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21篇 您的检索式:作者名="Krista S"
    题名 作者 年代 出处 被引量
1Evaluation of Sur- face Lignin on Cellulose Fibers with XPS显示文摘LEENA S J CAMPBELL J M KRISTA K 1999Applied Surface Science1999,14,:1
2Racial and ethnic disparity in participation in DNA collection at the atlanta site of the national birth defects prevention study显示文摘Crider Krista S Reefhuis 2006American Journal of Epidemiology2006,164,8:1
3Racial and ethnic disparity in participation in DNA collection at the atlanta site of the national birth defects prev ention study显示文摘Crider Krista S Reefhuis 0,,08:1
4Antibacterial Medication Use During Pregnancy and Risk of Birth Defects 显示文摘Krista S Crider Mario A Cleves Jennita Reefhuis 2009Arch PediatrAdolesc Med2009,163,:1
5Racial and ethnic disparity in participation in DNA collection at the Atlanta site of the national birth defects prevention study显示文摘Crider Krista S Reefhuis 2006Am J Epidemol2006,164,8:1
6Exceptional NegativeThermal Expansion in Iso-Reticular Metal-Organic Frameworks显示文摘David D Krista S W Donald E E ef al 2007Angewandte Chemie2007,129,:1
7Determination of olanzapine in a Postmortem case显示文摘Amanda J J Krista M S Heather N R 1998Journal of Analytical Toxicology1998,22,7:1
8Lactose modifications enhance its drug performance in the novel multiple dose Taifun DPI 显示文摘Pa ivi H Vesa- Pekka L Krista M Eero S Tapio L Petteri P Kristiina J 2002European Journal of Pharmaceutical Sciences2002,16,:1
9The selective dopamine D3 receptor antagonists SB-277011A and NGB 2904 and the putative partial D3 receptor agonist BP-897 attenuate methamphetamine-enhanced brain stimulation reward in rats 显示文摘Krista S Zheng XX Xiao QP 2008Psychopharmacology2008,196,:1
10Trisomies 13 and 18:Population prevalences,characteristics,and prenaal diagnosis,metropolitan atlanta,1994-2003显示文摘Krista S Richard S Janet D 2008J Clin Ultrasound2008,146,7:1
11Nuclear Factor-_K B activation in human testicular apoptosis显示文摘Virve P Laura S Krista E 2002American Journal of Pathology2002,160,1:1
12Glucocorticoid effects on changes in bone mineral density and cortical structure in childhood nephrotic syndrome显示文摘Anne Tsampalieros Pooja Gupta Michelle R Denburg Justine Shults Babette S Zemel Sogol Mostoufi‐Moab Rachel J Wetzsteon Rita M Herskovitz Krista M Whitehead Mary B Leonard 2013J Bone Miner Res2013,,3:1
13Racial and ethnic disparity in participation in DNA collection at the atlanta site of the national birth defects prevention study 显示文摘Crider Krista S Reefhuis 2006Am J Epidemiol2006,164,8:1
14CO2 adsorption in Y and X zeolites modified by alkali metal cation exchange 显示文摘Krista S W Morgan B A Douglas L M 2006Microporous and Mesoporous Materials2006,91,13:1
15Demonstrated brain insulin resistance in Alzheimer’s disease patients is associated with IGF-1 resistance, IRS-1 dysregulation, and cognitive decline显示文摘Talbot Konrad Wang Hoau-Yan Kazi Hala Han Li-Ying Bakshi Kalindi P Stucky Andres Fuino Robert L Kawaguchi Krista R Samoyedny Andrew J Wilson Robert S Arvanitakis Zoe Schneider Julie A Wolf Bryan A Bennett David A Trojanowski John Q Arnold 2012EN2012,,4:1
16Estradiol acts as a germ cell survival factor in the human testis in vitro显示文摘VIRVE P KRISTA E LAURA S 2000Clin Endocrinol Metab2000,85,:1
17Invasive pulmonaryaspergillosis显示文摘Krista S S Kern I MusicE 2002Respiratio2002,69,6:1
18Implications of new research and technologies for malolactic fermentation in wine显示文摘Krista M S Paul R G Vladimir J 2014Applied Microbiology and Biotechnology2014,98,19:1
19Racial and ethnic disparity in DNA eolleetion at the atlanta site of the national birth defects preverntion study显示文摘Crider Krista S Reefhuis 2006Am J Epidemiol2006,164,8:1
20Gene expression differences between stroke-associated and asymptomatic carotid plaques显示文摘Atherosclerotic carotid stenosis is an important risk factor for stroke. Carotid plaques(CPs) causing stroke may present a distinct type of molecular pathology compared with transient ischemic attack( TIA)-associated or asymptomatic plaques. We compared the gene expression profiles of CPs from stroke patients( n =12) and asymptomatic patients( n =9),both with similar risk factors and severity of carotid stenosis(>70%). Sixty probes showed over 1.5-fold expression difference at 5% false discovery rate. Functional clustering showed enrichment of genes in 51 GO categories and seven pathways, the most significant of which relate to extracellular-matrix interaction, PPAR gamma signaling, scavanger receptor activity, and lysosomal activity. Differential expression of ten genes was confirmed in an extended replication group(n=43), where the most significant expression differences were found in CD36( 2.1-fold change, p =0.005), CD163( 1.7-fold change, p =0.007) and FABP4(2.2-fold change, p=0.015). These include four genes not previously linked to plaque destabilization: GLUL(2.2-fold change,p=0.016), FUCA1(2.2-fold change, p=0.025), IL1RN(1.6-fold change, p=0.034), and S100A8(2.5-fold change, p=0.047). Strong correlations were found to plaque ulceration, plaque hemorrhage, and markers of apoptosis and proliferation(activated caspase 3, TUNEL, and Ki67). Protein expression of these genes was confirmed by immunohistochemistry and was found in the atheromatous areas of CPs critical for plaque destabilization. This study presents a comprehensive transcriptional analysis of stroke-associated CPs and demonstrates a significant transcriptome difference between stroke-associated and asymptomatic CPs. Followup studies on the identified genes are needed to define whether they could be used as biomarkers of symptomatic CPs or have a role in plaque destabilization.Jani Saksi Petra Ij s Krista Nuotio Riitta Sonninen Lauri Soinne Oili Salonen Eija Saimanen Jarno Tuimala Erno M.Lehtonen-Smeds Markku Kaste Petri T.Kovanen Perttu J.Lindsberg 2015世界最新医学信息文摘2015,15,5:0
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