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15篇 您的检索式:作者名="Jules T"
    题名 作者 年代 出处 被引量
1Geometric distortions in side-scan sonar images:a procedure for their estimation and correction显示文摘DANIEL T COBRA JULES S JAFFE 1992IEEE Journal of Occanic Engineering1992,17,3:1
2Mitchel,cynthia ernst,silvana cappi,william beasley,amy lau,yong joong kim,joon you meeting the challenges of internet-based clinical trials显示文摘Jules T 2004Applied Clinical Trials2004,13,6:1
3Holocene eli mate changes over the desert/loess transition of north centra China显示文摘XIAO Jule Nakamura T LU Huayu 2002Earth and Planetary Science Letters2002,197,:1
4Ho显示文摘XIAO Jule XU Qinghai Nakamura T 2004Quaternary Science Reviews2004,23,:1
5H olocene East Asi- an monsoonal precipitation pattern revealed by grain-size dis- tribution of core sediments of Daihai Lake in Inner Mongolia of north-central China显示文摘Peng Yanjia Xiao Jule Nakamura T 2005Earth and Planetary Science Letters2005,233,:1
6Physiologically based pharmacokinetic modeling of a ternary mixture of alkyl benzenes in rats and humans 显示文摘Robert T Ginette C Jules B 1997Toxicology and Applied Pharmacology1997,144,:1
7Modeling the mean residence time in a rotary dryer for various types of solids 显示文摘Jules T Pedro A Roman B 2010Drying Technology2010,28,10:1
8Geometric Distortion in sidescan sonar images:A Procedure for Their Estimation and Correction 显示文摘Deniel T Alan V Jules S 1992IEEE Journal of Oceanic Eegineering1992,3,17:1
9Effects of acetate, propionate and butyrate on the thermophilic anaerobic degradation of propionate by methanogenic sludge and defined cultures显示文摘Jules B L Katja C F Carla T M 1993Applied and Environmental Microbiology1993,59,4:1
10Bal- ance as bias: global warming andthe US prestige press 显示文摘MAXWELL T BOYKOFFA JULES M BOYKOFF 2004Global Environmental Change2004,,14:1
11Modelling of coagulant dosage in a water treatment plant显示文摘Gagnon C Grandjean B P A Jules T 1997Artificial Intelligence in Engineering1997,11,4:1
12Morphoscopic analysis of ex- perimentally produced bony wounds from low-velocity ballistic impact显示文摘Jules A Kieser Joy T 2011Forensic Sci Med Pat2011,7,4:1
13Theoretical study of the regioselectivity of the interaction of 3-methyl-4-pyrimidone and 1- methyl-2-pyrimidone with Lewis acids显示文摘Okuma E K Jules T M Lies B 2012J Phys Chem A2012,116,:1
14Diesel exhaust exposure favors T H 2 cell recruitment in nonatopic subjects by differentially regulating chemokine production显示文摘Ying Chang Stéphanie Sénéchal Patricia de Nadai Cécile Chenivesse Jules Gilet Han Vorng Benjamin Legendre André-Bernard Tonnel Beno?t Wallaert Philippe Lassalle Anne Tsicopoulos 2006The Journal of Allergy and Clinical Immunology2006,,2:1
15Spinal cord injury reprograms muscle fibroadipogenic progenitors to form heterotopic bones within muscles显示文摘The cells of origin of neurogenic heterotopic ossifications(NHOs), which develop frequently in the periarticular muscles following spinal cord injuries(SCIs) and traumatic brain injuries, remain unclear because skeletal muscle harbors two progenitor cell populations: satellite cells(SCs), which are myogenic, and fibroadipogenic progenitors(FAPs), which are mesenchymal. Lineage-tracing experiments using the Cre recombinase/Lox P system were performed in two mouse strains with the fluorescent protein Zs Green specifically expressed in either SCs or FAPs in skeletal muscles under the control of the Pax7 or Prrx1 gene promoter, respectively. These experiments demonstrate that following muscle injury, SCI causes the upregulation of PDGFRα expression on FAPs but not SCs and the failure of SCs to regenerate myofibers in the injured muscle, with reduced apoptosis and continued proliferation of muscle resident FAPs enabling their osteogenic differentiation into NHOs. No cells expressing Zs Green under the Prrx1 promoter were detected in the blood after injury, suggesting that the cells of origin of NHOs are locally derived from the injured muscle. We validated these findings using human NHO biopsies. PDGFRα+mesenchymal cells isolated from the muscle surrounding NHO biopsies could develop ectopic human bones when transplanted into immunocompromised mice, whereas CD56+myogenic cells had a much lower potential. Therefore, NHO is a pathology of the injured muscle in which SCI reprograms FAPs to undergo uncontrolled proliferation and differentiation into osteoblasts.Hsu-Wen Tseng Dorothée Girard Kylie A.Alexander Susan M.Millard Frédéric Torossian Adrienne Anginot Whitney Fleming Jules Gueguen Marie-Emmanuelle Goriot Denis Clay Beulah Jose Bianca Nowlan Allison R.Pettit Marjorie Salga François Genêt Marie-Caroline Le Bousse-Kerdilès Sébastien Banzet Jean-Pierre Lévesque 2022Bone Research2022,10,2:0
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