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7篇 您的检索式：作者名="Eitan Friedman"

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		题名	作者	年代	出处	被引量
	1	Spontaneous and therapeutic abortions and the risk of breastcancer among BRCA1mutation carriers显示文摘	Eitan Friedman Joanne Kotsopulos Jan Lubinski	2006	Breast Cancer Res2006，15，8：	1
	2	Detection of Pancreatic Carcinoma (Diagnostic Value of K-ras Mutations in Circulating DNA from Serum)显示文摘	Livia Theodor Ehud Melzer Michael Sologov Gila Idelman Eitan Friedman Simon Bar-Meir	1999	Digestive Diseases and Sciences1999，，10：	1
	3	Detection of Pancreatic Carcinoma (Diagnostic Value of K-ras Mutations in Circulating DNA from Serum)显示文摘	Livia Theodor Ehud Melzer Michael Sologov Gila Idelman Eitan Friedman Simon Bar-Meir	1999	Digestive Diseases and Sciences1999，，10：	1
	4	Spontaneous and therapeutic abortions and the risk of breast cancer among BR-CA1 mutation carriers显示文摘	Eitan Friedman Joanne Kotsopulos Jan Lubinski		0，，08：	1
	5	PSD-95 regulates D1 dopamine receptor resensitization, but not receptor-mediated Gs-protein activation显示文摘现在的学习试图定义 postsynaptic 的角色在多巴胺(DA ) 的规定的密度(PSD )-95 受体功能。我们发现 PSD-95 身体上在 co-transfected HEK-293 房间与 D1 或 D2 DA 受体联系。DA 受体的刺激以一种时间依赖者方式改变了在 D1 受体和 PSD-95 之间的协会。功能的试金显示 PSD-95 合作表示没影响 D1 刺激受体的营地生产, Gs 蛋白质激活或受体不敏感性。然而, PSD-95 由支持受体再循环加速了使内在化的膜受体的恢复,因此导致使内在化的 D1 受体的提高的促进感受性。我们的结果提供新奇机制因为调整再循环那的 DA 受体可以在 postsynaptic DA 功能的调整和 synaptic neuroplasticity 起一个重要作用。	Peihua Sun Jmgru Wang Welhua Gu Wei Cheng Guo-zhang Jin Eitan Friedman Jie Zheng Xuechu Zhen	2009	Cell Research2009，19，5：	0
	6	GSK3β regulates D1 dopamine receptor phosphorylation and function显示文摘	Wang Jing- Ru Sun Pei -Hua Friedman Eitan Jin Guo -Zhang Zhen Xue -Chu	2006	中国药理通讯2006，23，4：	0
	7	Protein Kinase Activity Changes in the Aging Brain显示文摘The aging process in mammals is correlated with changes in psychomotor performance, cognitive function, and ability to adapt to stress (Montgomery et al., 1982; Lorens et al., 1990). These changes may be related to alterations in neuronal tissue that occur during the aging process. The normal aging process may be conceived of as the neuronal cell's increasing inability to maintain normal cellular function which ultimately results in a number of morphological and biochemical changes. Morphologically, there is a loss of neuronal cells with increasing age (Brizzee	MICHAE LR.PISANO HOAU-YAN WANG EITAN FRIEDMAN	1991	Biomedical and Environmental Sciences1991，4，1：	0

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