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    题名 作者 年代 出处 被引量
1Cancer risk assessment from exposure to trihalomethanes in tap water and swimming pool water显示文摘我们在 Nakhon 路径的区域在自来水和游泳池水调查了 trihalomethanes (THM ) 的集中 -- om 自治市在时期 2005 年 4 月 2006 年 3 月期间。在自来水的全部的 THM ,氯仿, bromodichloromethane , dibromochloromethane 和 bromoform 的集中是 12.70 41.74 , 6.72 29.19 , 1.12 11.75 , 0.63 3.55 和 0.08 3.40 μg /L ,分别地而游泳池水里的那些是 26.15 65.09 , 9.50 36.97 , 8.90 18.01 , 5.19 22.78 并且 ND 6.56 μg /L 分别地。它暗示特别地,游泳池水里的 THM 的集中比在自来水的那些高 brominated-THMs。自来水和游泳池水在世界健康组织的标准下面包含了全部的 THM 的集中(),欧盟( EU )和美国环境保护机构( USEPA )分阶段执行我,但是 1 从 60 自来水取样, 60 从 72 游泳池水取样在 USEPA 阶段 II 的标准上包含了那些。从包括大小写的癌症风险评价的二个案例我非游泳者和案例 II 游泳者,对癌症风险的评价非--从暴露的游泳者到 THM 在最高并且平均集中是 4.43 ×1 0 ? 5 和 2.19 ×1 0 ? 5 ,分别地它能根据 USEPA 的标准作为可接受的风险被分类。对到 THM 在的从暴露的游泳者的癌症风险的评价最高并且平均集中是 1.47 × 1 0 ? 3 和 7.99 × 1 0 ? 4,分别地它能作为不能接受的风险和需要被分类被改善。从游泳的 THM 暴露的风险是 93.9%94.2% 全部的风险。THM 的癌症风险在下降从各种各样的线路断定顺序是:皮肤暴露当游泳时,从自来水吸入的胃肠的暴露和胃肠的暴露当游泳时。从皮肤暴露的癌症风险当游泳是 94.18% 全部的癌症风险时。PANYAKAPO Mallika SOONTORNCHAI Sarisak PAOPUREE Pongsri 2008Journal of Environmental Sciences2008,20,3:12
2Sympathectomy causes increased root resorption after orthodontic tooth movement in rats: immunohistochemical study显示文摘Sivakami Rethnam Haug Pongsri Brudvik Inge Fristad Karin J. Heyeraas 2003Cell and Tissue Research2003,,2:1
3Interleukin-1 α and tumor necrosis factor-α expression during the early phases of orthodontic tooth movement in rats显示文摘Athanasia B Ellen B Pongsri B 2006Eur J Oral Sci2006,114,5:1
4Thai nursing students’ adaption to problem-based learning: A qualitative study显示文摘Areewan Klunklin Pornpun Subpaiboongid Pongsri Keitlertnapha Nongkran Viseskul Sue Turale 2011Nurse Education in Practice2011,,6:1
5Polymorphic patterns of pfcrt and pfmdrl in Plasmodium falciparum isolates along the Thai-Myanmar border显示文摘Objective:To investigate the distribution and patterns of pfcrt and pfmdr1 polymorphisms in Plasmodium falciparum(P.falciparum)isolates collected from the malaria endemic area of Thailand along Thai-Myanmar border.Methods:Dried blood spot samples were collected from 172 falciparum malaria patients prior received treatment.The samples were extracted using chelex to obtain parasite DNA.PCR-RFLP was employed to detect pfert mutation at codons 76,220,271,326,3S6 and 371,and the pfmdr1mutation at codon 86.Pfmdr1 gene copy number was determined by SYBR Green 1 real-time PCR.Results:Mutant alleles of pfcrt and wild type allele of pfmdrl were found in almost all samples.Pfmdrl gene copy number in isolates collected from all areas ranged from 1.0 to S.0 copies and proportion of isolates carrying>1 gene copies was 38.1%.The distribution and patterns of pfcrt and pfmdrl mutations were similar in P.falciparum isolates from all areas.However,significant differences in both number of gfmdr1 copies and prevalence of isolates carrying>1 gene copies were observed among isolates collected from different areas.The median pfmdr1 copy number in P.falciparum collected from Kanchanaburi and Mae Hongson were 2.5 and 2.0,respectively and more than half of the isolates carried>1 gene copies.Conclusions:The observation of pfindr1 wild type and increasing of gene copy number may suggest declining of artesunate-mefloquine treatment efficacy in P.falciparum isolates in this border area.Phunuch Muhamad Wanna Chaijaroenkul Papichaya Phompradit Ronnatrai Rueangweerayut Pongsri Tippawangkosol Kesara Na-Bangchang 2013Asian Pacific Journal of Tropical Biomedicine2013,3,12:1
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