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21篇 您的检索式:作者名="LICHOTA"
    题名 作者 年代 出处 被引量
1Heterogenous distribution of ferroportin-containing neurons in mouse brain 显示文摘Boserup MW Lichota J Haile D 2011Biometals2011,24,:1
2Differential chromatin association and nucleosome binding of the maize HMGA,HMGB,and SSRP1 proteins显示文摘LICHOTA J GRASSER K D 2001Biochemistry2001,40,:1
3Endodontic treatment of a maxillary canine with type 3 dens invaginatus and large periradicu- lar lesion : a case report 显示文摘Lichota D Lipski M Woz'niak K 2008J Endod2008,34,6:1
4The arabidopsis genome en-codes strutturally and functionally diverse HMGB-type proteins 显示文摘Grasser M Lentz A Lichota J 2006J Mol Biol2006,358,3:1
5Anterior knee pain:symptom or syndrome? 显示文摘Lichota DK 2003Currents Womens Health Reo2003,3,1:1
6The Arabidopsis genome en- codes structurally and functionally diverse HMGB-type proteins 显示文摘Grasser M Lentz A Lichota J 2006J Mol Siol2006,358,3:1
7Anterior knee pain:symptom or syndrome?显示文摘Lichota DK 2003Current women''s health reports2003,3,1:1
8Anterior knee pain:symptom or syndrome?显示文摘Lichota DK 2003Currents Womens Health Rep2003,3,1:1
9Anterior knee pain:symptom or syndrome?显示文摘Lichota DK 2003Curr Womens Health Rep2003,3,1:1
10Erasure of CpG methylation in Arabidopsis alters patterns of histone H3 methylation in heterochromatin显示文摘Tariq M Saze H Probst A.V Lichota J Habu Y and Paszkowski J 0,,:1
11The arabidopsis genome encodesstrutturally and functionally diverse HMGB-type proteins显示文摘Grasser M Lentz A Lichota J 2006J Mol Biol2006,358,3:1
12Differential chromatin association and nucleosome binding of the maize HMGA, HMGB, and SSRP1 proteins 显示文摘Lichota J Grasser K D 2001Biochemistry2001,40,26:1
13Response of Human Dental Pulp Capped with Biodentine and Mineral Trioxide Aggregate显示文摘Alicja Nowicka Mariusz Lipski Miros?aw Parafiniuk Katarzyna Sporniak-Tutak Damian Lichota Anita Kosierkiewicz Wojciech Kaczmarek Jadwiga Buczkowska-Radlińska 2013Journal of Endodontics2013,,6:1
14The Arabidopsis genome encodes structurally and functionally diverse HMGB-type proteins 显示文摘Grasser M Lentz A Lichota J 2006J Mol Biol2006,358,3:1
15Root surface temperature rise of mandibular first molar during root canal filling with high-temperature thermoplasticized Gutta-Percha in the dog 显示文摘Lipski M Wozniak K Lichota D 2011Pol J Vet Sci2011,14,4:1
16Hetemgenous distribution of ferroportin-containing neurons in mouse brain 显示文摘Boserup MW Lichota J Halle D 2011Biometals2011,24,2:1
17Gene delivery by pullulan derivatives in brain capillary endothelial ceils for protein secretion 显示文摘THOMSEN L B LICHOTA J KIM K S 2011J Conlxol Release2011,151,1:1
18Differential chromatin association and nucleosome binding of the maize HMGA,HMGB,and SSRPl proteins显示文摘LICHOTA J GRASSER K D 0,,:1
19Brain delivery systems via mechanism independent of receptor-mediated endocytosis and adsorptive-mediated endocytosis显示文摘Thomsen L B Lichota J Eskehave T N 2012Curr Pharm Biotechnol2012,13,12:1
20Rethinking neurodegenerative diseases:neurometabolic concept linking lipid oxidation to diseases in the central nervous system显示文摘Currently,there is a lack of effective medicines capable of halting or reve rsing the progression of neurodegenerative disorde rs,including amyotrophic lateral sclerosis,Parkinson s disease,multiple sclerosis,or Alzheimer s disease.Given the unmet medical need,it is necessary to reevaluate the existing para digms of how to to rget these diseases.When considering neurodegenerative diseases from a systemic neurometabolic perspective,it becomes possible to explain the shared pathological features.This innovative approach presented in this paper draws upon exte nsive research conducted by the authors and researchers worldwide.In this review,we highlight the importance of metabolic mitochondrial dysfunction in the context of neurodegenerative diseases.We provide an overview of the risk factors associated with developing neurodegenerative disorders,including genetic,epigenetic,and environmental fa ctors.Additionally,we examine pathological mechanisms implicated in these diseases such as oxidative stress,accumulation of misfolded proteins,inflammation,demyelination,death of neurons,insulin resistance,dysbiosis,and neurotransmitter disturbances.Finally,we outline a proposal for the restoration of mitochondrial metabolism,a crucial aspect that may hold the key to facilitating curative therapeutic interventions for neurodegenerative disorders in forthcoming advancements.Steinunn Sara Helgudóttir Anne Skøttrup Mørkholt Jacek Lichota Preben Bruun-Nyzell Mads Christian Andersen Nanna Marie Juhl Kristensen Amanda Krøger Johansen Mikela Reinholdt Zinn Hulda Maria Jensdóttir John Dirk Vestergaard Nieland 2024Neural Regeneration Research2024,19,7:0
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