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21篇 您的检索式:作者名="NANNENGA B"
    题名 作者 年代 出处 被引量
1Inactivation of the Wipl phosphatase inhibits mammary tumorigenesis through p38 MARK- mediated activation of the p16 (Ink4a) -p19 (Aft) pathway 显示文摘Bulavin DV Phillips C Nannenga B 2004Nat Genet2004,36,4:1
2PPM1D dephosphorylates Chkl and p53 and abrogates cell cycle checkpoints显示文摘Lu X Nannenga B Donehower LA 2005Genes Dev2005,19,10:1
3Augmented cancer resistance and DNA damage response phenotypes in PPM1D null mice 显示文摘NANNENGA B LU X DUMBLE M 2006Mol Carcinog2006,45,8:1
4Mice deficient for the wild-type p53-induced phosphatase gene (Wipl) exhibit defects in rep roductiveor-gans, immune function, and cell cycle control 显示文摘Choi J Nannenga B Demidov ON 2002Mol Cell Biol2002,22,4:1
5Inactivation of the Wipl phosphatase inhibits mammary tumorigenesis through p38 MAPK-mediated activation of the p16 (Ink4a)-pl9 (Aft) pathway 显示文摘Bulavin DV Phillips C Nannenga B 2004Nat Genet2004,36,4:1
6Inactivation of the Wipl phosphatase inhibits mammary tumorigenesis through p38 MAPK-mediated activation of the p16 (Ink4a)-p19 (Aft) pathway显示文摘Bulavin DV Phillips C Nannenga B 2004Nat Genet2004,36,4:1
7Mice deficient for the wild-type p53-induced phosphatase gene (Wipl) exhibit defects in reproductive organs, immune function, and cell cycle control 显示文摘Choi J Nannenga B Demidov ON 2002Mol CellBiol2002,22,4:1
8Inactivation of the Wipl phosphatase inhibits mammary tumorigenesis through p38 MAPK -mediated activation of the p16 (Ink4a)-p19 (Aft) pathway 显示文摘Bulavin DV Phillips C Nannenga B 2004Nat Genet2004,36,4:1
9The p53 - induced oncogenic phosphatase PPM1D interacts with uracil DNA glycosylase and suppresses base excision repair 显示文摘Lu X Bocangel D Nannenga B 2004Mol Cell2004,15,4:1
10Inactivation of the Wipl phosphatase inhibits mammary tumorigenesis through p38 MAPK - mediated activation of the p16 (Ink4a) - pl9(Arf) pathway显示文摘Bulavin DV Phillips C Nannenga B 2004Nat Genet2004,36,4:1
11Inactivation of the Wipl phosphatase inhibits mammary tumorigenesis through p38 MAPK - mediated activation of the p16^Ink4a - p19^Arf pathway显示文摘Bulavin DV Phillips C Nannenga B 2004Nat Genet2004,36,4:1
12Mice deficient for the wild - type p53 - induced phosphatase gene ( Wipl ) exhibit defects in reproductiveor - gans,immune function, and cell cycle control 显示文摘Choi J Nannenga B Demidov ON 2002Mol Cell Bid2002,22,4:1
13Mice deficient for the wild-type p53-induced phosphatase gene (Wipl) exhibit defects in reproductive organs, immune function, and cell cycle control 显示文摘Choi J Nannenga B Demidov ON 2002Mol Cell Biol2002,22,4:1
14Inactivation of the Wipl phosphatase inhibits mammary tumorigenesis through p38 MAPK-mediated activation of the p16(Ink4a)-p19(Arf) pathway显示文摘Bulavin Dr Phillips C Nannenga B 2004Nat Genet2004,36,4:1
15PPM1D dephos- phorylates Chkl and p53 and abrogates cell cycle check- points显示文摘Lu X Nannenga B Donehower LA 2005Genes Dev2005,19,10:1
16Inactivation of the Wiplphosphatase inhibits mammary tumorigenesis through p38 MAPK-mediated activation of the p16(Ink4a)-p19(Arf)pathway显示文摘Bulavin DV Phillips C Nannenga B 2004NatGenet2004,36,4:1
17PPM1D dephosphorylates Chk1and p53 and abrogates cell cycle check-points显示文摘Lu X Nannenga B Donehower LA 2005Genes Dev2005,19,10:1
18PPM1D dephosphorylates Chkl and p53 and abrogates cell cycle checkpoints 显示文摘Lu X Nannenga B Donehower L A 2005Genes Dev2005,19,10:1
19Mice deficient for the Wild-type p53-induced phosphatase gene ( Wipl ) exhibit defects in reproductive organs, immune function, and cell cycle control显示文摘Choi J Nannenga B Demidov O D N 2002Mol Cell Biol2002,22,4:1
20PPM 1 D dephosphorylates Chk 1 and p53 and abrogates cell cycle checkpoints 显示文摘Lu X Nannenga B Donehower LA 2005Genes Development2005,19,10:1
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