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37篇 您的检索式：作者名="Gaussin"

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	题名	作者	年代	出处	被引量
1	Cardenolide-induced lysosomal membrane permeabilization demonstrates therapeutic benefits in experimental human non-small cell lung cancer显示文摘	Tatjana Mijatovic Veronique Mathieu Jean-Francois Gaussin	2006	Neoplasia2006，8，5：	1
2	TAK1 is activated in the myocardium after pressure overload and is sufficient to provoke heart failure in transgenic mice显示文摘	Zhang D Gaussin V Taffet GE et a1	2000	Nat Med2000，6，5：	1
3	Inhibition of myocardial glucose uptake by cyclic GMP显示文摘	Depre C Gaussin V Ponchaut S	1998	Am J Physiol1998，274，：	1
4	TAK1 is activated in the myocardium after pressure overload and is sufficient to provoke heart failure in transgenic mice显示文摘	Gaussin V Taffet GE	2000	Nat Med2000，6，：	1
5	Endcardial cushion and myocardial defects after cardiac myocyte-specific conditional deletion of the bone morphogenetic protein receptor ALK3显示文摘	Gaussin V Van de Putte T Mishina Y	2002	Proc NatlAcad Sci USA2002，99，5：	1
6	Cardenolide-induced lysosomal membrane permeabilization demonstrates therapeutic benefits in experimental human non-small cell lung cancers显示文摘	Mijatovic T Mathieu V Gaussin J F	2006	Neoplasia2006，8，5：	1
7	Endocardial cushion and myoeardial defects after cardiac myocyte-Schneider conditional deletion of the bone morphogenetic proteinreceptorALK3显示文摘	Gaussin V Van de Putte T Mishina Y	2002	Proc Natl Acad Sci USA2002，99，5：	1
8	Endocardial cushion and myocardial defects after cardiac myocyte-specific conditional deletion of the bone morphogenetic protein receptor ALK3显示文摘	Gaussin V Behringer RR Schneider M D	2002	Proc Natl Acad Sci USA2002，99，5：	1
9	Are syngeneic mouse tumor models still valuable experimental models in the field of anti-cancer drug discovery显示文摘	Darro F Decaestecker C Gaussin JF	2005	Int J Oncol2005，27，3：	1
10	TAK1 is activated in the myocardium after pressure overload and is sufficient to provoke heart failure in transgenic mice 显示文摘	Zhang D Gaussin V Taffet G E	2000	Nat Med2000，6，5：	1
11	Endocardial cushion and myocardial defects after cardiac myocyte-specific conditional deletion of the bone morphogenetic protein receptor ALK3显示文摘	Gaussin V Van de Putte T Mishina Y	2002	Proc Natl Acad Sci USA2002，99，5：	1
12	TAK1 is activatedin the myocardium after pressure ovedoad and is sufficient to provoke heart failure in transgenic mice 显示文摘	Zhang D Gaussin : Taffet G E	2000	Nat Med2000，6，5：	1
13	H11 kinase is a novel mediator of myocardial hypertrophy in vivo显示文摘	Depre C Hase M Gaussin V	2002	Circ Res2002，91，11：	1
14	Endocardial cushion and myocardial defects after cardiac myocyte-specific conditional deletion of the bone morphogenetic protein receptor ALK3显示文摘	Gaussin V Van de putte T Mishina Y	2002	Proc Natl Acad Sci USA2002，99，5：	1
15	H11 kinase is a novel mediator of myocardial hypertrophy in vivo显示文摘	Depre C Hase M Gaussin V	2002	Circ Res2002，91，11：	1
16	Assessing the environmental footprint of manufactured products: A survey of current literature显示文摘	M. Gaussin G. Hu S. Abolghasem S. Basu M.R. Shankar B. Bidanda	2012	International Journal of Production Economics2012，，：	1
17	Endocardial cushion and myocardial defects after cardiac myocyte-specific conditional deletion of the bone morphogenetic protein receptor ALK3显示文摘	GAUSSIN V van de PUTTE T MISHINA Y	2002	Proceedings of the National Academy of Sciences2002，99，5：	1
18	Sustained transgene expression using MAR elements显示文摘	Harraghy N Gaussin A Mermod N	2008	Curr Gene Ther2008，8，5：	1
19	Cardenolide-induced lysosomal membrane permeabilization demonstrates therapeutic benefits in experimental human non-small cell lung cancers 显示文摘	Mijatovic T Mathieu V Gaussin J F	2006	Neoplasia2006，8，：	1
20	H11 kinase is a novel mediator of myocardial hypertrophy in vivo显示文摘	Depre C Habe M Gaussin V	2002	Circulation Research2002，91，11：	1

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