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25篇 您的检索式:作者名="Burger SR"
    题名 作者 年代 出处 被引量
1Inactivation of the DNA repair gene O6-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,4:1
2Inactivation of theDNA repair gene O6-methylguanine DNA methyltransferase bypromoter hypermethylation is a common event in primary humanneoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,4:1
3Inactivation of the DNA repair gene the DNA repair gene06-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,:1
4Inactivation of the DNA repair gene 06-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia 显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,4:1
5Testosterone enhances estradiors effects on postmenopausal bone density and sexuality显示文摘Davis SR McCloud P Strauss BJ Burger H 1995Maturitas1995,21,:1
6Inactivation of the DNA repair gene O6-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘ESTELLER M HAMILTON SR BURGER PC 1999Cancer Res1999,59,:1
7Inactivation of the DNA repair gene O6-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,4:1
8Inactivation of the DNA repair gene O^6-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 1999CancerRes1999,59,4:1
9Inactivation of the DNA repair gene O^6 - methylguanine - DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia 显示文摘Esteller M Hamihon SR Burger PC 1999Cancer Res1999,59,4:1
10Inactivation of the DNA repair gene O6-methylguanine-DNAmethyItransferase by promoter hypermethylation is acommon event in primary humanneoplasia 显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,:1
11Inactivation of theDNA repair gene O6-methylguanine-DNA methyltransferase bypromoter hypermethylation is a common event in primary humanneoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer R es1999,59,4:1
12Inactivation of the DNA repair gene O6-methylguanine-DNA methyltransferase bypromoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,15:1
13Inactivation of the DNA repair gens O6-methylgnanine-DNA methyhransferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,4:1
14Inactivation of DNA repair gene O6-methyguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia 显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,:1
15Inactivation of the DNA repair gene O^6 - methylguanine - DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia 显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,:1
16Inactivation of the DNA repair gene 06 - methylguanine - DNA methyl- transferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 2009Cancer Res2009,59,4:1
17Inactivation of the DNA repair gene 06-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia 显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,:1
18Inactivation of the DNA repair gene O6-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,4:1
19Inactivation of the DNA repair gene 06-methylguanine-DNA methyltransferase by promoter hypermethylation is a common event in primary human neoplasia显示文摘Esteller M Hamilton SR Burger PC 1999Cancer Res1999,59,4:1
20Concentration of citrate anticoagulant in peripheral blood progenitor cell collections 显示文摘Burger SR Fautsch SK Stroncek DF 1996Transfusion1996,36,:1
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