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47篇 您的检索式:作者名="Krizaj"
    题名 作者 年代 出处 被引量
1Externally-induced meditative states: an exploratory fMRI study of architects' responses to contemplative architecture显示文摘Julio Bermudez David Krizaj David L. Lipschitz Charles Eiliott Bueler Jadwiga Rogowska Deborah Yurgelun-Todd Yoshio Nakamura 2017Frontiers of Architectural Research2017,6,2:2
2The dynamic range and domain-specific signals of intracellular calcium in photoreceptors显示文摘Szikra T Krizaj D 2006Neuroscience2006,,:1
3An instrumentation amplifier as a front-end for a four-electrode bioimpedanee measurement 显示文摘Zagar T Krizaj D 2007Physiol Meas2007,28,8:1
4Isolation and characterization of the Pseudobutyrivibrio xylanivorans Mz5T xylanase XynT-the first family 11 endoxylanase from rumen butyrivibrio-related bacteria显示文摘CEPELJNIK T KRIZAJ I MARINSEK L R 2004Enzyme Microb Technol2004,34,:1
5Molecular analysis of system N suggests novel physiological roles in ni- trogen metabolism and synaptic transmission显示文摘CHAUDHRY F A REIMER R J KRIZAJ D 1999Cell1999,99,7:1
6Restoration of enzymatic activity ina Ser-49 phospholipase A2 homologue decreases its Ca2+-independentmembrane damaging activity and increases its toxicity显示文摘PETAN T KRIZAJ I PUNGERCAR J 2007Biochemistry2007,46,12:1
7Neurotoxic phospholipases A2ammodytoxin and crotoxin bind to distinct high-affinity protein acceptors inTorpedo marmorata electric organ显示文摘KRIZAJ I FAURE G GUBENSEK F 1997Biochemistry1997,36,:1
8Coupled and uncoupled proton movement by amino acid transport sys- tem N显示文摘CHAUDHRY F A KRIZAJ D LARSSON P 2001EMBO J2001,20,24:1
9Understanding the molecular mechanism un-derlying the presynaptic toxicity of secreted phospholipases A2显示文摘PUNGERCAR J KRIZAJ I 2007Toxi-con2007,50,7:1
10Isolation and characterization of the Pseudobutyrivibrio xylanivorans MzST xylanase XynT-The first family 11 endoxylanase from rumen Butyrivibrio-related bacteria显示文摘CepeljnikT Krizaj I Marinsek-Logar R 2004Enzyme and Microbial Technology2004,34,:1
11Accuracy of DENTAPORT canal length determination with the impedance ratio method 显示文摘Jan J Krizaj D 2009Int Endod J2009,42,9:1
12A new analytical model for determination of breakdown voltage of RESURF structure显示文摘Krizaj D Charita G Amon S 0,,09:1
13Difference in specificity for the interactions of stefins A,B and D with cysteine proteinases显示文摘BRIGITA LENARCIC IGOR KRIZAJ PETRA ZUNEC 1996FEBS Lett1996,395,:1
14Spiral junction termination 显示文摘KRIZAJ D AMON S MINGUES C 1997IEEE Trans Elec Dev1997,44,11:1
15Dyslipidemia modulates Müller glial sensing and transduction of ambient information显示文摘Unesterified cholesterol controls the fluidity, permeability and electrical properties of eukaryotic cell membranes. Consequently, cholesterol levels in the retina and the brain are tightly regulated whereas depletion or oversupply caused by diet or heredity contribute to neurodegenerative diseases and vision loss. Astroglia play a central role in the biosynthesis, uptake and transport of cholesterol and also drive inflammatory signaling under hypercholesterolemic conditions associated with high-fat diet(diabetes) and neurodegenerative disease. A growing body of evidence shows that unesterified membrane cholesterol modulates the ability of glia to sense and transduce ambient information. Cholesterol-dependence of Müller glia-which function as retinal sentinels for metabolic, mechanical, osmotic and inflammatory signals-is mediated in part by transient receptor potential V4(TRPV4) channels. Cholesterol supplementation facilitates, whereas depletion suppresses, TRPV4-mediated transduction of temperature and lipid agonists in Müller cells. Acute effects of cholesterol supplementation/depletion on plasma membrane ion channels and calcium homeostasis differ markedly from the effects of chronic dyslipidemia, possibly due to differential modulation of modality-dependent energy barriers associated with the functionality of polymodal channels embedded within lipid rafts. Understanding of cholesterol-dependence of TRP channels is thus providing insight into dyslipidemic pathologies associated with diabetic retinopathy, glaucoma and macular degeneration.Monika Lakk Felix Vazquez-Chona Oleg Yarishkin David Krizaj 2018Neural Regeneration Research2018,13,2:1
16Molecular analysis of system N suggests novel physiological roles in nitrogen metabolism and synaptic transmission farrukh A chaudhry 显示文摘CHAUDHRY F A REIMER R J KRIZAJ D 1999Cell1999,99,:1
17Roles of secreted phospholipases A2 in the mammalian immune system 显示文摘Krizaj I 2014Protein Pept Lett2014,21,12:1
18Improved purification of steroid 1, 2 dehydrogenase from Nocardia opaca and partial characterization of its cloned gene sequence显示文摘Drobnic K Krizaj I Gubensek F 1993Biochemistry and Biophysiology Research Communication1993,190,2:1
19Difference in specifici-ty for the interactions of stefins A,B and D with cysteine proteinases显示文摘LENARCIC B KRIZAJ I ZUNEC P 1996FEBS Lett1996,395,23:1
20Restoration of enzymatic activity in a Ser-49 phospholipase A2 homologue decreases its Ca^2+-independent membrane-damaging activity and increases its toxicity 显示文摘Petan T Krizaj I Pungercar J 2007Biochemistry2007,46,12:1
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