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| 1 | Human herpesvirus 6 infections after liver transplantation显示文摘Human herpesvirus 6(HHV-6) infections occur in > 95% of humans.Primary infection,which occurs in early childhood as an asymptomatic illness or manifested clinically as roseola infantum,leads to a state of subclinical viral persistence and latency.Reactivation of latent HHV-6 is common after liver transplantation,possibly induced and facilitated by allograft rejection and immunosuppressive therapy.Since the vast majority of humans harbor the virus in a latent state,HHV-6 infections after liver transplantation are believed to be mostly due to endogenous reactivation or superinfection(reactivation in the transplanted organ).In a minority of cases,however,primary HHV-6 infection may occur when an HHV-6 negative individual receives a liver allograft from an HHV-6 positive donor.The vast majority of documented HHV-6 infections after liver transplantation are asymptomatic.In a minority of cases,HHV-6 has been implicated as a cause of febrile illness with rash and myelosuppression,hepatitis,pneumonitis,and encephalitis after liver transplantation.In addition,HHV-6 has been associated with a variety of indirect effects such as allograft rejection,and increased predisposition and severity of other infections including cytomegalovirus(CMV),hepatitis C virus,and opportunistic fungi.Because of the uncommon nature of the clinical illnesses directly attributed to HHV-6,there is currently no recommended HHV-6-specific approach to prevention.However,ganciclovir and valganciclovir,which are primarily intended for the prevention of CMV disease,are also active against HHV-6 and may prevent its reactivation after transplantation.The treatment of established HHV-6 disease is usually with intravenous ganciclovir,cidofovir,or foscarnet,complemented by reduction in the degree of immunosuppression.This article reviews the current advances in the pathogenesis,clinical diagnosis,and therapeutic modalities against HHV6 in the setting of liver transplantation. | Rima Camille Abdel Massih Raymund R Razonable | 2009 | World Journal of Gastroenterology2009，15，21： | 5 |
| 2 | Adsorption of copper (Ⅱ) and chromium (Ⅲ) ions onto amidoximated cellulose 显示文摘 | RIMA S HELENE G MICHELE P R | 2000 | Journal of Applied Polymer Science2000，75，13： | 1 |
| 3 | Differential receptor usage by measles virus strains 显示文摘 | Bartz R Firsching R Rima B | 1998 | J Gen Virol1998，79，5： | 1 |
| 4 | Differential recep- tor usage by measles virus stains显示文摘 | Bartz R Firsching R Rima B | 1998 | J Gen Virol1998，79，5： | 1 |
| 5 | Are Building-Level characteristics associated with indoor allergens in the household显示文摘 | Lindsay R Ginger LC Rima R | 2011 | J Urban Health2011，88，1： | 1 |
| 6 | CD25 expression on donor CD4^+ or CD8^+ T cells is associated with an increased risk for graft-verushost disease after HLA-identical stem cell transplantation in human显示文摘 | Stanzani M Sergio L R Rima M | | Blood0，103，3： | 1 |
| 7 | Adsorption of copper (II)and chromium(III)ions onto amidoximated cellulose显示文摘 | RIMA S HELENE G MICHELE P R | 2000 | Journal of Applied Polymer Science2000，75，13： | 1 |
| 8 | Systemic cap illy leak syndrome显示文摘 | Chihara R Nakamoto H A rima H | 2002 | Intern Med2002，41，11： | 1 |
| 9 | 显示文摘 | RIMA B FRANKLIN DOUGLAS R | 1999 | Microbiol Methods1999，35，3： | 1 |
| 10 | The evolution of morbilliviruses:a comparison of nucleocapsid gene sequences including a porpoise mobillivirus显示文摘 | Rima B K Wishaupt R G Welsh M J | 1995 | Vet Microbiol1995，44，： | 1 |
| 11 | Mapping amino acids of the measles virus hemagglutinin responsible for re- ceptor (CD46) downregulation显示文摘 | Bartz R Brinckmann U Dunster L M Rima B Ter Meulen V Schneider-Schaulies J | 1996 | Virology1996，224，1： | 1 |
| 12 | The evolution of morbilliviruses:a comparison of nucleocapsid gene sequencesincluding a porpoise morbillivirus显示文摘 | Rima B K R G Wishaupt M J Welsh | 1995 | Vet Microbiol1995，44，： | 1 |
| 13 | Implications of Cytogenetic Abnormalities and Azoospermia Factor Microdeletions in Assisted Procreation显示文摘Assisted procreation techniques have revolutionized the management of infertility and have offered hope to millions of infertile couples. The main aim of these proce- dures is to produce healthy offspring. However recent studies on short term outcome of ART have reported a higher incidence of low birth weight, development delay, imprinting defects, sex and autosomal structural abnormalities, major and minor congenital malformation and certain cancers in babies conceived via ART. Further the health of ART conceived children beyond the neonatal period have been less well evaluated. A large number of infertile couples opting for ART have an underlying genetic aetiology. These genetic aberrations are iatrogenitically transmitted via ART. Thus it is important that all couples undergo a detailed and comprehensive genetic evaluation prior to ART. | Rima Dada R Kumar M Bilal Shamsi Sarabpreet Singh Col R K Sharma A Varshney | 2007 | Journal of Reproduction and Contraception2007，18，3： | 0 |
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