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| 1 | Microstructure-toughness-wear relationship of tetragonal zirconia ceramics 显示文摘 | Basu B Jozef V Blest O V | 2004 | Journal of the European Ceramic Society2004，24，6： | 1 |
| 2 | Brain and other natriuretic peptides: molecular aspects显示文摘 | Marc V Jozef B Marc G | 2004 | Eur J Heart Failure2004，6，： | 1 |
| 3 | Experimental and model- ing studies on the enantio-soparation of 3,5-dinitrobensoyl- (R), (S)-leucine by continuous liquid-liquid extraction in a cascade of centrifugal contactor separators 显示文摘 | Boelo S Jozef W Johannes V | 2010 | Chcm Eng Sei2010，65，15： | 1 |
| 4 | Computing the metric di- mension of graphs by genetic algorithms显示文摘 | JOZEF K VERA K V MIRJANA C | 2009 | Computational Opti- mization and Applications2009，44，2： | 1 |
| 5 | Brain and other natriuretic peptides:molecular aspects显示文摘 | MARC V JOZEF B MARC G | 2004 | Eur J Heart Failure2004，6，： | 1 |
| 6 | Brain and other natriuretic peptides:molecularaspects显示文摘 | Marc V Jozef B Marc G | 2004 | Eur J Heart Fail2004，6，3： | 1 |
| 7 | Brain and other natriuretic peptides : molecular aspects显示文摘 | Marc V Jozef B Marc G | 2004 | Eur J Heart Failure2004，6，： | 1 |
| 8 | Charaterization of the Bifunctional γ,-Glutamate-cysteine Ligase/Glutathion Synthetase ( GSH ) of Pasteurella multoxida显示文摘 | Bjom V Dark D V Jozef J | 2006 | The Journal of Biological Chemistry2006，，7： | 1 |
| 9 | MULAN: a java library for multi-label learning 显示文摘 | Grigorios T Eleftherios S Jozef V | 2011 | J Mach Learn Res2011，12，7： | 1 |
| 10 | Characterization of oxidation progress by chemiluminescence: A study of polyethylene with pro-oxidant additives显示文摘 | Marek Koutny Tereza Václavková Lyda Matisová-Rychlá Jozef Rychly | 2008 | Polymer Degradation and Stability2008，，8： | 1 |
| 11 | Low-cost implementation of distance maps for path planning using matrix quadtrees and octrees显示文摘 | V(o)r(o)s Jozef | | 0，，06： | 1 |
| 12 | Microstructure-toughness-wear relationship o{ tetragonal zirconia eeramies显示文摘 | Basu B Jozef V Biest O V | 2004 | J Eur Ceram Soc2004，24，6： | 1 |
| 13 | Adult stem cells in the treatment of acute myocardial infarction 显示文摘 | FAISAL S JOZEF B MARC V | 2011 | Catheter Cardiovasc Inter2011，77，1： | 1 |
| 14 | Parameter identification of Wiener systems with discontinuous nonlinearities显示文摘 | Jozef Vrs | 2001 | System and Control Letters2001，44，3： | 1 |
| 15 | Solubility products of titanium sulphide and carbo-sulphide in ultra-low carbon steels显示文摘 | Yang X Y Dirk V Jozef D | 1996 | ISU Int1996，36，10： | 1 |
| 16 | Time-Mean Structure of Axisymmetric Synthetic Jets显示文摘 | Václav Tesa? Jozef Kordík | 2010 | Sensors & Actuators: A Physical2010，，1： | 1 |
| 17 | Rilmenidine prevents blood pressure increase in rats with compromised nitric oxide production显示文摘AIM: To search tools of high blood pressure in the model of nitric oxide (NO)-defective hypertension, and the study focused on the effect of rilmenidine, agonist of imidazoline receptors, which was suggested to modulate central sympathetic outflow. METHODS: Three experimental groups, each consisting of 7 rats, were used: (I) rats with inhibition of NO synthase (NOS) by NG-nitro-L-arginine methyl ester (L-NAME) 40 mg·kg-1·d-1 for 4 weeks in drinking water, (II) rats with inhibited NOS as in group I , plus agonist of imidazoline receptors rilmenidine 3 mg·kg-1·d-1 for 4weeks by gavage, and(III) control rats. Systolic blood pressure wasmeasured weekly noninvasively. At the end of experiment aortic ring isometric tension was followed, NOS expression (aorta, left ventricle), and NOS activity (left ventricle and brain) were determined. RESULTS: In the group I systolic blood pressure in- creased significantly, aortic ring relaxation to acetylcholine was significantly attenuated. Rilmenidine administered simultaneously with L-NAME (group II) prevented the increase of blood pressure which did not differ significantly from control values; aortic ring relaxation to acetylcholine did not differ from control. No change in NOS expres- sion (aorta and left ventricle) was found in groups I and II. Significant decline in NOS activity (left ventricle and brain) wasfound in groups I and II. CONCLUSION: Rilmenidinehas aremarkable role inNO-defective hypertension, possibly by inhibiting central sympathetic outflow and by affecting receptors in vascular smooth muscle also. The prime cause of hypertension in this experimental model - the compromised production of NO due to inhibition of NOS - was not affected by rilmenidine. | Mria GEROV Jozef TRK Ol’ga PECHV Jana MATUKOV | 2004 | Acta Pharmacologica Sinica2004，25，12： | 0 |
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