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15篇 您的检索式:作者名="Aranega"
    题名 作者 年代 出处 被引量
1Human cardiac tissue induces transdifferentiation of adult stem cells towards cardiomyocytes显示文摘Macarena Perán Juan A. Marchal Elena López Manuel Jiménez-Navarro Houria Boulaiz Fernando Rodríguez-Serrano Esmeralda Carrillo Gema Sánchez-Espin Eduardo de Teresa David Tosh Antonia Aranega 2010Cytotherapy2010,,3:1
2Circulating alphaactin protein and tropein in acute myocardial infarction显示文摘Aranega AE Reina A Muros MA 1993Int J Cardiol1993,1,:1
3Pitx2c modulates Pax3 +/Pax7 + cell populations and regulates Pax3 expression by repressing miR27 expression during myogenesis显示文摘Lozano-Velasco E Contreras A Crist C Hernandez-Torres F Franco D Aranega AE 2011Dev Biol2011,357,1:1
4Evolution of serum angiogenic growth factor levels after breast cancer surgery显示文摘Emilo A Aranega A Sevilla M 2000ASCO Proc2000,19,:1
5A role for p38alpha mitogen-activated protein kinase in embryonic cardiac differentiation 显示文摘Herrmndez-Torres F Martinez-Fernandez S Zuluaga S Nebreda A Porras A Aranega AE 2008FEBS Lett2008,582,7:1
6Circulation α-actin protein in acute myocardial infarction显示文摘Aranega AE Reina A Muroo MA 1993Int J Cardiol1993,38,:1
7Periapical tissue reactions to calcium hydroxide and MTA after external root resorption as a sequela of delayed tooth replantation 显示文摘MarCo HF Panzarini SR Aranega AM 2012Dent Traumatol2012,28,4:1
8Detection of circulating alpha-actinin in non-insulin-dependent diabetics with autonomic dysfunction 显示文摘Prados J Melguizo C Aranega AE 1996Cardiology1996,87,4:1
9Periapical tissue reactions to calcium hydroxide and MTA after external root resorption as a sequela of delayed tooth replantation显示文摘Marao H F Panzarini S R Aranega A M 2012Dent Traumatol2012,284,:1
10Morphometric study of right ventricle in 32 cases of tetralogy of Fallot显示文摘Alvarez L Aranega A Contreras JA 1988Herz1988,13,:1
11Circulating alpha-actin protein in acute myocardial infarction显示文摘Aranega AE Reina A Muros MA 1993Int J Cardiol1993,1,:1
12Transgenic insights linking pitx2 and atrial arrhythmias显示文摘Franco D Chinchilla A Aranega AE 0,,01:1
13Periapical tissue reac-tions to calcium hydroxide and MTA after external root resorptionas狂 sequela of delayed tooth replantatio 显示文摘Marao HF Panzarini SR Aranega AM 2012Dent Traumatol?20122012,313,:1
14Training Health Professionals in Teaching Competences显示文摘Aranega Susanna Serrat Nuria 2016US-China Education Review(A)2016,6,3:0
15Gene regulatory networks in atrial fibrillation显示文摘Atrial fibrillation(AF)is the most frequent arrhythmogenic syndrome in humans.With an estimate incidence of1%-2%in the general population,AF raises up to almost10%-12%in 80+years.Thus,AF represents nowadays a highly prevalent medical problem generating a large economic burden.At the electrophysiological level,distinct mechanisms have been elucidated.Yet,despite its prevalence,the genetic and molecular culprits of this pandemic cardiac electrophysiological abnormality have remained largely obscure.Molecular genetics of AF familiar cases have demonstrated that single nucleotide mutations in distinct genes encoding for ion channels underlie the onset of AF,albeit such alterations only explain a minor subset of patients with AF.In recent years,analyses by means of genome-wide association studies have unraveled a more complex picture of the etiology of AF,pointing out to distinct cardiac-enriched transcription factors,as well as to other regulatory genes.Furthermore a new layer of regulatory mechanisms have emerged,i.e.,post-transcriptional regulation mediated by non-coding RNA,which have been demonstrated to exert pivotal roles in cardiac electrophysiology.In this manuscript,we aim to provide a comprehensive review of the genetic regulatory networks that if impaired exert electrophysiological abnormalities that contribute to the onset,and subsequently,on self-perpetuation of AF.Diego Franco Estefanía Lozano-Velasco Amelia Aranega 2016World Journal of Medical Genetics2016,6,1:0
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