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20篇 您的检索式:作者名="Dorr O"
    题名 作者 年代 出处 被引量
1Neuropeptide Y as an indicator of successful alterations in sympathetic nervous activity after renal sym- pathetic denervation显示文摘DORR O EWEN S LIEBETRAU C 2015Clin Res Cardiol2015,104,12:1
2Neutrophil ge- latinase-associated lipocalin(NGAL) for the early detection of cardiac surgery associated acute kidney injury显示文摘Liebetrau C Dorr O Baumgarten H 2013Scand J Clin Lab Invest2013,73,5:1
3222Rn flux and soil air concentration profiles in West-Germany:soil 222Rn as atracer for gas transport in the unsaturated soil zone显示文摘DORR H MUNNICH K O 1990Tellus1990,42,:1
4Annual variation in soil respiration in selected areas of the temperate zone 显示文摘Dorr J W & Munnich K O 1987Tellus1987,39,12:1
5Release kinetics of circulating muscle - enriched microRNAs in patients undergoing transcoronary ab- lation of septal hypertrophy 显示文摘Liebetrau C Mollmann H Dorr O 2013J Am Coil Cardiol2013,62,11:1
6Lead and Cesium transport in European forest soils显示文摘Dorr H Munnich K O 1991Water Air and Soil Pollution1991,57,:1
7Annual variations in the ^14C content of soil CO2显示文摘Dorr H Munnich K O 1986Radiocarbon1986,28,:1
8GRACE riskscore as predictor of in-hospital mortality in patientswith chest pain显示文摘Stracke S Dorr O Heidt MC 2010Clin Res Cardiol2010,99,10:1
9Annual variation in soil respiration in selected areas of temperate zone显示文摘 Munnich K O 1987Tellus1987,39,:1
10Annual variation in soil respiration in selected areas of the temperate zone 显示文摘Dorr H K O Munich 1987Tellus1987,,:1
11Annual variation in soil respi ration in selected areas of the temperate zone显示文摘Dorr H Minnich K O 1987Tel- lus B1987,39,12:1
12Genotypic features of lentivirus transgenic mice显示文摘Sauvain M O Dorr A P Stevenson B Quazzola A Naef F Wiznerowicz M Schiatz F Jongeneel V Duboule D Spitz F Trono D 2008J Virol2008,82,14:1
13Annual variation in soil respiration in selected areas of the temperate zone 显示文摘Dorr H Munnich K O 1987Tellus1987,9,:1
14Neuropeptide Y as an indicator of successful alterations in sympathetic nervous activity after renal sympathetic denervation显示文摘Dorr O Ewen S Liebetrau C 2015Clin Ras Cardiol2015,104,12:1
15Downward movement of soil organic matter and its influence on trace-element transport (210Pb,137Cs) in the soil显示文摘 Munnich K O 1989Radiocarbon1989,31,3:1
16Downward movement of soil organic matter and its influence on trace-element transport (^210 Pb, ^137Cs)in the soil显示文摘Dorr H Munnich K O 1989Radiocarbon1989,31,3:1
17Annual variation in soil respiration in selected areas of the temperate zone 显示文摘Dorr H Mfinich K O 1987Tellus1987,,:1
18Rationale of the knee society clinical rating system显示文摘Insall J N Dorr L D Scott R O 2014Clin Orthrop Relat Res2014,248,248:1
19Renal sym- pathetic denervation does not aggravate functional or structur- al renal damage 显示文摘DORR O LIEBETRAU C MOLLMANN H 2013J Am Coil Cardiol2013,61,4:1
20Is hepatic steatosis associated with left ventricular mass index increase in the general population?显示文摘AIM To investigate the association between hepatic steatosis and change in left ventricular mass index(LVMI) over five years, and examine whether systolic and diastolic blood pressures are mediators of the association between hepatic steatosis and LVMI using a general population sample.METHODS We analyzed data from the Study of Health in Pomerania. The study population comprised 1298individuals aged 45 to 81 years. Hepatic steatosis was defined as the presence of a hyperechogenic pattern of the liver together with elevated serum alanine transferase levels. Left ventricular mass was determined echocardiographically and indexed to height2.7. Path analyses were conducted to differentiate direct and indirect paths from hepatic steatosis to LVMI encompassing systolic and diastolic blood pressure as potential mediating variables.RESULTS Hepatic steatosis was a significant predictor for all measured echocardiographic characteristics at baseline. Path analyses revealed that the association of hepatic steatosis with LVMI change after five years was negligibly small(β =-0.12, s.e. = 0.21, P = 0.55). Systolic blood pressure at baseline was inversely associated with LVMI change(β =-0.09, s.e. = 0.03, P < 0.01), while no association between diastolic blood pressure at baseline and LVMI change was evident(β = 0.03, s.e. = 0.05, P = 0.56). The effect of the indirect path from hepatic steatosis to LVMI via systolic baseline blood pressure was small(β =-0.20, s.e. = 0.10, P = 0.07). No indirect effect was observed for the path via diastolic baseline blood pressure(β = 0.03, s.e. = 0.06, P = 0.60). Similar associations were observed in the subgroup of individuals not receiving beta-blockers, calcium channel blockers, or drugs acting on the reninangiotensin system.CONCLUSION Baseline associations between hepatic steatosis and LVMI do not extend to associations with LVMI change after five years. More studies are needed to study the longitudinal effects of hepatic steatosis on LVMI.Katharina Piontek Carsten O Schmidt Sebastian E Baumeister Markus M Lerch Julia Mayerle Marcus Dorr Stephan B Felix Henry Volzke 2017World Journal of Hepatology2017,9,19:0
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