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11篇 您的检索式：作者名="Fruciano"

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		题名	作者	年代	出处	被引量
	1	Anti-inflammatory and antifi- brotic effects of resveratrol in the lung 显示文摘	Conte E Fagone E Fruciano M	2015	Histol HJstopatbol2015，30，5：	1
	2	A short history of phage therapy 显示文摘	Dublanchet A Fruciano E	2008	Med Mal Infect2008，38，8：	1
	3	Inhibition of PI3K prevents the proliferation and differentiation of human lung fibroblasts into myofibroblasts:the role of class I Pll0 isoforms显示文摘	CONTE E FRUCIANO M FAGONE E		0，，：	1
	4	Nhibition of P13K prevents the proliferation and differentiation of human lung fi- broblasts into myofibroblasts: the role of class I P110 isoforms显示文摘	CONTE E FRUCIANO M FAGONE E	2011	PLoS One2011，6，：	1
	5	Inhibition of PI3K prevents the prolifetaion and differentiation of human lung fibroblasts into myo- fibrobalsts: the role of class I P110 isoforms显示文摘	Conte E Fruciano M Fagone E	2011	PLoS One2011，6，24：	1
	6	A short history of phage therapy 显示文摘	Dublanchet A Fruciano E	2008	Med Mal Infect2008，38，8：	1
	7	Inhibition of PI3K prevents the proliferation and differentiation of human lung fibroblasts into myofibroblasts:The Role of Class I P110 Isoforms显示文摘	Conte E Fruciano M Fagone E		0，，10：	1
	8	Inhibition of PI3K prevents the proliferation and differentiation of human lung fibroblasts into myofibroblasts: the role of class I Pll0 isofomls显示文摘	Conte E Fruciano M Fagone E	2011	PLoS One2011，6，24：	1
	9	Inhibition of PI3K prevents the proliferation and differentiation of human lung fibroblasts into myofibroblasts: the role of class I P110 isoforms显示文摘	Conte E Fruciano M Fagone E	2011	PLoS One2011，6，24：	1
	10	Inhibition of PI3K prevents the proliferation and differentiation of human lung fibroblasts into myofibroblasts: the role of class I P110 isoforms 显示文摘	Conte E Fruciano M Fagone E	2011	PLoS One2011，6，24：	1
	11	Does divergence from normal patterns of integration increase as chromosomal fusions increase in number?A test on a house mouse hybrid zone显示文摘Chromosomal evolution is widely considered an important driver of speciation because it can promote the establishment of reproductive barriers.Karyotypic reorganization is also expected to affect the mea n phe no type,as well as its developme nt and patter ns of phenotypic in tegrati on,through processes such as variation in genetic linkage between quantitative trait loci or between regulatory regions and their targets.Here we explore the relationship between chromosomal evolution and phenotypic integration by analyzing a well-known house mouse parapatric contact zone between a highly derived Robertsonian(Rb)race(2n=22)and populations with standard karyotype(2n=40).Populations with hybrid karyotypes are scattered throughout the hybrid zone connecting the two parental races.Using mandible shape data and geometric morphometries,we test the hypothesis that patter ns of in tegration progressively diverge from the'normal'integration pattern observed in the standard race as they accumulate Rb fusions.We find that the main pattern of integration observed between the posterior and anterior part of the mandible can be largely attributed to allometry.We find no support for a gradual increase in divergence from normal patterns of integration as fusions accumulate.Surprisingly,however,we find that the derived Rb race(2n=22)has a distinct allometric trajectory compared with the standard race.Our results suggest that either individual fusions disproportionately affect patterns of integration or that there are mechanisms which'purge'extreme variants in hybrids(e.g.reduced fitness of hybrid shape).	Carmelo FRuciANO Paolo Colangelo Riccardo Castiglia Paolo Franchini	2020	Current Zoology2020，66，5：	0

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