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11篇 您的检索式:作者名="Ghayad S"
    题名 作者 年代 出处 被引量
1Inhibitors of the PI3K/ Akt/mTOR pathway: new hope for breast cancer patients显示文摘GHAYAD S E COHEN P A 2010Recent Pat Anticancer Drug Discov2010,5,1:1
2A20/TNFAIP3, a new estrogen-regulated gene that confers tamoxifen resistance in breast cancer cells显示文摘Vendrell JA Ghayad S Ben-Larbi S 2007Oneogene2007,26,:1
3Endocrine resistance associated with activated ErbB system in breast cancer ceils is re-versed by inhibiting MAPK or P13 K/Akt signaling pathways 显示文摘Ghayad SE VendreU JA Ben Larbi S 2010lnt J Cancer2010,126,2:1
4Endocrine resistance associated with activated ErbB system in breast cancer cells is reversed by inhibiting MAPK or PI3K/Akt signaling pathways显示文摘Ghayad S E Vendrell J A Ben Larbi S 0,,02:1
5Effect of different modes of light curing and resin composites on micreleak - age of Class Ⅱ restora- tions-Part Ⅱ 显示文摘Hardan L S Amm E W Ghayad A 2009Odontostom - atol Trop2009,32,126:1
6A20/TNFAIP3,a newestrogen-regulated gene that confers tamoxifen resistance in breast cancer cells显示文摘Vendrell JA Ghayad S Ben-Larbi S 0,,32:1
7Endocrine resistance associated with activated ErbB system in breast cancer cells is reversed by inhibiting MAPK or PI3K/Akt signaling pathways 显示文摘Ghayad SE Vendrell JA Ben Larbi S 2010Int J Cancer2010,126,2:1
8Endocrine resist-ance associated with activated Eri)B system in breast cancer cells isreversed by inhibiting MAPK or PI3K/Akt signaling pathways显示文摘Ghayad S E Vendieli J A Ben Larbi S 2010Int J Cancer2010,126,2:1
9Endocrine resistance associated with activated ErbB system in breast cancer cells is reversed by inhibiting MAPK or PI3K/Akt signaling pathways显示文摘Ghayad S E Vendrell J A Larbi S B 2010Int J Cancer2010,126,2:1
10A20 / TNFAIP3 ,a new estrogen-regulated gene that confers tamoxifen resistance in breast cancer cells 显示文摘Vendrell JA Ghayad S Ben-Larbi S 2007Oneogene2007,26,32:1
11Endocrine resistance as- sociated with activated ErbB system in breast cancer cells is reversed by inhibiting MAPK or PBK/Akt signaling pathways显示文摘Ghayad S E Vendrell J A Larbi S B 2010Int J Canc- er2010,126,2:1
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