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19篇 您的检索式:作者名="Presser T"
    题名 作者 年代 出处 被引量
1Short chain aliphatic acid anions in oil field waters and their contribution to the measured alkalinity显示文摘Willey L M Kharaka Y K Presser T S 1975Geochim et Cosmochim Acta1975,39,:1
2Geochemistry of tectonically expelled fluids from the northern Coast Ranges, Rumsey Hills显示文摘Davisson M L Presser T S Criss R E 1994Geochimica et Cosmochimica Acta1994,58,:1
3Anomalous chemical changes in welt waters and possible relation to earthquakes显示文摘King C Y Evans W C Presser T 1981Geophys Res Lett1981,8,:1
4Short chain aliphatic acid anions in oil field waters and their contribution to the measured alkalinity显示文摘 Kharaka Y K Presser T S 1975Geochimica et Cosmochimica Acta1975,39,:1
5Geochemical evidence for Se mobilization by the weathering of pyritic shale, San Joaquin valley, California, USA显示文摘Presser T S Swain W C 1990Applied Geochemistry1990,5,:1
6Modelling the growth limits (growth no growth interface) of Escherichiacoli as a function of temperature, pH, lactic acid concentration and water activity显示文摘Presser K A Ross T Ratkowsky D A 1998Appl Environ Microbiol1998,64,5:1
7Community Structure of Ammonia-Oxidizing Bacteria within Anoxie Marine Sediments 显示文摘Freitag T E Presser J 2003Appl Environ Microb2003,69,3:1
8Exploring the visible human using the VOXEL-MAN framework 显示文摘Schiemann T Freudenberg J Presser B 2000Comput Med Imaging Graph2000,24,2:1
9Combustion of methanol and methanol/dodecanol spray flames显示文摘Presser C Gupta A K Avedisian C T 1992Journal of Propulsion and Power1992,8,3:1
10Modeling the growth limits (growth/no growth) interface of Escherichia coli as a function of pH, lactic acid and temperature显示文摘Presser K Ross T Ratkowsky D A 1998Appl Environ Microbiol1998,64,:1
11Geochemistry of tectoni-cally expelled fluids from the northern Coast Ranges,RumseyHills显示文摘Davisson M L Presser T S Criss R E 1994Geochim et Cosmochimica Acta1994,58,:1
12The kesterson effect显示文摘Presser T S 1994Environmental Management1994,18,3:1
13Modelling the growth rate of Escherichia coli as a function of pH and lactic acid concentration显示文摘Presser KA Ratkowsky DA Ross T 1997Applied and Environmental Microbiology1997,63,6:1
14The added burden of comorbidity in patients with asthma 显示文摘Zhang T Carleton BC Presser R J 2009J Asthma2009,46,:1
15Removal of selenium from contaminated agricultural drainage water by nanofiltration membranes 显示文摘Kharaka Y K Ambats G Presser T S 1996Applied Geochemistry1996,11,6:1
16In search of earthquake-related hydrological and chemical changes along Hayward fault显示文摘King C Y Basler D Presser T S 1994Application of Geochemistry1994,9,:1
17Modelling the growth limits (growth/no growth interface)of Escherichia coli as a function of temperature,pH,actic acid and water activity显示文摘Presser K A Ross T Ratkowsky D A 1998App Environ Microbiol1998,64,:1
18Removal of selenium from contaminated agricultural drainage water by nanofiltration membranes 显示文摘Kharaka Y K Ambats G Presser T S 1996Applied Geochemistry1996,11,6:1
19An unhappy triad:Hemochromatosis, porphyria cutanea tarda and hepatocellular carcinoma-A case report显示文摘Liver fibrosis and cirrhosis are predisposing factors for the development of hepatocellular carcinoma (HCC). Hemosiderosis has also been described to trigger carcinogenesis. A significant iron overload, as found in hereditary hemochromatosis (HHC), is a risk factor for HCC and may also promote the symptoms of porphyria cutanea tarda (PCT). A 68-year old male patient presented to our clinic with a suspected HCC, elevated alpha-fetoprotein but normal liver function tests. He reported a 25 year-old history of vitiligo upon exposure to sunlight. The patient underwent an extended left hemihepatectomy, and the recovery was uneventful, with the exception of a persistent hyperbilirubinemia. Perfusion problems and extrahepatic cholestasis were ruled out by CT-scan with angiography and MR-cholangiopancreatography. However, MRI showed an iron overload. Histology confirmed the HCC (pT3, pN0, G3, R0) and revealed a portal fibrosis and hemosiderosis. Based on the skin lesions we suspected a PCT that was confirmed by laboratory tests showing elevated porphyrin, uroporphyrin, coproporphyrin and porphobilinogen. Concurrently, molecular diagnostics revealed homozygosity for the C282Y mutation within the hemochromatosis HFE gene. After phlebotomy and normalization of liver function tests the patient was discharged. This is the first case ever showing the unusual combination of HCC in a fibrotic liver with HHC and PCT. This diagnosis not only warrants oncological follow-up but also symptomatic therapy to normalize iron metabolism and thereby improve liver function and alleviate the symptoms of HHC and PCT. Thus progression of fibrosis may be prevented and liver regeneration supported.Martina T Mogl Andreas Pascher Sabine J Presser Michael Schwabe Peter Neuhaus Natascha C Nuessler 2007World Journal of Gastroenterology2007,13,13:0
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