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9篇 您的检索式：作者名="Joseph Torresi"

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		题名	作者	年代	出处	被引量
	1	Modulation of MAPK pathways and cell cycle by replicating hepatitis B virus: Factors contributing to hepatocarcinogenesis显示文摘	Ruth Chin Linda Earnest-Silveira Bernd Koeberlein Susanne Franz Hanswalter Zentgraf Xuebin Dong Eric Gowans C.-Thomas Bock Joseph Torresi	2007	Journal of Hepatology2007，，3：	1
	2	Immunogenicity, safety and tolerability in adults of a new single-dose, live-attenuated vaccine against Japanese encephalitis: Randomised controlled phase 3 trials显示文摘	Joseph Torresi Karen McCarthy Emmanuel Feroldi Claude Méric	2010	Vaccine2010，，50：	1
	3	The virological and clinical significance of mutations in the overlapping envelope and polymerase genes of hepatitis B virus显示文摘	Joseph Torresi	2002	Journal of Clinical Virology2002，，2：	1
	4	Selection of hepatitis B virus polymerase mutants with enhanced replication by lamivudine treatment after liver transplantation显示文摘	C.-Thomas Bock Hans L. Tillmann Joseph Torresi Jürgen Klempnauer Stephen Locarnini Michael P. Manns Christian Trautwein	2002	Gastroenterology2002，，2：	1
	5	Progress in the development of preventive and therapeutic vaccines for hepatitis C virus显示文摘	Joseph Torresi Doug Johnson Heiner Wedemeyer	2011	Journal of Hepatology2011，，6：	1
	6	Reduced Antigenicity of the Hepatitis B Virus HBsAg Protein Arising as a Consequence of Sequence Changes in the Overlapping Polymerase Gene That Are Selected by Lamivudine Therapy显示文摘	Joseph Torresi Linda Earnest-Silveira Georgia Deliyannis Kristy Edgtton Hui Zhuang Stephen A Locarnini Janet Fyfe Tina Sozzi David C Jackson	2002	Virology2002，，2：	1
	7	Enhanced apoptosis in post-liver transplant hepatitis C:Effects of virus and immunosuppressants显示文摘Hepatitis C(HCV)-infected patients have a poorer survival post-liver transplantation compared to patients transplanted for other indications,since HCV recurrence post-transplant is universal and commonly follows an aggressive course.There is increasing evidence that in the non-transplant setting,induction of hepatocyte apoptosis is one of the main mechanisms by which HCV drives liver inflammation and fibrosis,and that HCV proteins directly promote apoptosis.Recent studies have shown that post-liver transplant,there is a link between high levels of HCV replication,enhanced hepatocyte apoptosis and the subsequent development of rapidly progressive liver fibrosis.Although the responsible mechanisms remain unclear,it is likely that immunosuppressive drugs play an important role.It is well known that immunosuppressants impair immune control of HCV,thereby allowing increased viral replication.However there is also evidence that immunosuppressants may directly induce apoptosis and this may be facilitated by the presence of high levels of HCV replication.Thus HCV and immunosuppressants may synergistically interact to further enhance apoptosis and drive more rapid fibrosis.These findings suggest that modulation of apoptosis within the liver either by changing immunosuppressive therapy or the use of apoptosis inhibitors may help prevent fibrosis progression in patients with post-transplant HCV disease.	Eu Jin Lim Ruth Chin Peter W Angus Joseph Torresi	2012	World Journal of Gastroenterology2012，18，18：	1
	8	Neutralizing antibodies in patients with chronic hepatitis C infection treated with (Peg)-interferon/ribavirin显示文摘	Irene Boo Alexandra E. Fischer Doug Johnson Ruth Chin Maxine Giourouki Mandvi Bharadwaj Scott Bowden Joseph Torresi Heidi Drummer	2007	Journal of Clinical Virology2007，，4：	1
	9	Progress in the development of preventive and therapeutic vaccines for hepatitis C virus显示文摘	Joseph Torresi Doug Johnson Heiner Wedemeyer	2011	Journal of Hepatology2011，，6：	1

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