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37篇 您的检索式:作者名="Paciga M"
    题名 作者 年代 出处 被引量
1AKT1/PKBalpha kinase is frequently elevated in human cancers and its constitutive activation is required for oneogenic transformation in NIH3T3 cells显示文摘Sun M Wang G Paciga JE 2001Am J Pathol2001,159,2:1
2AKT1/PKB alpha kinase is frequently elevated in human cancers and its constitutive activation is required for oncogenic transformation in NIH3T3 cells显示文摘Sun M Wang G Paciga JE 2001Am J Pathol2001,159,:1
3Phosp hatidylinositol-3-OH Kinase(Pl3K) / A KT2,activated in breast cancer,regulates and is induced by estrogen receptor alpha (ERalpha)via interaction between ERalpha and PI3K显示文摘SUn M Paciga JE Feldman RI 2001Cancer Res2001,61,16:1
4AKT1/PKBalpha kinase is frequently elevated in human cancers and its constitutive activation is required for oncogenic transformation in NIH3T3 cells显示文摘Sun M Wang G Paciga JE 0,,02:1
5Activation and overexpression of centrosome kinase BTAK/Aurora-A in human ovarian cancer显示文摘GRITSKO T M COPPOLA D PACIGA J E 2003Clin Cancer Res2003,9,4:1
6AKT1/PKB alpha kinase is frequently elevated in human cancers and its constitutive activation is required for oncogenic transformation in NIH3T3 cells 显示文摘SUN M WANG G PACIGA JE 2001Am J Pathol2001,159,2:1
7Activation and overexpression of centrosome kinase BTAK/Aurora A in human o varian cancer显示文摘Gritsko T M Coppola D Paciga J E 2003Clin Cancer Res2003,9,4:1
8Activation and Overexpression of Centrosome Kinase BTAK/Aurora-A in Human Ovarian Cancer显示文摘GRITSKO T M COPPOLA D PACIGA J E 2003Clin Cancer Res2003,9,4:1
9Activation and overexpression of centrosome kinase BTAK/Aurora A in human ovarian cancers显示文摘Gritsko T M Coppola D Paciga J E 2003Clin Cancer Res2003,9,:1
10Phosphatidylinositol-3-OH Kinase (PI3K)/AKT2,activated in breast cancer,regulates and is induced by estrogen receptor alpha (ERalpha) via interaction between ERalpha and PI3K显示文摘SUN M PACIGA JE FELDMAN RI 2001Cancer Res2001,61,16:1
11Evidence for cross - talk between stannioealcins 显示文摘Paciga M James K Gillespie JR 2005Can J Physiol Pharmacol2005,83,11:1
12Phosphatidylinositol-3-OH kinase (PI3K)/AKT2, activated in breast cancer, regulates and is induced by estrogen a(ER a) via interaction between ER a and PI3K显示文摘Sun M Paciga JE Feldman RI ct al 2001Cancer Res2001,61,:1
13AKT1/PKB alpha kinase is frequently elevated in human cancer and its constitutive activation is required for oncogenic transformation in NIH3T3 cells 显示文摘Sun M Wang G Paciga JE 2001Am J Pathol2001,159,2:1
14Phosphatidylinositol-3-OH kinase (PI3K)/Akt2,activated in breast cancer,regulates and is induced by estrogen receptor alpha (ERalpha) via interaction between ERalpha and PI3K显示文摘 Paciga JE Feldman RI 2001Cancer Res2001,61,16:1
15AKT1/PKB alpha kinase is frequently elevated in human cancer and its constitutive activation is required for oncogenic transformation in NIH3T3 cells显示文摘Sun M Wang G Paciga JE 2001Am J Pathol2001,159,2:1
16AKT1 /PKBalpha kinase is fre-quently elevated in human cancers and its constitutive activation isrequired for oncogenic transformation in NIH3T3 cells 显示文摘Sun M Wang G Paciga JE 2001Am JPathol2001,159,2:1
17Phosphatidylinositol-3OH Kinase (P13K)/AKT2,activated in breast cancer,regulates and is induced by estrogen receptor alpha (ERalpha) via interaction between ERalpha and PI3K显示文摘Sun M Paciga JE Feldman RI 2001Cancer Res2001,61,16:1
18AKT1/PKBalpha kinase is frequently elevated in human cancers and its constitutive activation iS required for oncogenic transformation in NIH3T3 cells显示文摘Sun M Wang G Paciga JE 2001Am J Pathol2001,159,2:1
19Targeting of big stannio- calcin and its receptor to lipid storage droplets of ovarian steroidogenic cells 显示文摘Paciga M Mccudden CR Londos C 2003J Biol Chem2003,278,49:1
20Evidence for cross-talk between stanniocalcins 显示文摘PACIGA M JAMES K GILLESP1E JR 2005Can J Physiol Pharmacol2005,83,11:1
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