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    题名 作者 年代 出处 被引量
1Intracytoplasmic sperm injection may increase the risk of imprinting defects显示文摘Cox GF Btirger J Lip V 2002Am J Hum Genet2002,71,1:1
2Effectiveness of multiplex ligation-dependent probe amplification assay used for detecting deletion of Prader-Willi syndrome 显示文摘Shao H Lip V Wu B L 2005Beijing Da Xue Xue Bao2005,37,1:1
3Intracytoplasmic sperm injection may increase the risk of imprinting defects显示文摘Cox GF Burger J Lip V 0,,01:1
4Intracytoplasmic sperm injection may increase the risk of imprinting defects 显示文摘Cox GF Burger J Lip V 2002Am J Hum Genet2002,71,1:1
5Markers of oxidative stress in exhaled breath of subjects with lung cancer 显示文摘Chan liP Tran V Lewis C 2008Respimlogy2008,13,:1
6Idiopathic orbital inflammation syndrome with retro-orbital involvement :a retrospective study of eight pa- tients显示文摘Li Y Lip G Chong V 2013PLoS One2013,8,57:1
7Podoplanin Immunopositive lymphatic vessels at the implant interface in a rat model of osteoporotic fractures 显示文摘Lips KS Kauschke V Hartmann S 2013PLoS One2013,8,77:1
8Fibrinolytic function and atrial fibrillation显示文摘Marin F Roldan V Lip GY 2003Thromb Res2003,109,:1
9Hypertension to heart failure: a pathophysiological spectrum relating blood pressure, drug treatments and stroke 显示文摘Subramaniam V Lip GY 2009Rev Cardiovasc Ther2009,7,70:1
10Novel concepts of statin therapy for cardiovascular risk reduction in hypertension 显示文摘VARUGHESE G I PATEL J V LIP G Y 2006Curr Pharm Des2006,12,13:1
11Intracytoplasmic sperm injection may increase the risk of imprinting defects显示文摘Cox GF Burger J Lip V 2002Am J Hum Genet2002,71,:1
12Fibrinolytic function and Atrial Fibrillation显示文摘Marin F Roldan V Lip GY 2003Thromb Res2003,109,15:1
13Solubie Eselectin in cardio-vascular disease and its risk factors:a review of the literature 显示文摘Roldan V Marin F Lip G Y 2003Thromb Haemost2003,90,7:1
14Drug Fever显示文摘Lip sky B A Hirchmann J V 1981JAMA1981,245,8:1
15Reactive oxygen species production by circulating monocytes : insights from pathophysiology to clinical hypertension 显示文摘Lira H S Patel J V Lip G Y 2006J Hum Hypertens2006,20,5:1
16lntraeytoplasmic sperm injection may increase the risk of imprinting defects显示文摘Coxa F Burger J Lip V 2002Am J Hum Gen- et2002,71,1:1
17Inhibitory activity of oxyresveratrol on wild-type and drug-resistant varicella-zoster virus replication in vitro显示文摘SASIVIMOLPHAN P LIP/PUN V LIKHITWlTAYAWLr/D K 2009Antiviral Research2009,84,1:1
18Quality of life in patientswith osteoporosis 显示文摘Lips P Natasja M Schoor V et-al 2005Osteoporosis Int2005,16,5:1
19Intracytoplusmic sperm injection may increase the risk of imprinting defects显示文摘 Burger J Lip V 2002Am J Hum Genet2002,71,1:1
20CD36 expression and lipid metabolism following an oral glucose challenge in South Asians显示文摘AIM:To investigate lipid metabolism and the relationship with monocyte expression of the fatty acid translocase CD36 in South Asians.METHODS:An observational study of South Asians whom as an ethnic group have-a higher risk of developing diabetes.The susceptibility to diabetes is coupled with an earlier and more rapid progression of micro-,and macro-vascular complications.Twentynine healthy South Asian participants [mean age 34.6(8.9) years,76.2% male,mean body-mass index 25.0(5.2) kg/m2] were recruited from an urban residential area of central Birmingham(United Kingdom).The main outcomes measured were post prandial(30 min) and post absorptive(120 min) changes from fasting(0 min) in circulating lipoproteins,lipds and hormones,andmonocyte expression of CD36 post injection of a 75 g oral glucose challenge.The inducements of variations of monocyte CD36 expression were analysed.RESULTS:Our results showed evident changes in monocyte CD36 expression following the glucose challenge(P < 0.001).Non-esterified fatty acids(NEFA) levels decreased progressively during the challenge(P < 0.001),in contrast to increased cholesterol(but not triglyceride) concentrations within very low density lipoprotein(VLDL) and low density lipoprotein subfractions(P < 0.01).Levels of,glucose,serum triglycerides and high density lipoprotein cholesterol remained largely unchanged.Variations of monocyte CD36 were negatively(r =-0.47,P = 0.04) associated to fat from the diet and positively to carbohydrate from the diet(r = 0.65,P < 0.001).CONCLUSION:These data suggest that the initiation of VLDL genesis follows the consumption of glucose within this population,inferring that the sequestration of NEFA from these particles happens due to the increased availability of CD36 receptors.While these are preliminary results,it would appear that lifestyle exposures have a role in moderating the expression of CD36.Jeetesh V Patel Amitava Banerjee Silvia Montoro-Garcia Eduard Shantsila Mushfique Alam Paul Flinders Kathleen AL Houlton Elizabeth A Hughes Gregory YH Lip Paramjit S Gill 2015World Journal of Diabetes2015,6,7:1
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