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| 1 | Hepatitis B virus and hepatitis C virus dual infection显示文摘Hepatitis B virus(HBV)and hepatitis C virus(HCV)share common mode of transmission and both are able to induce a chronic infection.Dual HBV/HCV chronic coinfection is a fairly frequent occurrence,especially in high endemic areas and among individuals at high risk of parenterally transmitted infections.The intracellular interplay between HBV and HCV has not yet been sufficiently clarified,also due to the lack of a proper in vitro cellular model.Longitudinal evaluation of serum HBV DNA and HCV RNA amounts has revealed that complex virological profiles may be present in coinfected patients.Dual HBV/HCV infection has been associated to a severe course of the liver disease and to a high risk of developing hepatocellular carcinoma.Despite the clinical importance,solid evidence and clear guidelines for treatment of this special population are still lacking.This review summarizes the available data on the virological and clinical features as well as the therapeutic options of the dual HBV/HCV infection,and highlights the aspects that need to be better clarified. | Gaia Caccamo Francesca Saffioti Giovanni Raimondo | 2014 | World Journal of Gastroenterology2014，20，40： | 18 |
| 2 | Occult HBV infection显示文摘 | Giovanni Raimondo Gaia Caccamo Roberto Filomia Teresa Pollicino | 2013 | Seminars in Immunopathology2013，，1： | 4 |
| 3 | Cardiovascular risk after orthotopic liver transplantation, a review of the literature and preliminary results of a prospective study显示文摘Improved surgical techniques and greater efficacy of new anti-rejection drugs have significantly improved the survival of patients undergoing orthotopic liver transplantation(OLT). This has led to an increased incidence of metabolic disorders as well as cardiovascular and cerebrovascular diseases as causes of morbidity and mortality in OLT patients. In the last decade, several studies have examined which predisposing factors lead to increased cardiovascular risk(i.e., age, ethnicity, diabetes, NASH, atrial fibrillation, and some echocardiographic parameters) as well as which factors after OLT(i.e., weight gain, metabolic syndrome, immunosuppressive therapy, and renal failure) are linked to increased cardiovascular mortality. However, currently, there are no available data that evaluate the development of atherosclerotic damage after OLT. The awareness of high cardiovascular risk after OLT has not only lead to the definition of new but generally not accepted screening of high risk patients before transplantation, but also to the need for careful patient follow up and treatment to control metabolic and cardiovascular pathologies after transplant. Prospective studies are needed to better define the predisposing factors for recurrence and de novo occurrence of metabolic alterations responsible for cardiovascular damage after OLT. Moreover, such studies will help to identify the timing of disease progression and damage,which in turn may help to prevent morbidity and mortality for cardiovascular diseases. Our preliminary results show early occurrence of atherosclerotic damage, which is already present a few weeks following OLT, suggesting that specific, patient-tailored therapies should be started immediately post OLT. | Giuseppina Pisano Anna L Fracanzani Lucio Caccamo Maria F Donato Silvia Fargion | 2016 | World Journal of Gastroenterology2016，22，40： | 4 |
| 4 | Aortic stenosis: insights on pathogenesis and clinical implications显示文摘大动脉的狭窄(作为) 是在西方的人口的普通的瓣膜的心疾病,与在在 75 年的成年人的 3% 的估计的全面流行。为了理解它的 patho 生物的过程,代表优先级。在老病人,作为通常包含 trileaflet 阀门并且被叫作退化石灰质的过程。科学证据建议活跃 “ 的参与; atherosclerosis-like”在开始阶段的致病退化作为。到矛盾,前进能被不同力量驾驶(例如在发炎和石灰化之间的机械应力,基因因素和相互作用) 。改进理解为阻止并且禁止疾病的发展和前进介绍潜在地新的治疗学的目标。而且,在临床的实践管理作为病人暗示概括动脉粥样硬化患者表明的评估(即,在里面冠并且颈动脉动脉) 甚至为预示的原因。在 counselling 老病人,风险层化应该超过通用风险分数探讨单个脆弱。在这些,问候,合作病态,和特别地那些连接了到全球动脉粥样硬化患者负担,应该小心地被调查以便为侵略治疗策略定义风险 / 利益比率。我们在场在致病的卓见的详细概述就象可能的实际含意。 | Patrizia Carita Giuseppe Coppola Giuseppina Novo Giuseppa Caccamo Marco Guglielmo FabioBalasus Salvatore Novo Sebastiano Castrovinci Marco Moscarelli Khalil Fattouch Egle Corrado | 2016 | Journal of Geriatric Cardiology2016，13，6： | 3 |
| 5 | Mechanisms underlying lineage commitment and plasticity of human yo T cells显示文摘Phenotypic 和功能的异质是受动器和记忆 T 房间的特点。在抗原刺激之上, γ δT 房间区分进记忆 T 房间的二种主要类型:中央记忆房间,它巡逻血和第二等的淋巴的机关,和受动器记忆房间,它移居到外部纸巾。γ δT 房间在他们在常规 CD4 T 房间被观察的使人想起那的函数在 vitro 显示粘性的某个度。类似于 CD4 T 房间,在哪个专业化子集的过多影响主人反应, γ δT 房间可以乐意地并且很快假定不同 Th1- , Th2- , Th17- , T FH 和 T 象规章一样受动器功能,建议他们深刻地影响调停房间、体液的有免疫力的回答。除了在 cytokine 全部剧目的差别, γ δT 房间在 homing 展出差异,例如到淋巴节点滤泡的移植,到到煽动的纸巾的帮助 B 房间对移植。这里,我们考察我们 γ 的当前的理解; δT 房间系异质和灵活性与人的系统上的一个重音,并且建议受动器 γ 的一个分类; δT 房间基于不同功能的显型。 | Nadia Caccamo Matilde Todaro Guido Sireci Serena Meraviglia Giorgio Stassi Francesco Dieli | 2013 | Cellular & Molecular Immunology2013，10，1： | 2 |
| 6 | Handling execution overruns in hard real-time control systems显示文摘 | Caccamo M Buttazzo G Sha L | 2002 | IEEE Trans on Computers2002，51，7： | 1 |
| 7 | Non-sequencing molecular approaches to identify preS2-defective hepatitis B virus variants proved to be associated with severe liver diseases显示文摘 | Raimondo G Costantino L Caccamo G | 2004 | J Hepatol2004，40，3： | 1 |
| 8 | Cytokine profile,HLA restrictionand TCR sequence analysis of human CD4+ T clonesspecific for an immunodominant epitope of Mycobacte-rium tuberculosis 16-kDa protein显示文摘 | Caccamo N Barera A Di Sano C Meraviglia S IvanyiJ Hudecz F | 2003 | Clin Exp Immu-nol2003，133，： | 1 |
| 9 | Insulin resistance and acute coronarysyndrome显示文摘 | Caccamo G Bonura F Bonura F | 2010 | Atherosclerosis2010，211，2： | 1 |
| 10 | Differential antigen or homeostatic cytokJnes for proliferation and requirements differentiation human Vgamma 9V~elta 2 naive,memory and effector T cell subsets显示文摘 | Caccamo N Nerayiglia S Ferlazzo V et a~ | 2005 | Eur J Immunol2005，55，6： | 1 |
| 11 | Glutamate promotes NF-kappaB pathway in primary astrocytes: protective effects of IRFI016, a synthetic vitamin E analogue显示文摘 | Caccamo D Campisi A Marini H | 2005 | Exp Neurol2005，193，2： | 1 |
| 12 | Monitoring of transglutaminase2 under different oxidative stress conditions显示文摘 | Daniela Caccamo Monica Currò Nadia Ferlazzo Salvatore Condello Riccardo Ientile | 2012 | Amino Acids2012，，2： | 1 |
| 13 | roTor regulates tan phosphorylation and degradation: implications for alzheimer' s disease and other tauopathies显示文摘 | Caccamo A Magri A Medina DX | 2013 | Aging Cell2013，12，3： | 1 |
| 14 | Celsior solution and clinical liver transplantation显示文摘 | Maggi U Caccamo L Gatti S | 2000 | Transplant Proc2000，32，1： | 1 |
| 15 | Th0 to Th1 switch of CD4 T cell clones specific from the 16-kDa antigen of Mycobacterium tuberculosis after successful therapy:lack of involvement of epitope repertoire and HLA-DR显示文摘 | CACCAMO N MERAVIGLIA S DIELI F | 2005 | Immunol Lett2005，98，2： | 1 |
| 16 | Nuclear translocation of a clusterin isoform is associated with induction of anioikis in SV40-immortalized human prostate epithelial cell显示文摘 | Caccamo AE Scaltriti M Caporali A | | 0，，： | 1 |
| 17 | Loss of muscarinic M1 receptor exacerbates alzheimer's disease-like pathology and cognitive decline显示文摘 | Medeiros R Kitazawa M Caccamo A | 2011 | The American journal of pathology2011，179，2： | 1 |
| 18 | Triple-transgenic model of Alzheimers disease with plaques and tangles:intracellular Abeta and synaptic dysfunction显示文摘 | Oddo S Caccamo A Shepherd JD | 2003 | Neuron2003，39，3： | 1 |
| 19 | Atypical and malignant meningiomas:a clinicopathological review显示文摘 | Mahmood A Caccamo DV Tomecek FJ | 1993 | Neurosurgery1993，33，： | 1 |
| 20 | Identification of thermophilic and marine bacilli from shallow thermal vents by restriction analysis of their amplified 16S rDNA显示文摘 | Caccamo D Maugeri TL Gugliandolo C | 2001 | Journal of Applied Microbiology2001，91，3： | 1 |
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