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24篇 您的检索式:作者名="Carter MG"
    题名 作者 年代 出处 被引量
1Molecular analysis of circu- lating tumour cells-biology and biomarkers显示文摘Krebs MG Metcalf RL Carter L 2014Nat Rev Clin Oncol2014,11,3:1
2Age -associated alteration of gene expression patterns in mouse oocytes 显示文摘Hamatani T Falco G Carter MG 2004Hum Mol Genet2004,13,:1
3谷氨酰胺增强结肠直肠切除病人的T淋巴细胞反应显示文摘研究目的:作者测定了在行直结肠切除的患者中,谷氨酰胺强化的肠外营养支持对手术后外周血T细胞反应及炎症细胞因子生成的影响。 背景资料:包括免疫系统在内的几种有机体组织的正常功能依赖于谷氨酰胺;然而这种氨基酸,虽然可能为应激状态所必需,近年才正式将它用于肠外营养液中。这种强化的肠外营养液对处于应激状态的外科病人的免疫功能的影响尚不清楚。 研究方法:患者(n=20)随机分为两组,在术后1~6天,一组接受传统的肠外营养液治疗(每天0.2克氮/kg),另一组每天接受含有0.188/kg谷氨酰胺的等氮等热卡方案治疗。分别在术前和术后第1~6天,体外测定:T细胞DNA合成、IL-2的生成情况以及外周血单核细胞IL-6和肿瘤坏死因子(TNF)的生成情况。 研究结果:行肠外营养治疗5天后,与术前相比,T细胞DNA的合成在谷氨酰胺强化组病人中增加了(术前~3H标记的胸腺嘧啶的平均摄取为78.3×103cpm;第6天为95.0×103cpm,P<0.05),而在对照的治疗组患者中,T细胞DNA合成没有增加(术前为89.0×103cmp,第6天为69.4×10~3cpm,P<0.05)。添加谷氨酰胺并不影响IL-2,TNF和IL-6的生成水平。 结论:添加谷氨酰胺可能是增强接受肠外营养治疗的外科病人的T细胞功能的一种方法。O'Riordain MG Fearon KCH Ross JA Rogers P Falconer JS Bartolo DCC Garden OJ Carter DC 赵志全 1996中华临床营养杂志1996,14,1:1
4Heterozygous disruption of Hie1 predisposes mice to a gender-dependent spectrum of malignant tumors 显示文摘Chen WY Zeng X Carter MG 2003Nat Genet2003,33,2:1
5Molecular analysis of circulating tumour cells-biology and biomarkers显示文摘Krebs MG Metcalf RL Carter L 2014Nat Rev Clin Oncol2014,11,3:1
6The effects of supplementation with creatine and protein on muscle strength following a traditional resistance training'program in middle-aged and older men显示文摘Bemben MG Witten MS Carter JM 2010J Nutr Health Aging2010,14,15:1
7Heterozygous disruption of Hic1 predisposes mice to a genderdependent Spectrum of malignant tumors 显示文摘Chen WY Zeng X Carter MG 2003Nat Genet2003,33,:1
8An in situ hybridization-based screen for heterogeneously expressed genes in mouse ES cells显示文摘Carter MG Stagg CA Falco G 2008Gene Expr Patterns2008,8,3:1
9Dissecting Oct3/4-regulated gene networks in embryonic stem cells by expression profiling显示文摘Matoba R Niwa H Masui S Ohtsuka S Carter MG Sharov AA 2006PLoS ONE2006,20,1:1
10Heterozygous disruption of Hiel predisposes mice to a gender-dependent spectrum of malignanl tunors显示文摘Chen WY Zeng X Carter MG 2003Nat Genet2003,33,2:1
11Heterozygous disruption of Hic1 predisposes mice to a gender-dependent speetrum of malignant tumors显示文摘Chen WY Zeng X Carter MG 2003Nat Genet2003,33,2:1
12Heterozygous disruption of Hiel predisposes mice to a gender-dependent spectrumm of malignanl tunors 显示文摘Chen WY Zeng X Carter MG 2003nat Genet2003,33,2:1
13Molecular analysis of circulating tumor cells-biology and biomarkers 显示文摘Krebs MG Metcalf RL Carter L 2014Na- ture Rev Clin Oncol2014,11,3:1
14The ineidenee of prostate cancer progression with undetectable serum prostate specific antigen in a series of 394 radical prostatectomies显示文摘Oefelein MG Smith N Carter M 1995J Urel1995,154,6:1
15A helminth glycan induces APC maturation via alternative NF-kappa B activation independent of I kappa B alpha degradation显示文摘Thomas PG Carter MG Da'dara AA 2005J Immunal2005,175,:1
16The incidence of prostate cancer progression with undetectable serum prostate specific antigen in a series of 394 radical prostatectomies显示文摘Oefelein MG Smith N Carter M 1995J Urol1995,154,6:1
17Dynamics of global gene expression changes during mouse preimplantation development 显示文摘Hanmtani T Carter MG Sharov AA 2004Dev Cell2004,6,1:1
18Mice deficient in the candidate tumor suppressor gene Hicl exhibit developmental defects of structures affected in the Miller-Dieker syndrome 显示文摘Carter MG Johns MA Zeng X 2000Hum Mol Genet2000,9,:1
19Molecular analysis of circulating tumour cells-biology and biomarkers显示文摘Krebs MG Metcalf RL Carter L 2014Nat Rev Clin Oncol2014,11,3:1
20Cell host response to infection with novel human coronavirus EMC predicts potential antivirals and important differences with SARS coronavirus 显示文摘Josset L Menachery VD Gralinski LE Agnihothram S Sova P Carter VS Yount BL Graham RL Baric RS Katze MG 2013MBio2013,4,00:1
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