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45篇 您的检索式:作者名="Keavney"
    题名 作者 年代 出处 被引量
1Large-scale evidence that the cardiotoxicity of smoking is not ignificantly modified by the apolipoprotein E2 /E3/E4 genotype显示文摘Keavney B Parish S Palmer A 2003Lancet2003,361,9355:1
2Fibfinogen and coronary heart disease:test of causality by'Mendelian randomization显示文摘Keavney B Danesh J Parish S 2006Int J Epidemiol2006,35,4:1
3Genetic mechanisms mediating atherosclerosis susceptibility at the chromosome 9p21 locus显示文摘Cunnington MS Keavney B 2011Curr Atheroscler Rep2011,13,3:1
4Large - scale evidence that the cardiotoxicity of smoking is not significantly modified by the apolipoprotein E2/E3/FA genotype 显示文摘Keavney B Parish S Palmer A 2003Lancet2003,361,9355:1
5Teacher Con- ceres and Teacher Anxiety: A Neglected Topic of Classroom Research 显示文摘KEAVNEY G SINCLAIR K E 1978Review of Educational Research1978,48,2:1
6Where Does the Spin Reside in Ferromagnetic Cu-doped ZnO? 显示文摘Keavney D J Buchholz D B Ma Q 2007Applied Physics Letters2007,91,01:1
7Genetic Association Study in Complex Disease显示文摘Keavney B 2000Human Hypertension2000,14,:1
8Fibrinogen and coronaryheart disease:test of causality by 'mendelian randomization' 显示文摘Keavney B Danesh J Parish S 2006lnt J Epidemio12006,35,4:1
9Large-scale test of hypothesised associations between the angiotensinconverting-enzyme insertion/deletion polymorphism and myocardial infarction in about 5000 cases and 6000 controls显示文摘Keavney B McKenzie C Parish S 0,,9202:1
10Genetic mechanisms mediating atherosclerosis susceptibility at the chromosome 9p21 locus 显示文摘Cunnington M S Keavney B 2011Curr Atheroscler Rep2011,13,3:1
11Prediction of patient responses to antihypertensive drugs using genetic polymorphisms: investigation of the renin-angiotensin system genes显示文摘DUDLEY C KEAVNEY B CASADEI B 1996J Hypertens1996,14,:1
12Genetic mechanisms mediating atherosclerosis susceptibility at the chromosome 9p21 locus 显示文摘Cunnington MS Keavney B 2011Curr Atheroscler Rep2011,13,3:1
13Large-scale evidence that the cardiotoxicity of smoking is not significantly modified by the apolipoprotein E2/E3/E4 genotype显示文摘Keavney B Parish S Palmer A 2003Lancet2003,361,7:1
14Ebstein anomaly associated with left ventricular noncompaction: An autosomal dominant condition that can be caused by mutations in MYH7显示文摘Alexa M.C. Vermeer Klaartje van Engelen Alex V. Postma Marieke J.H. Baars Imke Christiaans Simone De Haij Sabine Klaassen Barbara J.M. Mulder Bernard Keavney 2013Am. J. Med. Genet2013,,:1
15Plasma fibrinogen and fibrinogen geno-types in 4685 cases of myocardial infarction and in 6002 controls;test of cansality by endelian randomization 显示文摘YOUNGMAN LD KEAVNEY BD PALMER A 2000Circulation2000,102,:1
16Genetic Association studies in complex diseases 显示文摘Keavney B 2000J Hum Hypertens2000,14,6:1
17Fibrinogen and coronary heart disease: test of causality by 'Mendelian randomization ' 显示文摘Keavney B Danesh J Parish S 2006Int J Epidemiol2006,35,4:1
18Large-scale evidence that the cardiotoxicity of okmsis not significantly modified by the apolipoprotein, E3, E4 genotype 显示文摘Keavney B Pailsh S Palmer A 2003Lancet2003,361,9355:1
19Genetic association study in complex disease 显示文摘Keavney B 2000J Hum Hypertens2000,14,:1
20Evaluation oi the Angiotensinogen Locus in Human Essential Hypertension显示文摘Brand E Chatelain N Keavney B 1998Hypertension1998,31,3:1
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