 2004Chest2004,126,:1
15'Blocking Cpability of Planar Devices with Field Limiting Rings' 显示文摘K P Brieger W Gerlanch J Pelka 1983Solid-state Electronics1983,,26:1
16The expression of the Wilms' tumor gene in acute myelocytic leukemias as a possible marker for leukemic blast cells显示文摘 Weidmann E Fenchel K 1994Leukemia1994,8,12:1
17The con- tour of an optimal field plate an analytical approach 显示文摘BRIEGER K P FALCK E GERLACH W 1988IEEE Trans Elec Dev1988,35,5:1
18Heparin-induced thrombocytopenia显示文摘Brieger DB Mak KH Kottke-Marchant K 1998Journal of American College of Cardiology1998,31,7:1
19Reactive oxygen species:from health to disease显示文摘Brieger K Schiavone S Miller F J Jr 0,,:1
20Image cytometric DNA analysis of mucosal biopsies in patients with primary achalasia显示文摘瞄准:为随后的快速的诊断在弛缓不能病人的粘膜活体检视决定 DNA 非整倍性。方法:从中间的活体检视第三根食管与弛缓不能在 15 个病人被获得。染色的 Immunohistochemical 为 p53 为 Ki67 和 PAb 1801 与单音的同种细胞的抗体 MIB-1 被执行,除了为发育异常的常规 histologic 考试。新鲜活体检视材料的原子核是酶的联盟者并且机械地孤立,并且 DNA 内容在福尔根的染色以后与图象血细胞计数被决定。恶意分级的 DNA 根据 Boecking 被估计决定在正常双山峰附近注意的 DNA 价值的可变性。测量的进一步的索引包括了整倍体的率,和 5c- , 7c-exceeding 和 9c-exceeding 评价。结果:组织学的考试没表明发育异常;当时 MIB-1 (基础) 在 8/15 弛缓不能标本显示出阳性反应, p53 在所有标本是否定的。图象 cytometric DNA 分析在 4/15 (26.7%) 检测了非整倍性标本。从有象标本一样的有鳞的房间癌的 15 个病人的样品只获得了 2 对作为参考测试服务的肿瘤厘米近似。象 9 件 peritumoral 样品(9/15 ) 一样的所有癌(15/15 ) 是一整倍体。从有 peritumoral 和癌标本的弛缓不能病人的活体检视的比较统计上揭示了有效差量考虑整倍体的率(双:P < 0.0001;四倍的:P = 0.001 ) ,根据 Boecking 恶意分级(P < 0.0001 ) 并且 5c-(P < 0.0001 ) , 7c-(P < 0.0001 ) ,并且 9c-(P = 0.0001 ) 在各自的顺序与 progredient DNA 改变超过率。结论:发现那 DNA 非整倍性被图象血细胞计数与弛缓不能在病人的食道的标本识别,它可能由于在 27% 病人作为癌前期损害介绍的特定的染色体改变,导致我们断定图象血细胞计数代表一个珍贵屏蔽工具。I Gockel P K(a|¨)mmerer J Brieger UR Heinrich WJ Mann F Bittinger VF Eckardt T Junginger 2006World Journal of Gastroenterology2006,12,19:0
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