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17篇 您的检索式：作者名="Lesina"

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		题名	作者	年代	出处	被引量
	1	IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality显示文摘	Zhang Hong Neuh?fer Patrick Song Liang Rabe Bj?rn Lesina Marina Kurkowski Magdalena U Treiber Matthias Wartmann Thomas Regnér Sara Thorlacius Henrik Saur Dieter Weirich Gregor Yoshimura Akihiko Halangk Walter Mizgerd Joseph P Schmid Roland	2013	Journal of Clinical Investigation2013，，3：	2
	2	Pancreas-specif- ic RelA/p65 truncation increases susceptibility of acini to inflammation-associated cell death following cerulein pancreatitis显示文摘	Algal H Treibei M Lesina M	2007	J Clin Invest2007，117，6：	1
	3	Persistent mani-festations of cerebrocardiac syndrome in the immediate post-operative period(a clinical observation)显示文摘	Lesina SS Kondrat'Ev AN Kozliakov AV		0，，：	1
	4	Persistent manifestations of cerebreeardiac syndrome in the immediate post-operative period (a clini- cal observation ) 显示文摘	Lesina SS Kondrat Ev AN Kozliakov AV	2007	Anesteziol Reanlmato12007，42，3：	1
	5	STAT3/Socs3 activation by IL-6 transsignaling promotes progression of pancreatic intraepithelial neoplasia and development of pancreatic cancer显示文摘	Lesina M Kurkowski MU Ludes K	2011	Cancer Cell2011，19，4：	1
	6	Pancreas-specific RelA/ p65 truncation increases susceptibility of acini to inflammation-associated cell death following cerulein pancreatitis显示文摘	Algul H Trelber M Lesina M	2007	J Clin Invest2007，117，6：	1
	7	Stat3/Socs3 acti-vation by IL-6 transsignaling promotes progression of pancre-atic intraepithelial neoplasia and development of pancreaticcancer显示文摘	Lesina M I Kurkowski M U Ludcs K	2011	Cancer Cell2011，19，45：	1
	8	Mechanisms of disease: chronic inflammation and cancer in the pancreas-a potential role for pancreatic stellate cells? 显示文摘	Algul H Treiber M Lesina M	2007	Nat Clin Pract Gastroenterol Hepatol2007，4，8：	1
	9	Parallel implementation of a 3D subgridding FDTD algorithm for large simulations显示文摘	VACCARI A CALA' LESINA A CRISTOFORETTI L	2011	Progress in Electromagnetics Research (PIER)2011，120，：	1
	10	Stat3/Socs3 activation by IL-6 transsignaling promotes progression of pancreatic intraepi- thelial neoplasia and development of pancreatic cancer 显示文摘	Lesina M Kurkowski MU Ludes K	2011	Canc- er Cell2011，19，4：	1
	11	Pancreas-specific Rel A/ p65 truncation increases susceptibility of acini to inflammation- associated cell death following cerulein pancreatitis显示文摘	Algul H Trelber M Lesina M	2007	J Clin Invest2007，117，6：	1
	12	Pancreas-specific Re- 1A/p65 truncation increases susceptibility of acini to inflammation-associated cell death following cerulein pancreatitis 显示文摘	Algul H Treiber M Lesina M	2007	J Clin Invest2007，117，6：	1
	13	Interleukin-6 in inflammatory and malignant diseases of the pancreas显示文摘	Marina Lesina Sonja Maria W?rmann Patrick Neuh?fer Liang Song Hana Algül	2014	Seminars in Immunology2014，，：	1
	14	Persistent manifestations of cerebrocardiac syndrome in the immediate postoperative period (a clini- cal observation)显示文摘	Lesina S S Kondrat'ev A N Kozliakov A V	2012	Anesteziologiia i reanimatologiia2012，，3：	1
	15	Stat3/Socs3 ac-tivation by IL-6 transsignaling promotes progression ofpancreatic intraepithelial neoplasia and development ofpancreatic cancer显示文摘	Lesina M Kurkowski MU Ludes K	2011	Cancer Cell2011，19，4：	1
	16	Stat3/Socs3 activation by IL-6 transsignaling promotes progression of pancreatic in- traepithelial neoplasia and development of pancreatic cancer显示文摘	Lesina M Kurkowski MU Ludes K	2011	Cancer Cell2011，19，4：	1
	17	Combating pancreatic cancer during its Rip Van Winkle sleep显示文摘In a recent paper published in Cell,Ruscetti et al.showed that therapy-induced senescence in pancreatic ductal adenocarcinoma(PDAC)rewires vascular network within the cancer,which in turn makes tumor susceptible to chemotherapy.Moreover,senescence-associated secretory phenotypedependent activation of cancer vasculature increases T cell invasion into PDAC structures,thereby allowing better outcome of immunotherapy.	Kivanc Gorgülü Marina Lesina Hana Algul	2020	Cell Research2020，30，8：	0

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