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| 1 | Intracellular calcium sig- nals and control of cell proliferation: how many mechanisms? 显示文摘 | Munaron L Antoniotti S Lovisolo D | 2004 | J Cell Mol Med2004，8，2： | 1 |
| 2 | Calcium signalling and control of cell proliferation by tyrosine kinase receptors (review) 显示文摘 | Munaron L | 2002 | Int J Mol Med2002，10，6： | 1 |
| 3 | Hydrogen sulfide and endothelial dysfunction:relationship with nitric oxide显示文摘 | Altaany Z Moccia F Munaron L | 2014 | Curr Med Chem2014，21，32： | 1 |
| 4 | Intracellular calcium signals and control of cell proliferation:how many mechanisms显示文摘 | ANTONIOTTIS LOVISOLOD | 2004 | J Cell Mol Med2004，8，2： | 1 |
| 5 | Activation of functional oxytocin receptors stimulates cell proliferation in human trophoblast and chorioearcinoma cell lines显示文摘 | Cassoni P Sapino A Munaron L | 2001 | Endocrinology2001，142，3： | 1 |
| 6 | Endothelial calcium machinery and angiogenesis : understanding physiology to interfere with pathology 显示文摘 | MUNARON L FIORIO PLA A | 2009 | Curr Med Chem2009，16，35： | 1 |
| 7 | Hydrogen sulfide as a regulator of calcium channels显示文摘 | Munaron L Avanzato D Moccia F | 2013 | Cell Calcium2013，53，2： | 1 |
| 8 | Endothelial calcium machinery and angiogen esis: understanding physiology to interfere with pathology 显示文摘 | Munaron L Fiorio Pla A | 2009 | Curr Med Chem2009，16，35： | 1 |
| 9 | The role of ion channels and transporters in cell proliferation and cancer显示文摘 | Becchetti A Munaron L Arcangeli A | 2013 | Front Physiol2013，4，： | 1 |
| 10 | Hydrogen sulfide as a regulator of calcium channels显示文摘 | Munaron L Avanzato D Moccia F | 2013 | Cell Calcium2013，53，2： | 1 |
| 11 | Cytosolic calcium microdomains by arachidonic acid and nitric oxide in endothelial cells显示文摘 | Tomatis C Fiorio Pla A Munaron L | 2007 | Cell Calcium2007，41，： | 1 |
| 12 | The role of ion channels and transporters in cell proliferation and cancer显示文摘 | Becchetti A Munaron L Arcangeli A | 2013 | Front Physiol2013，4312，： | 1 |
| 13 | Regulation of noncapacitative calcium entry by arachidonic acid and nitric oxide in endothelial cells显示文摘 | Mottola A Antoniotti S Lovisolo D Munaron L | 2005 | Faseb J2005，19，： | 1 |
| 14 | Organic UV filter concentrationsin marine mussels from French coastal regions显示文摘 | Bachelot M Li Z Munaron D | 2012 | Sci Total Environ2012，420，： | 1 |
| 15 | Hydrogen sulfide as a regulator of calcium channels 显示文摘 | MUNARON L AVANZATO D MOCCIA F | 2013 | Cell Calcium2013，53，2： | 1 |
| 16 | Activation of functional oxytocin receptors stimulates cell proliferation in human trophoblast and choriocarcinoma cell lines显示文摘 | Sapino A Munaron L | 2001 | Endocrinology2001，142，3： | 1 |
| 17 | Organic UV filter concentrations in marine mussels from French coastal regions显示文摘 | Bachelot M Li Z Munaron D | 2012 | Science of the Total Environment2012，420，6： | 1 |
| 18 | Shuffling the cards in signal transduction: Calcium, arachidonic acid and mechanosensitivity显示文摘Cell signaling is a very complex network of biochemical reactions triggered by a huge number of stimuli coming from the external medium. The function of any single signaling component depends not only on its own structure but also on its connections with other biomolecules. During prokaryotic-eukaryotic transition, the rearrangement of cell organization in terms of diffusional compartmentalization exerts a deep change in cell signaling functional potentiality. In this review I briefly introduce an intriguing ancient relationship between pathways involved in cell responses to chemical agonists (growth factors, nutrients, hormones) as well as to mechanical forces (stretch, osmotic changes). Some biomolecules (ion channels and enzymes) act as 'hubs', thanks to their ability to be directly or indirectly chemically/mechanically co-regulated. In particular calcium signaling machinery and arachidonic acid metabolism are very ancient networks, already present before eukaryotic appearance. A number of molecular 'hubs', including phospholipase A2 and some calcium channels, appear tightly interconnected in a cross regulation leading to the cellular response to chemical and mechanical stimulations. | Luca Munaron | 2011 | World Journal of Biological Chemistry2011，2，4： | 0 |
| 19 | Multilevel complexity of calcium signaling:Modeling angiogenesis显示文摘Intracellular calcium signaling is a universal,evolutionary conserved and versatile regulator of cell biochemistry.The complexity of calcium signaling and related cell machinery can be investigated by the use of experimental strategies,as well as by computational approaches.Vascular endothelium is a fascinating model to study the specific properties and roles of calcium signals at multiple biological levels.During the past 20 years,live cell imaging,patch clamp and other techniques have allowed us to detect and interfere with calcium signaling in endothelial cells(ECs),providing a huge amount of information on the regulation of vascularization(angiogenesis) in normal and tumoral tissues.These data range from the spatiotemporal dynamics of calcium within different cell microcompartments to those in entire multicellular and organized EC networks.Beside experimental strategies,in silico endothelial models,specifically designed for simulating calcium signaling,are contributing to our knowledge of vascular physiol-ogy and pathology.They help to investigate and predict the quantitative features of proangiogenic events moving through subcellular,cellular and supracellular levels.This review focuses on some recent developments of computational approaches for proangiogenic endothelial calcium signaling.In particular,we discuss the creation of hybrid simulation environments,which combine and integrate discrete Cellular Potts Models.They are able to capture the phenomenological mechanisms of cell morphological reorganization,migration,and intercellular adhesion,with single-cell spatiotemporal models,based on reaction-diffusion equations that describe the agonist-induced intracellular calcium events. | Luca Munaron Marco Scianna | 2012 | World Journal of Biological Chemistry2012，3，6： | 0 |
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