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| 1 | Burden of Gastrointestinal Disease in the United States: 2012 Update显示文摘 | Anne F. Peery Evan S. Dellon Jennifer Lund Seth D. Crockett Christopher E. McGowan William J. Bulsiewicz Lisa M. Gangarosa Michelle T. Thiny Karyn Stizenberg Douglas R. Morgan Yehuda Ringel Hannah P. Kim Marco Dacosta DiBonaventura Charlotte F. Carroll Je | 2012 | Gastroenterology2012，，5： | 15 |
| 2 | Acute liver failure due to natural killer-like T-cell leukemia/ lymphoma: A case report and review of the Literature显示文摘尖锐的肝失败(ALF ) 是为肝移植要求立即的评估的一种医药紧急情况。我们描述与腹水,黄疸,和脑病介绍了并且被发现由于象 T 房间白血病 / 淋巴瘤一样的生来的杀手(NK ) 有 ALF 的一个病人的一个不平常的盒子。关键免疫显型是 CD2+ , CD3+ , CD7+ , CD56+ 。这诊断,在外部血和腹水基于调查结果,与肝活体检视被证实,并且是到肝移植的禁止徵候。hematologic 恶意是暴发性的肝的失败的一个不平常的原因的文学表演的评论,和那个象 NK 一样 T 房间白血病 / 淋巴瘤是一最近相对表示特性地认出了是的实体 CD3+ 和 CD56+ 。这个盒子证明那肝活体检视在与淋巴瘤 / 白血病能引起尖锐的肝失败的象 NK 一样 T 房间诊断肝的尖锐的肝失败,和那渗入的不平常的原因是必要的。 | Evan S Dellon Shannon R Morris Wozhan Tang Cherie H Dunphy Mark W Russo | 2006 | World Journal of Gastroenterology2006，12，25： | 5 |
| 3 | Eosinophilic esophagitis: Updated consensus recommendations for children and adults显示文摘 | Chris A. Liacouras Glenn T. Furuta Ikuo Hirano Dan Atkins Stephen E. Attwood Peter A. Bonis A. Wesley Burks Mirna Chehade Margaret H. Collins Evan S. Dellon Ranjan Dohil Gary W. Falk Nirmala Gonsalves Sandeep K. Gupta David A. Katzka Alfredo J. Lucendo Jo | 2011 | The Journal of Allergy and Clinical Immunology2011，，1： | 3 |
| 4 | 3T magnetic resonance neurography of pudendal nerve with cadaveric dissection correlation显示文摘AIM: To evaluate the pudendal nerve segments that could be identified on magnetic resonance neurography(MRN) before and after surgical marking of different nerve segments.METHODS: The hypothesis for this study was that pudendal nerve and its branches would be more easily seen after the surgical nerve marking. Institutional board approval was obtained. One male and one female cadaver pelvis were obtained from the anatomy board and were scanned using 3 Tesla MRI scanner using MR neurography sequences. All possible pudendal nerve branches were identified. The cadavers were then sent to the autopsy lab and were surgically dissected by a peripheral nerve surgeon and an anatomist to identify the pudendal nerve branches. Radiological markers were placed along the course of the pudendal nerve and its branches. The cadavers were then closed and rescanned using the same MRN protocol as the premarking scan. The remaining pudendal nerve branches were attempted to be identified using the radiological markers. All scans were read by an experienced musculoskeletal radiologist.RESULTS: The pre-marking MR Neurography scans clearly showed the pudendal nerve at its exit from the lumbosacral plexus in the sciatic notch, at the level of the ischial spine and in the Alcock's Canal in both cadavers. Additionally, the right hemorrhoidal branch could be identified in the male pelvis cadaver. The perineal and distal genital branches could not be identified. On post-marking scans, the markers were used as identifiable structures. The location of the perineal branch, the hemorroidal branch and the dorsal nerve to penis(in male cadaver)/clitoris(in female cadaver) could be seen. However, the visualization of these branches was suboptimal. The contralateral corresponding nerves were poorly seen despite marking on the surgical side. The nerve was best seen on axial T1W and T2W SPAIR images. The proximal segment could be seen well on 3D DW PSIF sequence. T2W SPACE was not very useful in visualization of this small nerve or its branches.CONCLUSION: Proximal pudendal nerve is easily seen on MR neurography, however it is not possible to identify distal branches of the pudendal nerve even after surgical marking. | Avneesh Chhabra Courtney A McKenna Vibhor Wadhwa Gaurav K Thawait John A Carrino Gary P Lees A Lee Dellon | 2016 | World Journal of Radiology2016，8，7： | 3 |
| 5 | Feasibility, safety, acceptability, and yield of office-based, screening transnasal esophagoscopy (with video)显示文摘 | Anne F. Peery Toshitaka Hoppo Katherine S. Garman Evan S. Dellon Norma Daugherty Susan Bream Alejandro F. Sanz Jon Davison Melissa Spacek Diane Connors Ashley L. Faulx Amitabh Chak James D. Luketich Nicholas J. Shaheen Blair A. Jobe | 2012 | Gastrointestinal Endoscopy2012，，5： | 2 |
| 6 | Diagnosis and Management of Eosinophilic Esophagitis显示文摘 | Evan S. Dellon | 2012 | Clinical Gastroenterology and Hepatology2012，，10： | 2 |
| 7 | Endoscopic Retrograde Cholangiopancreatography with Single-Balloon Enteroscopy Is Feasible in Patients with a Prior Roux-en-Y Anastomosis显示文摘 | Evan S. Dellon Geoffrey P. Kohn Douglas R. Morgan Ian S. Grimm | 2009 | Digestive Diseases and Sciences2009，，8： | 2 |
| 8 | A phenotypic analysis shows that eosinophilic esophagitis is a progressive fibrostenotic disease显示文摘 | Evan S. Dellon Hannah P. Kim Sarah L.W. Sperry David A. Rybnicek John T. Woosley Nicholas J. Shaheen | 2013 | Gastrointestinal Endoscopy2013，，： | 2 |
| 9 | Safety and Efficacy of Endoscopic Mucosal Therapy With Radiofrequency Ablation for Patients With Neoplastic Barrett’s Esophagus显示文摘 | William J. Bulsiewicz Hannah P. Kim Evan S. Dellon Cary C. Cotton Sarina Pasricha Ryan D. Madanick Melissa B. Spacek Susan E. Bream Xiaoxin Chen Roy C. Orlando Nicholas J. Shaheen | 2013 | Clinical Gastroenterology and Hepatology2013，，6： | 2 |
| 10 | Transcriptome analysis of proton pump inhibitor–responsive esophageal eosinophilia reveals proton pump inhibitor–reversible allergic inflammation显示文摘 | Ting Wen Evan S. Dellon Fouad J. Moawad Glenn T. Furuta Seema S. Aceves Marc E. Rothenberg | 2014 | The Journal of Allergy and Clinical Immunology2014，，： | 2 |
| 11 | Advances in Clinical Management of Eosinophilic Esophagitis显示文摘 | Evan S. Dellon Chris A. Liacouras | 2014 | Gastroenterology2014，，： | 2 |
| 12 | Zone of traction injury of the common peroneal nerve显示文摘 | Prasad AR Steck JK Dellon AL | 2007 | Ann Plast Surg2007，59，3： | 1 |
| 13 | A single biopsy is valid for genetic diagnosis of eosinophilic esophagitis regardless of tissue preservation or location in the esophagus显示文摘 | Dellon ES Yellore V Andreatta M | 2015 | J Gastrointestin Liver Dis2015，24，2： | 1 |
| 14 | Prevention ofulceration,amputation,and reduction of hospitalization:outcomes of a prospective multicenter trial of tibial neurol-ysis in patients with diabetic neuropathy显示文摘 | Dellon AL Muse VL Nickerson DS | 2012 | J ReconstrMicrosurg2012，28，4： | 1 |
| 15 | ' Components separation' method for closure of abdominal-wall defects:an anatomic and clinical study显示文摘 | Ramirez OM Ruas E Dellon AL | | 0，，03： | 1 |
| 16 | ACG clinical guideline: Evidenced based approach to the diagnosis and management of esophageal eosinophilia and eosinophilic esophagitis显示文摘 | Dellon ES Gonsalves N Hirano I | 2013 | Am J Gastroenterol2013，108，5： | 1 |
| 17 | Complications from silicone polymer entubulation of nerves显示文摘 | MERLE M DELLON AL CAMPBELL JN | 1989 | Microsurgery1989，10，： | 1 |
| 18 | Diabetic neuropathy:review of a surgical approach to restore sensation, relieve pain, and prevent ulceration and amputation显示文摘 | Dellon AL | 2004 | Foot Ankle Int2004，25，10： | 1 |
| 19 | Epidemiology of Eosinophilic Esophagitis显示文摘 | Evan S. Dellon | 2014 | Gastroenterology Clinics of North America2014，，： | 1 |
| 20 | Prevalence and associated predictors for patients developing chronic neuropathic pain following burns显示文摘Background:Chronic pain,unrelated to the burn itself,can manifest as a long-term complication in patients sustaining burn injuries.The purpose of this study was to determine the prevalence of chronic neuropathic pain(CNP)and compare burn characteristics between patients who developed CNP and patients without CNP who were treated at a burn center.Methods:A single-center,retrospective analysis of 1880 patients admitted to the adult burn center was performed from 1 January 2014 to 1 January 2019.Patients included were over the age of 15 years,sustained a burn injury and were admitted to the burn center.CNP was diagnosed clinically following burn injury.Patients were excluded from the definition of CNP if their pain was due to an underlying illness or medication.Comparisons between patients admitted to the burn center with no pain and patients admitted to the burn center who developed CNP were performed.Results:One hundred and thirteen of the 1880 burn patients developed CNP as a direct result of burn injury over 5 years with a prevalence of 6.01%.Patients who developed CNP were a significantly older median age(54 years vs.46 years,p=0.002),abused alcohol(29%vs.8%,p<0.001),abused substances(31%vs.9%,p<0.001),were current daily smokers(73%vs.33%,p<0.001),suffered more full-thickness burns(58%vs.43%,p<0.001),greater median percent of total body surface area(%TBSA)burns(6 vs.3.5,p<0.001),were more often intubated on mechanical ventilation(33%vs.14%,p<0.001),greater median number of surgeries(2 vs.0,p<0.001)and longer median hospital length of stay(LOS)(10 days vs.3 days,p<0.001),compared to those who did not develop CNP,respectively.Median patient follow-up was 27 months.Conclusions:The prevalence of CNP over 5 years was 6.01%in the burn center.Older ages,alcohol abuse,substance abuse,current daily smoking,greater percent of total body surface area(%TBSA)burns,third degree burns,being intubated on mechanical ventilation,having more surgeries and longer hospital LOS were associated with developing CNP following burn injury,compared to patients who did not develop CNP following burn injury. | Kevin M.Klifto A.Lee Dellon C.Scott Hultman | 2020 | Burns & Trauma2020，8，1： | 1 |
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