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| 1 | Major comorbid disease processes associated with increased incidence of acute kidney injury显示文摘Acute kidney injury(AKI) is commonly seen amongst critically ill and hospitalized patients. Individuals with certain co-morbid diseases have an increased risk of developing AKI. Thus, recognizing the co-morbidities that predispose patients to AKI is important in AKI prevention and treatment. Some of the most common co-morbid disease processes that increase the risk of AKI are diabetes, cancer, cardiac surgery and human immunodeficiency virus(HIV) acquired immune deficiency syndrome(AIDS). This review article identifies the increased risk of acquiring AKI with given co-morbid diseases. Furthermore, the pathophysiological mechanisms underlying AKI in relation to co-morbid diseases are discussed to understand how the risk of acquiring AKI is increased. This paper reviews the effects of various comorbid diseases including: Diabetes, cancer, cardiovascular disease and HIV AIDS, which all exhibit a significant increased risk of developing AKI. Amongst these comorbid diseases, inflammation, the use of nephrotoxic agents, and hypoperfusion to the kidneys have been shown to be major pathological processes that predisposes individuals to AKI. The pathogenesis of kidney injury is complex, however, effective treatment of the co-morbid disease processes may reduce its risk. Therefore, improved management of co-morbid diseases may prevent some of the underlying pathology that contributes to the increased risk of developing AKI. | Salwa Farooqi Jeffrey G Dickhout | 2016 | World Journal of Nephrology2016，5，2： | 4 |
| 2 | Decreased Endogenous Production of Hydrogen Sulfide Accelerates Atherosclerosis显示文摘 | Sarathi Mani Hongzhu Li Ashley Untereiner Lingyun Wu Guangdong Yang Richard C. Austin Jeffrey G. Dickhout ?árka Lhoták Qing H. Meng Rui Wang | 2013 | Circulation2013，，25： | 2 |
| 3 | Structural and functional analysis of arteries from young spontaneously hypertensive rats显示文摘 | Dickhout JG Lee RM | 1997 | Hypertension1997，29，： | 1 |
| 4 | Interrelationship between cardiac hypertrophy,heart failure,and chronic kidney disease:endoplasmic reticulum stress as a mediator of pathogenesis显示文摘 | Dickhout JG Carlisle RE Austin RC | 2011 | Circ Res2011，108，5： | 1 |
| 5 | Structural and functional analysis of small arteries from young spontaneously hypertensive rats显示文摘 | Dickhout JG Lee RMKW | 1997 | Hypertension1997，29，3： | 1 |
| 6 | Structural and fimcfional analysis of small arteries from young spontaneomdy hypertensive rats显示文摘 | Dickhout JG Lee RM | 1997 | Hypertension1997，29，3： | 1 |
| 7 | Apoptosis in the muscular arteries from young spontaneously hypertensive rats显示文摘 | Dickhout JG Lee RM | 1999 | Hypertens1999，17，10： | 1 |
| 8 | Structural and functional analysis of small arteries from young spontaneously hypertensive rats显示文摘 | Dickhout JG Lee RM | 1997 | Hypertension1997，29，3： | 1 |
| 9 | Interrelationship between cardiac hypertrophy, heart failure, and chronic kidney disease: endoplasmic reticulum stress as a mediator of pathogenesis 显示文摘 | Dickhout JG Carlisle RE Austin RC | 2011 | Circ Res2011，108，5： | 1 |
| 10 | Blood pressure and heart rate development in young spontaneously hypertensive rats 显示文摘 | Dickhout JG Lee RMKW | 1998 | American Journal of Physiology-Heart and Circulatory Physiology1998，274，3： | 1 |
| 11 | Structural and functional analysis of small arter- ies from young spontaneously hypertensive rats 显示文摘 | Dickhout JG Lee RM | 1997 | Hypertension1997，29，3： | 1 |
| 12 | Structural and functional analysis of small arteries from young spontaneously hypertensive rats 显示文摘 | Dickhout JG Lee RMKW | 1997 | Hypertension1997，29，： | 1 |
| 13 | Tubulovascular nitric oxide crosstalk: buff ering of angiotensin 1I induced medullary vasoconstriction 显示文摘 | Dickhout JG Mori T Cowley AW Jr | 2002 | Circ Res2002，91，6： | 1 |
| 14 | Increased medial smooth muscle cell length is responsible for vascular hypertrophy in young hypertensive rats显示文摘 | Dickhout JG Lee RM | 2000 | Am J Physiol Heart Circ Physiol2000，279，： | 1 |
| 15 | Brown Norway Chromosome 13 Confers Protection From High Salt to Consomic Dahl S Rat显示文摘 | Allen W. Cowley Richard J. Roman Mary L. Kaldunski Pierre Dumas Jeffrey G. Dickhout Andrew S. Greene Howard J. Jacob | | 2001 (2, P)0，，： | 1 |
| 16 | Increased endoplasmic reticulmn stress in atherosclerotic plaques associated with acute coronary syndrome: a balancing act between plaque stability and rupture显示文摘 | Dickhout JG Colgan SM Lhotak S | 2007 | Circulation2007，116，11： | 1 |
| 17 | Peroxynitrite causes endoplasmic reticulum stress and apoptosis in human vascular endothelium: implications in atherogenesis 显示文摘 | Dickhout JG Hossain GS Pozza LM | 2005 | Arterioscler Thromb Vase Bio12005，25，： | 1 |
| 18 | Structural and functional analysis of arteries from young spontaneously hypertensive rats显示文摘 | Dickhout JG Lee RM | 1997 | Hypertension1997，29，3： | 1 |
| 19 | Interrelationship between cardiac hypertrophy,heart failure,and chronic kidney disease:endoplasmic reticulum stress as a mediator of pathogenesis显示文摘 | Dickhout JG Carlisle RE Austin RC | | 0，，5： | 1 |
| 20 | Vasopressin increases intracellular NO concentration via Ca2+ signaling in inner medullary collecting duct显示文摘 | Mori T Dickhout JG Cowley AW | | 0，，2： | 1 |
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