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| 1 | Treatment of K562 cells with 1,25-dihydroxyvitamin D3induces distinct alterations in the expression of apoptosis-related genes Bcl-2,BAX,Bcl(xL),and p21显示文摘 | Kizildag S Ates H Kizildag S | 2010 | Ann Hematol2010，89，1： | 1 |
| 2 | Effects of imazamox on soil carbon and nitrogen mineralization un- der Mediterranean climate 显示文摘 | Kizildag N Sagliker H Cenkseven S | 2014 | Turkish Journal of Agricul- ture and Forestry2014，38，3： | 1 |
| 3 | Microstatellite instability in early-onset breast cancer显示文摘 | Ozer E Yuksel E Kizildag S | 2002 | Pathol Res Pract2002，198，8： | 1 |
| 4 | Interferon gamma and lipopolysaccharide upregulate TNF-related apoptosis-inducing ligand expression in routine microglia 显示文摘 | Genc S Kizildag S Genc K Ates H Atabey N | 2003 | Immunol Lett2003，85，3： | 1 |
| 5 | Methamphetamine induces oligodendroglial cell death in vitro 显示文摘 | Genc K Genc S Kizildag S | 2003 | Brain Res2003，982，1： | 1 |
| 6 | Analysis of cytotoxic T lymphocyte antigen-4(CTLA-4)exon 1polymorphism in patients with type 1 diabetes mellitus in a Turkishpopulation显示文摘 | Genc S Genc K Sercan O Kizildag S Gulay Z Atay T | 2004 | J Pediatr Endocrinol Metab2004，17，5： | 1 |
| 7 | beta-catenin gene mutation in invasive ductal breast cancer显示文摘 | Kizildag S Zengel B Vardar E | 2008 | J Buon2008，13，4： | 1 |
| 8 | Beta-catenin gene mutation in invasive ductal breast cancer显示文摘 | Kizildag S Zengel B Vardar E | 2008 | J BUON2008，13，4： | 1 |
| 9 | The role of dopamine 2 receptor gene and personality characteristics in alcohol dependence in Turkish population显示文摘 | Kitis A Akvardar Y Eresen C Kizildag S Akpulat U Yildirimcan S Alptekin K Akdede BB Arkar H | | 0，，04： | 1 |
| 10 | Relationship between periodontitis and cardiovascular diseases: A literature review显示文摘Periodontitis and cardiovascular disease have a complex etiology and genetics and share some common risk factors(i.e., smoking, age, diabetes, etc.). In recent years, the relationship between periodontal disease and cardiovascular disease has been investigated extensively. This research mostly focused on the fact that periodontitis is an independent risk factor for cardiovascular disease. Our aim in this article is to investigate the etiological relationship between periodontal disease and cardiovascular disease and the mechanisms involved in this association. According to the current literature, it is concluded that there is a strong relationship between these chronic disorders. | Alper Kizildag Taner Arabaci Gulnihal Emrem Dogan | 2014 | World Journal of Stomatology2014，3，1： | 0 |
| 11 | Do PON1-Q192R and PON1-L55M polymorphisms modify the effects of hypoxic training on paraoxonase and arylesterase activity?显示文摘Background:Low levels of antioxidant paraoxonase 1(PON 1)enzyme activity,PON1-Q192R polymorphism(a glutamine(Q)to arginine(R)substitution at position 192),PON1-L55M polymorphism(a leucine(L)to methionine(M)substitution at position 55),and oxidized low-density lipoprotein(oxLDL)are risk factors for coronary heart disease.Aerobic exercise improves PON1 activity,but the effects of hypoxic exercise are yet unclear.The aim of this study was to determine the effects of hypoxic underwater rugby training on PON1 activity and oxLDL levels and the role of the mentioned polymorphisms.Methods:Serum PON1 and arylesterase activities(ARE),PON1,PON3,and oxLDL protein levels(by using the enzyme-linked immunosorbent assays)were determined in an athletic group(42 trained male underwater rugby players;age=21.7±4.2 years,mean±SD)and a control group(43 sedentary men;age=23.9±3.2 years).The polymorphisms were determined from genomic DNA samples.Results:PON1 activity(25.1%,p=0.052),PON3(p<0.001),and oxLDL(p<0.001)of the athletic group,including most genotype groups,were higher than those of the control group.In comparison to the controls,PON1 activity levels(p=0.005)of the PON1-Q192R homozygote QQ genotype group and PON1 activity levels(30%,p=0.116)of the PON1-L55M homozygote LL genotype group were higher,whereas ARE activity values of athletic R allele carrier(Rc=QR+RR)(p=0.005)and LL group(p=0.002)were lower than the control genotype groups related to their polymorphisms.Conclusion:Hypoxic training can cause(1)significant oxidative stress,including oxLDL,and an antioxidant response(increase in PON1 activity and PON3),(2)differences in the activity of PON1 and ARE,which are modified by PON1-Q192R and PON1-L55M polymorphisms,respectively,and(3)improvements in PON1 activity of QQ and LL groups.However,hypoxic training can cause a disadvantage of LL and Rc groups for ARE. | Oya Yigittürk Faruk Turgay Servet Kizildag DuaÖzsoylu Görkem Aybars Balci | 2023 | Journal of Sport and Health Science2023，12，2： | 0 |
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