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| 1 | Biology of chronic graft-vs-host disease:Immune mechanisms and progress in biomarker discovery显示文摘Chronic graft-vs-host disease(c GVHD) is the leading cause of long-term morbidity and mortality following allogeneic hematopoietic stem cell transplantation. It presents as a chronic inflammatory and sclerotic autoimmune-like condition that most frequently affects the skin, oral mucosa, liver, eyes and gastrointestinal tract. Both clinical and animal studies have shown that multiple T cell subsets including Th1, Th2, Th17, T follicular helper cells and regulatory T-cells play some role in cG VHD development and progression; B cells also play an important role in the disease including the production of antibodies to HY and nuclear antigens that can cause serious tissue damage. An array of cytokines and chemokines produced by different types of immune cells also mediate tissue inflammation and damage of cG VHD target tissues such as the skin and oral cavity. Many of these same immune regulators have been studied as candidate cG VHD biomarkers. Recent studies suggest that some of these biomarkers may be useful for determining disease prognosis and planning long-term clinical followup of cG VHD patients. | Richard B Presland | 2016 | World Journal of Transplantation2016，6，4： | 4 |
| 2 | Decreased profilaggrin expression in ichthyosis vulgaris is a result of selectively impaired posttranscriptional control显示文摘 | Nirunsuksiri W Presland RE Brumbaugh SG | 1995 | J Bioi Chern1995，270，2： | 1 |
| 3 | Barrier function in transgenic mice overexpressing K16, involucrin, and filaggrin in the suprabasal epidermis显示文摘 | Presland RE Coulombe PA Eckert RL | 2004 | J Invest Dermatol2004，123，3： | 1 |
| 4 | Loss of normal profilaggrin and filaggrin in flaky tail (ft/ft) mice: an animal model for the filaggrin -deficient skin disease ichthyosis vulgaris显示文摘 | Presland RB Boggess D Lewis SP | 2000 | J Invest Dermatol2000，115，6： | 1 |
| 5 | Regulation of human profilaggrin promoter activity in cultured epithelial cells by retinoic acid and glucocorticids显示文摘 | Presland RB Tomic-Canic M Lew is SP | 2001 | J Dermatol Sci2001，27，3： | 1 |
| 6 | Evidence that TRPC1 contributes to calcium-induced differentiation of human keratinocytes 显示文摘 | Cai S Fatherazi S Presland RB | 2006 | Pflugers Arch2006，452，1： | 1 |
| 7 | Epithelial structural proteins of the skin and oral cavity:function in health and disease 显示文摘 | Presland RB Dale BA | 2000 | Crit Rev Oral Biol Med2000，11，4： | 1 |
| 8 | Decreased profilaggrin expression in ichthyosis vulgaris is a result of selectively impaired posttranscriptional control 显示文摘 | Nirunsuksiri W Presland RB Brumbaugh SG | 1995 | Biol Chem1995，270，2： | 1 |
| 9 | Regulation of human profilaggrin promoter activity in cultured epithelial cells by retinoic acid and glucocorticoids显示文摘 | Presland RB Tomic-Canic M Lewis SP | 2001 | J Dermatol Sci2001，27，3： | 1 |
| 10 | Information richTemporal virtual models using X3D显示文摘 | Hetherington R Farrimond B Presland S | 2006 | Computers&Graphics2006，30，2： | 1 |
| 11 | Barrier function in transgenic mice overexpressing K16, involucrin, and filaggrin in the suprabasal epidermis显示文摘 | presland RB Coulombe PA Eckert RL | 2004 | J Invest Dermatol2004，123，3： | 1 |
| 12 | Information richTem- poral virtual models using X3D 显示文摘 | Hetherington R Farrimond B Presland S | 2006 | Computers&Graphics2006，30，2： | 1 |
| 13 | Decreased profilaggrin expression in iehthyosis vulgaris is a result of selectively impaired posttranseriptional control显示文摘 | Nirunsuksiri W Presland RB Brumbaugh SG | 1995 | J Biol Chem1995，270，2： | 1 |
| 14 | Information rich temporal virtual models using X3D显示文摘 | Robina Hetherington Brian Farrimond and Steve Presland | 2006 | Computers & Graphics2006，30，2： | 1 |
| 15 | Barrier function in transgenic mice overexpressing K16, involucrin, and filaggrin in the suprabasal epidermis 显示文摘 | Presland RB Coulombe PA Eckert RL | 2004 | J Invest Dermatol2004，123，3： | 1 |
| 16 | Evidence for specific proteolyticcleavage of the N-terminal domain of human profilaggrin during epidermaldifferentiation显示文摘 | PRESLAND RB KIMBALL JR KAUTSKY MB | 1997 | J Invest Deratol1997，108，： | 1 |
| 17 | Loss of normal profilaggrin and filaggrin in flaky tail (ft/ft) mice:an animal model for the filaggrin-deficient skin disease ichthyosis vulgaris显示文摘 | Presland RB Boggess D Lewis SP | 2000 | J Invest Dermatol2000，115，6： | 1 |
| 18 | Salivary α-synuclein and DJ-1:potential biomarkers for Parkinson's disease显示文摘 | Devic I Hwang H Edgar J S Izutsu K Presland R Pan C Goodlett D R Wang Y Armaly J Tumas V Zabetian C P Leverenz J B Shi M Zhang J | | 0，，07： | 1 |
| 19 | Graphite formation by the dissolution-precipitation of carbon in cobalt, nickel and iron显示文摘 | Derbyshire F J Presland A E B Trimm D L | 1975 | Carbon1975，13，2： | 1 |
| 20 | Information Rich Temporal Virtual Models Using X3D显示文摘 | Hetherington R Farrimond B Presland S | 2006 | Computers & Graphics2006，30，2： | 1 |
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