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17篇 您的检索式:作者名="Susan Jose"
    题名 作者 年代 出处 被引量
1A community-derived classification for extant lycophytes and ferns显示文摘发展史长通知了蕨类植物分类。当我们推断进化的树的能力改善了,针对认出生来的组的分类变得逐渐地预兆、稳定。这里,我们为 lycophytes 和蕨纲植物提供一个现代、全面分类,在下面,利用一条基于社区的途径类水平。我们 monophyly 用作主要标准让 taxa,而且目的识别保存两个广泛地被接受的存在 taxa 和界限并且与我们蕨类植物发展史的理解一致。总共,这个分类对待一在 337 个类, 51 个家庭, 14 目,和二个班估计了 11  916 种类。这个分类没在 lycophyte 和蕨纲植物上作为最后的词被打算分类,而是当前的假设的概括陈述,源于最好的可得到的数据并且在问题由熟悉植物的那些大多数塑造了。我们希望它将在蕨类植物上为最近的文学的那些想要的参考用作一个资源发展史和分类,为指导未来调查的一个框架,和推进讲话的刺激。Eric Schuettpelz Harald Schneider Alan R. Smith Peter Hovenkamp Jefferson Prado Germinal Rouhan Alexandre Salino Michael Sundue Thafs Elias Almeida Barbara Parris Emily B. Sessa Ashley R. Field Andre Luis de Gasper Carl J. Rothfels Michael D. Windham Marcus Lehnert Benjamin Dauphin Atsushi Ebihara Samuli Lehtonen Pedro Bond Schwartsburd Jordan Metzgar Li-Bing Zhang Li-Yaung Kuo Patrick J. Brownsey Masahiro Kato Marcelo Daniel Arana Francine C. Assis Michael S. Barker David S. Barrington Ho-Ming Chang Yi-Han Chang Yi-Shan Chao Cheng-Wei Chen De-Kui Chen Wen-Liang Chiou Vinicius Antonio de Oliveira Dittrich Yi-Fan Duan Jean-Yves Dubuisson Donald R. Farrar Susan Fawcett Jose Maria Gabriel y Galan Luiz Armando de Araujo Goes-Neto Jason R. Grant Amanda L. Grusz Christopher Haufler Warren Hauk Hai He Sabine Hennequin Regina Yoshie Hirai Layne Huiet Michael Kessler Petra Korall Paulo H. Labiak Anders Larsson Blanca Leen Chun-Xiang Li Fay-Wei Li Melanie Link-Perez Hong-Mei Liu Ngan Thi Lu Esteban I. Meza-Torres Xin-Yuan Miao Robbin Moran Claudine Massi Mynssens Nathalie Nagalingum Benjamin Ollgaard Alison M. Paul Jovani B. de S. Pereira Leon R. Perrie Monica Ponce Tom A. Ranker Christian Schulz Wataru Shinohara Alexander Shmakov Erin M. Sigel Filipe Soares de Souza Lana da Silva Sylvestre Weston Testo Luz Amparo Triana-Moreno Chie Tsutsumi Hanna Tuomisto IvAn A. Valdespino Alejandra Vasco Raquel Stauffer Viveros Alan Weakley Ran Wei Stina Weststrand Paul G. Wolf George Yatskievych Xiao-Gang Xu Yue-Hong Yan Liang Zhang Xian-Chun Zhang Xin-Mao Zhou 2016Journal of Systematics and Evolution2016,54,6:44
2Cell Wall Microstructure Analysis Implicates Hemicellulose Polysaccharides in Cell Adhesion in Tomato Fruit Pericarp Parenchyma显示文摘开发从未熟、成熟的西红柿水果果皮实质孤立未经触动的房间的方法允许房间在分开的房间的表面的多糖 epitopes 的生物分析。LM7 果胶的 homogalacturonan epitope 在实质系统是粘附飞机和细胞间的空格的连接的一个标记。LM7 epitope 固执地在用 Cnr 变化从水果从未熟、成熟的西红柿水果并且也分开的房间的表面标记粘附飞机的以前的边。LM11 木聚糖 epitope 被联系,在节,与房间墙衬里细胞间的空间但是 epitope 没在孤立的房间的表面被检测,在房间隔离期间被失去。LM15 xyloglucan epitope 在在反映房间粘附 planes/intercellular 空间,但是与差距和明显的裂缝的以前的边的一个模式从未熟的水果孤立的房间的表面是在场的。LM15 epitope 出现的一个相等的模式在 pectate lyase 行动孤立,但是不是在从成熟水果或从 Cnr 水果孤立的房间的现在的房间的表面被揭示。与野类型的房间相对照, LM5 galactan 和 LM21 mannan epitopes 在在 Cnr 反映细胞间的空间的位置主要发生了,建议在响应这个变化的房间墙微观结构的一致改变。与果胶的降级 homogalacturonan 酶一起, Galactanase 和 mannanase 能够免除房间未熟的水果实质。这些观察显示 hemicellulose 聚合物在反映房间粘附的建筑上的上下文是在场的并且几个房间墙多糖班是可能的在西红柿水果果皮实质贡献房间粘附 / 房间分离。Jose J. Ordaz-Ortiz Susan E. Marcus J. Paul Knox 2009Molecular Plant2009,2,5:4
3Predictors of High-grade Esophagitis After Definitive Three-dimensional Conformal Therapy, Intensity-modulated Radiation Therapy, or Proton Beam Therapy for Non-small cell Lung Cancer显示文摘Daniel R. Gomez Susan L. Tucker Mary K. Martel Radhe Mohan Peter A. Balter Jose Luis Lopez Guerra Hongmei Liu Ritsuko Komaki James D. Cox Zhongxing Liao 2012International Journal of Radiation Oncology, Biology, Physics2012,,:2
4Metastatic and non-metastatic colorectal cancer (CRC) cells induce host metalloproteinase production in vivo显示文摘Susan Mc Donnell Vivek Chaudhry Jorge Mansilla-Soto Zhao-Shi Zeng Wei-ping Shu Jose G. Guillem 1999Clinical and Experimental Metastasis1999,,4:1
5Lipid profiling identifies a triacylglycerol signature of insulin resistance and improves diabetes prediction in humans显示文摘Rhee Eugene P Cheng Susan Larson Martin G Walford Geoffrey A Lewis Gregory D McCabe Elizabeth Yang Elaine Farrell Laurie Fox Caroline S O’Donnell Christopher J Carr Steven A Vasan Ramachandran S Florez Jose C Clish Clary B Wang Thomas J Ger 2011Journal of Clinical Investigation2011,,4:1
6Double knockout pigs deficient in N ‐glycolylneuraminic acid and G alactose α‐1,3‐ G alactose reduce the humoral barrier to xenotransplantation显示文摘Andrew J. Lutz Ping Li Jose L. Estrada Richard A. Sidner Ray K. Chihara Susan M. Downey Christopher Burlak Zheng‐Yu Wang Luz M. Reyes Bess Ivary Fuqin Yin Ross L. Blankenship Leela L. Paris A. Joseph Tector 2013Xenotransplantation2013,,1:1
7Double knockout pigs deficient in N ‐glycolylneuraminic acid and G alactose α‐1,3‐ G alactose reduce the humoral barrier to xenotransplantation显示文摘Andrew J. Lutz Ping Li Jose L. Estrada Richard A. Sidner Ray K. Chihara Susan M. Downey Christopher Burlak Zheng‐Yu Wang Luz M. Reyes Bess Ivary Fuqin Yin Ross L. Blankenship Leela L. Paris A. Joseph Tector 2013Xenotransplantation2013,,1:1
8Insulin-like growth factor- I stimulation of lymphopoiesis显示文摘Clark R Jose Strasser Susan McCabe 2007J Clin Invest2007,,92:1
9Prediction of leaf area index in almonds by vegetation indexes显示文摘Jose L. Zarate-Valdez Michael L. Whiting Bruce D. Lampinen Samuel Metcalf Susan L. Ustin Patrick H. Brown 2012Computers and Electronics in Agriculture2012,,:1
10The Safety and Efficacy of the Artificial Bowel Sphincter for Fecal Incontinence显示文摘W. Douglas Wong M.D. Susan M. Congliosi M.D. Michael P. Spencer M.D. Marvin L. Corman M.D. Patrick Tan M.D. Frank G. Opelka M.D. Marcus Burnstein M.D. Juan J. Nogueras M.D. H. Randolph Bailey M.D. Jose Manuel Devesa M.D. Robert D. Fry M.D. Burt Cagir M.D. 2002Diseases of the Colon & Rectum2002,,9:1
11Characterization of food waste and bulking agents for composting显示文摘Kijaya K.Adhikari Suzelle Barrington Jose Martinez Susan King 0,,:1
12Selenium-binding protein-1 in smooth muscle cells is downregulated in a rhesus monkey model of chronic allograft nephropathy显示文摘JOSE R T MATTHEW C SUSAN G 2005American Journal of Transplantation2005,5,1:1
13Age Estimation Using Single-Rooted Teeth of Indian Residents by Modified Kvaal’s Method:A Pilot Study显示文摘Background:This study aimed to create an age estimation formula with high sensitivity and specificity using the modified Kvaal’s dental radiographic method.The parameters for age estimation were assessed by measuring the length and width of tooth,root,and pulp with the help of a stereomicroscope and digital image analysis.Materials and Methods:Only single-rooted noncarious teeth were included.A total of 203 samples of teeth,with an age ranging from 15 to 85 years,were collected,of which only 123 samples were included in the study.Images of vertically sectioned teeth specimens were captured under a stereomicroscope(version SZ-790,×10 mm eyepiece),and parameters were measured using Adobe Photoshop.Results:Karl Pearson’s correlation coefficient was used to correlate age with the dental parameters,and step-wise regression analysis was used to derive the age estimation formula.A significant correlation of age with pulp length and root and pulp width having P<0.001 was observed,and an age estimation formula with a standard estimation error of 14 could be derived.Conclusion:Kvaal’s parameter in extracted single-rooted tooth showed a better reliability and reproducibility in age estimation.Further studies with a larger sample size and tooth-specific formula using principal component analysis should be considered to validate the accuracy of the present result.Deepa Jose Deepa R Mane Soma Susan Varghese Pramod Philip Mathews Joseph Sebastian 2023Journal of Forensic Science and Medicine2023,9,1:0
14Mobilizing Crop Biodiversity显示文摘Over the past 70 years,the world has witnessed extraordinary growth in crop productivity,enabled by a suite of technological advances,including higher yielding crop varieties,improved farm management,synthetic agrochemicals,and agricultural mechanization.While this'Green Revolution'intensified crop production,and is credited with reducing famine and malnutrition,its benefits were accompanied by several undesirable collateral effects(Pingali,2012).These include a narrowing of agricultural biodiversity,stemming from increased monoculture and greater reliance on a smaller number of crops and crop varieties for the majority of our calories.This reduction in diversity has created vulnerabilities to pest and disease epidemics,climate variation,and ultimately to human health(Harlan,1972).Susan McCouch Zahra Katy Navabi Michael Abberton Noelle L.Anglin Rosa Lia Barbieri Michael Baum Kirstin Bett Helen Booker Gerald L.Brown Glenn J.Bryan Luigi Cattivelli David Charest Kellye Eversole Marcelo Freitas Kioumars Ghamkhar Dario Grattipaglia Robert Henry Maria Cleria Valadares Inglis Tofazzal Islam Zakaria Kehel Paul J.Kersey Graham J.King Stephen Kresovich Emily Marden Sean Mayes Marie Noelle Ndjiondjiop Henry T.Nguyen Samuel Rezende Paiva Roberto Papa Peter W.B.Phillips Awais Rasheed Christopher Richards Mathieu Flouard Maria Jose Amstalden Sampaio Uwe Scholz Paul D.Shaw Brad Sherman S.Evan Staton Nils Stein Jan Svensson Mark Tester Jose Francisco Montenegro Valls Rajeev Varshney Stephen Visscher Eric von Wettberg Robbie Waugh Peter Wenzl Loren H.Rieseberg 2020Molecular Plant2020,13,10:0
15A comprehensive review of genetic causes of obesity显示文摘Background Obesity is a multifactorial chronic disease with a high,increasing worldwide prevalence.Genetic causes account for 7%of the cases in children with extreme obesity.Data sources This narrative review was conducted by searching for papers published in the PubMed/MEDLINE,Embase and SciELO databases and included 161 articles.The search used the following search terms:'obesity','obesity and genetics','leptin','Prader-Willi syndrome',and'melanocortins'.The types of studies included were systematic reviews,clinical trials,prospective cohort studies,cross-sectional and prospective studies,narrative reviews,and case reports.Results The leptin-melanocortin pathway is primarily responsible for the regulation of appetite and body weight.However,several important aspects of the pathophysiology of obesity remain unknown.Genetic causes of obesity can be grouped into syndromic,monogenic,and polygenic causes and should be assessed in children with extreme obesity before the age of 5 years,hyperphagia,or a family history of extreme obesity.A microarray study,an analysis of the melanocortin type 4 receptor gene mutations and leptin levels should be performed for this purpose.There are three therapeutic levels:lifestyle modifications,pharmacological treatment,and bariatric surgery.Conclusions Genetic study technologies are in constant development;however,we are still far from having a personalized approach to genetic causes of obesity.A significant proportion of the affected individuals are associated with genetic causes;however,there are still barriers to its approach,as it continues to be underdiagnosed.Marcio Jose Concepcion-Zavaleta Juan Eduardo Quiroz-Aldave Maria del Carmen Durand-Vasquez Elman Rolando Gamarra-Osorio Juan del Carmen Valencia de la Cruz Claudia Mercedes Barrueto-Callirgos Susan Luciana Puelles-Leon Elena de Jesus Alvarado-Leon Frans Leiva-Cabrera Francisca Elena Zavaleta-Gutierrez Luis Alberto Concepcion-Urteaga Jose Paz-Ibarra 2024World Journal of Pediatrics2024,20,1:0
16Spinal cord injury reprograms muscle fibroadipogenic progenitors to form heterotopic bones within muscles显示文摘The cells of origin of neurogenic heterotopic ossifications(NHOs), which develop frequently in the periarticular muscles following spinal cord injuries(SCIs) and traumatic brain injuries, remain unclear because skeletal muscle harbors two progenitor cell populations: satellite cells(SCs), which are myogenic, and fibroadipogenic progenitors(FAPs), which are mesenchymal. Lineage-tracing experiments using the Cre recombinase/Lox P system were performed in two mouse strains with the fluorescent protein Zs Green specifically expressed in either SCs or FAPs in skeletal muscles under the control of the Pax7 or Prrx1 gene promoter, respectively. These experiments demonstrate that following muscle injury, SCI causes the upregulation of PDGFRα expression on FAPs but not SCs and the failure of SCs to regenerate myofibers in the injured muscle, with reduced apoptosis and continued proliferation of muscle resident FAPs enabling their osteogenic differentiation into NHOs. No cells expressing Zs Green under the Prrx1 promoter were detected in the blood after injury, suggesting that the cells of origin of NHOs are locally derived from the injured muscle. We validated these findings using human NHO biopsies. PDGFRα+mesenchymal cells isolated from the muscle surrounding NHO biopsies could develop ectopic human bones when transplanted into immunocompromised mice, whereas CD56+myogenic cells had a much lower potential. Therefore, NHO is a pathology of the injured muscle in which SCI reprograms FAPs to undergo uncontrolled proliferation and differentiation into osteoblasts.Hsu-Wen Tseng Dorothée Girard Kylie A.Alexander Susan M.Millard Frédéric Torossian Adrienne Anginot Whitney Fleming Jules Gueguen Marie-Emmanuelle Goriot Denis Clay Beulah Jose Bianca Nowlan Allison R.Pettit Marjorie Salga François Genêt Marie-Caroline Le Bousse-Kerdilès Sébastien Banzet Jean-Pierre Lévesque 2022Bone Research2022,10,2:0
17Does a biological link exist between periodontitis and rheumatoid arthritis?显示文摘Periodontitis or Periodontal disease(PD) and Rheumatoid arthritis(RA) are two the most common chronic inflammatory diseases. Periodontitis is a biofilm associated destructive inflammatory disease of the periodontium caused by specific microorganisms. Rheumatoid arthritis is an autoimmune condition and is identified by elevated serum autoantibody titre directed against citrullinated peptides or rheumatoid factor. Periodontitis may involve some elements of autoimmunity. Recent studies have established that PD and RA show a common pathway and could be closely associated through a common dysregulation and dysfunction in inflammatory mechanism. The enzyme peptidyl arginine deiminase(PAD), expressed by Porphyromonas gingivalis(P. gingivalis) is responsible for the enzymatic deimination of arginine residuals to citrulline resulting in protein citrullination and its increased accumulation in RA.Citrullination by PAD may act as a putative biologic link between PD and RA. Association of Human leukocytic antigen-DR4 antigen has been established both with RA and PD. Several interleukins and inflammatory mediators(ILs) and Nuclear factor kappa beta ligand are linked to these common chronic inflammatory diseases. Antibodies directed against heat shock protein(hsp 70 ab) of P. gingivalis, P. melanogenicus and P. intermedia are raised in PD as well as RA. Both the conditions share many pathological and immunological similarities. Bacterial infection, genetic susceptibility, altered immune reaction and inflammatory mediators considered responsible for RA are also associated with PD. So it is plausible that a biological link may exist between PD and RA. Therapies aimed at modifying the expression and effect of inflammatory mediators and effector molecules such as matrix metalloproteinases, proinflammatory cytokines and autoantibodies of structural proteins may probably reduce the severity of both RA and PD.Rosamma Joseph MG Jose Raj Shobha Sundareswaran Priyanka Chand Kaushik Amol Vijay Nagrale Susan Jose Sreeraj Rajappan 2014World Journal of Rheumatology2014,4,3:0
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