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    题名 作者 年代 出处 被引量
1The basics of epithelial-mesenchymal transition显示文摘Kalluri Raghu Weinberg Robert A 2009Journal of Clinical Investigation2009,,6:17
2An evolving function of DNA-containing exosomes in chemotherapy-induced immune response显示文摘Paul Kurywchak Raghu Kalluri 2017Cell Research2017,27,6:6
3Current state of renal transplant immunosuppression:Present and future显示文摘For kidney transplant recipients, immunosuppression commonly consists of combination treatment with a calcineurin inhibitor, an antiproliferative agent and a corticosteroid. Many medical centers use a sequential immunosuppression regimen where an induction agent, either an anti-thymocyte globulin or interleukin-2 receptor antibody, is given at the time of transplantation to prevent early acute rejection which is then followed by a triple immunosuppressive maintenance regimen. Very low rejection rates have been achieved at many transplant centers using combinations of these agents in a variety of protocols. Yet, a large number of recipients suffer chronic allograft injury and adverse events associated with drug therapy. Regimens designed to limit or eliminate calcineurin inhibitors and/or corticosteroid use are actively being pursued. An ideal immunosuppressive regimen limits toxicity and prolongs the functional life of the graft. This article contains a critical analysis of clinical data on currently available immuno-suppressive strategies and an overview of therapeutic moieties in development.Hari Varun Kalluri Karen L Hardinger 2012World Journal of Transplantation2012,2,4:5
4显示文摘Kalluri R 2003Nat Rev Cancer2003,3,6:1
5Tumstatin,the NCI domain of a3 chain of type Ⅳ collagen, is an endogenous inhibitor of pathological angiogenesis and suppresses tumor growth 显示文摘Hamano Y Kalluri R 2005Biochem Biophys Res Commun2005,333,2:1
6The basics of epithelial- mesenchymal transition 显示文摘Kalluri R Weinberg R A 2009J Clin Invest2009,119,8:1
7EMT:when epithelial cells decide to become mesenchymal-like cells显示文摘Kalluri R 2009J Clin Invest2009,119,6:1
8The epithelial mesenchymaltransition:new insights in sighaling,development and disease显示文摘Lee JM Dedhar S Kalluri R5 2006J Cell Biol2006,172,7:1
9Fibroblasts in cancer 显示文摘Kalluri R Zeisberg M 2006Nat Rev Cancer2006,6,5:1
10The basics of epithelial-mesenchymaltransition显示文摘Kalluri R Weinberg RA 2009J Clin Invest2009,119,6:1
11The role of epithelial-to-mesenchymal transition in renal fibrosis显示文摘Zeisberg M Kalluri R 0,,03:1
12Distortion-product source unmixing: A test of the two-mechanism model for DPOAE generation 显示文摘Kalluri R Shera C A 2001JAcoust Soc Am2001,109,:1
13Cancer without disease显示文摘Folkman J Kalluri R 2004Nature2004,427,6977:1
14Tumstatin, the NC1 domain of alpha3 chain of type Ⅳ collagen, is an endogenous inhibitor of pathological angiogenesis and suppresses tumor growth 显示文摘Hamano Y Kalluri R 2005Biochem Biophys Res Commun2005,333,2:1
15Epithelial-mesenchymal transition and its implications for fibrosis显示文摘Kalluri R Neilson EG 2003J Clin Ivest2003,112,12:1
16Early role of Fspl in epithelialmesenchymal transformation 显示文摘Okada H Danoff TM Kalluri R 1997Am J Physiol1997,273,42:1
17Genome-wide analysis of Aux/IAA and ARF gene families in Populus trichocarpa显示文摘Kalluri U C Difazio S P Brunner A M 2007BMC Plant Biology2007,7,:1
18The origin of fibroblasts and mechanism of cardiac fibrosis 显示文摘Krenning G Zeisberg E M Kalluri R 2010J Cell Physio12010,225,3:1
19Ion channels in mammalian vestibular afferents may set regularity of fir-ing显示文摘EATOCK RA XUE J KALLURI R 2008J Exp Biol2008,211,:1
20Basement membranes: structure, assembly and role in tumour angiogenesis 显示文摘Kalluri R 2003Nat Rev Cancer2003,3,6:1
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