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| 1 | Consequences of gestational and pregestational diabetes on placental function and birth weight显示文摘Maternal diabetes constitutes an unfavorable environment for embryonic and fetoplacental development. Despite current treatments, pregnant women with pregestational diabetes are at increased risk for congenital malformations, materno-fetal complications, placental abnormalities and intrauterine malprogramming. The complications during pregnancy concern the mother (gravidic hypertension and/or preeclampsia, cesarean section) and the fetus (macrosomia or intrauterine growth restriction, shoulder dystocia, hypoglycemia and respiratory distress). The fetoplacental impairment and intrauterine programming of diseases in the offspring's later life induced by gestational diabetes are similar to those induced by type 1 and type 2 diabetes mellitus. Despite the existence of several developmental and morphological differences in the placenta from rodents and women, there are similarities in the alterations induced by maternal diabetes in the placenta from diabetic patients and diabetic experimental models. From both human and rodent diabetic experimentalmodels, it has been suggested that the placenta is a compromised target that largely suffers the impact of maternal diabetes. Depending on the maternal metabolic and proin ammatory derangements, macrosomia is explained by an excessive availability of nutrients and an increase in fetal insulin release, a phenotype related to the programming of glucose intolerance. The degree of fetal damage and placental dysfunction and the availability and utilisation of fetal substrates can lead to the induction of macrosomia or intrauterine growth restriction. In maternal diabetes, both the maternal environment and the genetic background are important in the complex and multifactorial processes that induce damage to the embryo, the placenta, the fetus and the offspring. Nevertheless, further research is needed to better understand the mechanisms that govern the early embryo development, the induction of congenital anomalies and fetal overgrowth in maternal diabetes. | Anne Vambergue Isabelle Fajardy | 2011 | World Journal of Diabetes2011，2，11： | 14 |
| 2 | Pregnancy induced hypertension in women with gestational carbohydrate intolerance:the diagest study显示文摘 | Vambergue A Nuttens MC Goeusse P | | 0，，01： | 1 |
| 3 | Gestational diabetes: Diagnosis, short and long term management 显示文摘 | Vambergue A | 2013 | Presse Med2013，42，5： | 1 |
| 4 | Pregnancy induced hypertension in women with gestational carbohydrate intolerance:the diagest study显示文摘 | Vambergue A Nuttens MC Goeusse P | 2002 | Eur J Obstet Gynecol Reprod Biol2002，102，1： | 1 |
| 5 | Gestational diabetes mellitus and HLA class Ⅱ(-DQ,-DR) association显示文摘 | Vambergue A Fajardy I Bianchi F | 1997 | Eur J Immunogenet1997，24，5： | 1 |
| 6 | Pathophysiology of gestational diabetes显示文摘 | Vambergue A Valat AS Dufour P | 2002 | J Gynecol Obstet Biol Reprod (Paris)2002，31，6： | 1 |
| 7 | Pathophysiology of gestational diabetes显示文摘 | Vambergue A Valat AS Dufour P | 2002 | J Gynecol Obstet Biol Reprod(Paris)2002，31，6： | 1 |
| 8 | Blood ketone monitoring:a comparison between gestational diabetes and nondiabetic pregnant women显示文摘 | Gin H Vambergue A Vasseur C | 2006 | Diabetes Metab2006，32，： | 1 |
| 9 | Autoimmune markers in slow typeldiabetes: confrontation to type 1 diabetes 显示文摘 | Desailloud R Fajardy I Vambergue A | 2000 | Diabetes-Metab2000，26，： | 1 |
| 10 | Pregnancy induced hypertension in women with gestation carbohydrate intolerance显示文摘 | Vambergue A Nuttens MC Goeusse P | 2002 | Eur J Obstet Gynecol Reprod Biol2002，102，： | 1 |
| 11 | Pregnancy induced hypertension in women with gestational carbohydrate intolerance: the diagnosis study 显示文摘 | Vambergue A Nuttens MC | 2002 | E J Obstet Gynecol Reprod Biol2002，102，1： | 1 |
| 12 | Gestational diabetes melli tus and HLA class Ⅱ (-DQ,-DR) association:the diagest study显示文摘 | Vambergue A Fajarby I Bianchi F | 1997 | EurJ Immunogenet1997，24，5： | 1 |
| 13 | Pregnancy-induced hypertension in women with gestational carbohydrate intolerance显示文摘 | Vambergue A Nuttens MC Goeusse P | 2002 | Eur J Obstet Gynecol Reprod Biol2002，102，1： | 1 |
| 14 | Pregnancy induced hypertension in women with gestational carbohydrate intolerance:the diagest study显示文摘 | Vambergue A Nuttens MC Goeusse P | 2002 | E J of Obstetrics& Gynecology and Reproductive Biology2002，102，： | 1 |
| 15 | Blood ketone monitoring:a comparison between geBtational diabetes and non - diabetic pregnantwomen 显示文摘 | Gin H Vambergue A Vasseur C | 2006 | Diabetes Metab2006，32，6： | 1 |
| 16 | Increasing incidence of abnor malucose tolerance in women the prior abnormal Oucose tolerance duringpregnancy:DIAGEST 2 study显示文摘 | Vambergue A Dognin C Boulogne A | 2008 | Diabet Med2008，25，1： | 1 |
| 17 | Pregnancy induced hypertension in women with gestational carbohydrate intolerance: the diagest study显示文摘 | Vambergue A Nuttens M C Goeusse P | 2002 | Eur J Obstet Gynecol Reprod Biol2002，102，1： | 1 |
| 18 | Pregnancy induced hypertension in women with gestational carbohydrate intoler ance: the diagest study显示文摘 | Vambergue A Nuttens MC Goeusse P | 2002 | Eur J Ob- stet Gynecol Reprod Biol2002，102，1： | 1 |
| 19 | Autoimmune markers in slow type 1 diabetes: confrontation to type 1 diabetes显示文摘 | Fajardy I Vambergue A | 2000 | Diabetes Metab2000，26，5： | 1 |
| 20 | Pregnancy-induced hypertension in women with gestational carbohydrate intolerance显示文摘 | Vambergue A Nuttens MC Goeusse P | 2002 | Eur J Obstet Gynecol Reprod Biol2002，102，1： | 1 |
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