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| 1 | 抗稻瘿蚊品种多抗1的抗性遗传分析及抗性基因定位显示文摘稻瘿蚊是亚洲稻区主要害虫,采用抗虫品种进行防治是最理想的方法。1993~1995年,广东省农科院与国际水稻研究所有关专家紧密合作,对能抗华南4个稻瘿蚊生物型的品种多抗1作进一步抗性遗传分析,确认多抗1对中国稻瘿蚊生物型1和4的抗性受显性单基因控制,这个基因暂定名为GM—6(t)。以多抗1×丰银占1组合的F3代160个家系作基因标记,据DNA库分离个体分析(BSA)原理,用随机扩增多态性DNA(RAPD)标记物OPM6(1.4kb),首次成功地标记了这个抗性基因。随后多态性扩增产物经^(32)p标记,用作探针,检测另一个参考作图群体IR64×Azucena,将这个抗性基因定位在水稻第4条染色体上,位于RG214和RG163两个DNA限制性片段长度多态性(RFLP)标记之间。应用这些分子标记辅助选择有可能不必通过稻瘿蚊的直接筛选,快速准确地选育抗稻瘿蚊品种或进行抗性基因累加。 | 谭玉娟 张扬 黄炳超 徐炎康 赵丽霞 S.K.Katiyar N.Huang G.S.Khush J.Bennett | 1996 | 植物保护学报1996，23，4： | 17 |
| 2 | 查看详情显示文摘 | F.Zhang N.Huang P.Yang | | 0，，： | 1 |
| 3 | Vertebrate cardiac regeneration:evolutionary and developmental perspectives显示文摘Cardiac regeneration is an ancestral trait in vertebrates that is lost both as more recent vertebrate lineages evolved to adapt to new environments and selective pressures,and as members of certain species developmentally progress towards their adult forms.While higher vertebrates like humans and rodents resolve cardiac injury with permanent fibrosis and loss of cardiac output as adults,neonates of these same species can fully regenerate heart structure and function after injury–as can adult lower vertebrates like many teleost fish and urodele amphibians.Recent research has elucidated several broad factors hypothesized to contribute to this loss of cardiac regenerative potential both evolutionarily and developmentally:an oxygen-rich environment,vertebrate thermogenesis,a complex adaptive immune system,and cancer risk trade-offs.In this review,we discuss the evidence for these hypotheses as well as the cellular participators and molecular regulators by which they act to govern heart regeneration in vertebrates. | Stephen Cutie Guo N.Huang | 2021 | Cell Regeneration2021，10，1： | 1 |
| 4 | Mending a broken heart with novel cardiogenic small molecules显示文摘Adult mammalian cardiomyocytes are unable to proliferate to regenerate lost tissue after heart injury.Du et al.,reporting in Cell Stem Cell,employ a FUCCI-and MADM-based system to screen for small molecules combinations that produced a collaborative effect on cardiomyocyte cycling and cytokinesis.The authors generate a cocktail of five small molecules that increase cardiomyocyte proliferation and regeneration in vitro and in vivo with high efficiency,and explore its potential in cardiac regenerative repair after myocardial infarction through a new potential pathway for cardiomyocyte cell-cycle re-entry. | Nevan Powers Guo N.Huang | 2022 | Cell Regeneration2022，11，1： | 0 |
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