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20篇 您的检索式:作者名="GREMESE E"
    题名 作者 年代 出处 被引量
1Patelet GPIIb/IIIa (PIAI/2)Pol- ymorphism in SLE : clinical and laboratory association 显示文摘Tolusso B Fabrism Gremese E 2003Ann Rheum Kis2003,40,8:1
2Autoantibodies and thrombophilia in RA: TNF and TNF blockers显示文摘Ferraccioli GF Gremese E 2004Annals of the Rheumatic Diseases2004,63,:1
3The metabolic syndrome: the cross- roads between rheumatoid arthritis and cardiovascular risk 显示文摘Gremese E Ferraccioli G 2011Autoimmun Rev2011,10,:1
4Pathogenetic, clinical and pharmaco-economic assessment in rheuma- toid arthritis (RA)显示文摘Ferraccioli G Gremese E 2011Intern Emerg Med2011,6,1:1
5Thrombagenicity of TNF alpha in rheumatoid arthritis defined through biological probes: TNF alpha blockers显示文摘Ferraceioli G Gremese E 2004Autoimmun Rev2004,3,4:1
6Platelet GPIlb/IIIa (PIA 1/2) polymorphism in SLE:clinical and laboratory association显示文摘i Tolusso B Fabris M Gremese E 2003Ann Rheum Dis2003,62,8:1
7Platelet GPIIb/IIIa ( P1 A 1/ 2) polymorphism in SLE: clinical and laboratory association 显示文摘Tolusso B Fabris M Gremese E 2003Ann Rheum Dis2003,62,8:1
8Platelet GPIIb/IIIa(PIA1/2) Polymorphism in SEE:clinical and labora tory association显示文摘Tolusso B fabrism Gremese E 2003Ann Rheum Kis2003,40,8:1
9Platelet GPⅡb/Ⅲa (PIA 1/2) polymorphism in SLE: clinical and laboratory association 显示文摘Tolusso B Fabris M Gremese E 2003Ann Rheum Dis2003,62,8:1
10Platelet GP Ⅱb/Ⅲa (PIA 1/2) polymorphism in SLE: clinical and laboratory association显示文摘Tolusso B Fabris M Gremese E 2003Ann Rheum Dis2003,62,78:1
11Analysis of improvements, full responses, remissionand toxicity in rheumatoid patients treated with step-up combination therapy (mathotraxate, cyclosporine A, sulphasalazine) or monotherapy for three years 显示文摘Ferraccoill GF Gremese E Tomletto P 2002Rheumatology (Oxford)2002,41,8:1
12Thrombogenicity of TNF alpha in rheumatoid arthritis defined through biological probes:TNF alpha blockers显示文摘FERRACCIOLI G GREMESE E 2004Autoimmun Rev2004,3,4:1
13Memory B cell suhsets and plasmablasts are lower in early than in long-standing rheuma- toid arthritis显示文摘Fedele AL Tolusso B Gremese E 2014BMC Immunol2014,15,:1
14Platelet GPⅡb/Ⅲa(PIA 1/2)polymorphism in SLE: clinical and laboratory association 显示文摘Tolusso B Fabris M Gremese E 2003Ann Rheum Dis2003,62,8:1
15Pathogenetic, clinical and pharmaco-economic assessment in rheuma- toid arthritis (RA)显示文摘Ferraccioli G Gremese E 2011Intern Emerg Med2011,6,1:1
16Obesity and reduction of the response rate to anti-tumor necrosis factor α in rheumatoid arthritis:an approach to a personalized medicine显示文摘Gremese E Carletto A Padovan M 0,,01:1
17Ptatelet GPⅡb/ Ⅲa(PIA1/2) Polymorphism in SLE: clinical and laboratory association显示文摘Tolusso B Fabrism Gremese E 2003Ann Rheum Dis2003,62,8:1
18Analysis of improvements, full responses, remission and toxicity in rheumatoid patients treated with step-up combination therapy (methotrexate, cyclosporin A, sulphasalazine) or monotherapy for three years显示文摘Ferraccioli GF Gremese E Tomoetto P 2002Rheumatology (Oxford)2002,41,8:1
19Pathogenetic, clinical and pharmaco-economic assessment in rheumatoid arthritis (RA)显示文摘Ferraccioli G Gremese E 2011Intern Emerg Med2011,6,1:1
20Benefit/risk of cyclosporine in rheumatoid arthritis显示文摘Gremese E Ferraccioli GF 2004Clin Exp Rheumatol2004,22,5135:1
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