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| 1 | Endovascular technique using a snare and suture for retrieving a migrated peripherally inserted central catheter in the left pulmonary artery显示文摘We report a successful endovascular technique using a snare with a suture for retrieving a migrated broken peripherally inserted central catheter(PICC)in a chemotherapy patient.A 62-year-old male received monthly chemotherapy through a central venous port implanted into his right subclavian area.The patient completed chemotherapy without complications 1 mo ago;however,he experienced pain in the right subclavian area during his last chemotherapy session.Computed tomography on that day showed migration of a broken PICC in his left pulmonary artery,for which the patient was admitted to our hospital.We attempted to retrieve the ectopic PICC through the right jugular vein using a gooseneck snare,but were unsuccessful because the catheter was lodged in the pulmonary artery wall.Therefore,a second attempt was made through the right femoral vein using a snare with triple loops,but we could not grasp the migrated PICC.Finally,a string was tied to thetop of the snare,allowing us to curve the snare toward the pulmonary artery by pulling the string.Finally,the catheter body was grasped and retrieved.The endovascular suture technique is occasionally extremely useful and should be considered by interventional cardiologists for retrieving migrated catheters. | Hiroki Teragawa Takashi Sueda Yuichi Fujii Hiroaki Takemoto Yasushi Toyota Shuichi Nomura Keigo Nakagawa | 2013 | World Journal of Cardiology2013，5，9： | 9 |
| 2 | Coronary spasm: It's common, but it's still unsolved显示文摘Coronary spasm is caused by a transient coronary nar -rowing due to the constriction of epicardial coronary artery, which leads to myocardial ischemia. More than 50 years have passed since the first recognition of coronary spasm, and many findings on coronary spasm have been reported. Coronary spasm has been considered as having pivotal roles in the cause of not only rest angina but also exertional angina, acute coronary syndrome, and heart failure. In addition, several new findings of the mechanism of coronary spasm have emerged recently. The diagnosis based mainly on coronary angiography and spasm provo-cation test and the mainstream treatment with a focus on a calcium-channel blocker have been established. At a glance, coronary spasm or vasospastic angina(VSA) has become a common disease. On the contrary, there are several uncertain or unsolved problems regarding coronary spasm, including the presence of medically refractory coronary spasm(intractable VSA), or an appropriate use of implantable cardioverter defibrillator in patients with cardiac arrest who have been confirmed as having coronary spasm. This editorial focused on coronary spasm, including recent topics and unsolved problems. | Hiroki Teragawa Chikage Oshita Tomohiro Ueda | 2018 | World Journal of Cardiology2018，10，11： | 7 |
| 3 | Case of angina pectoris at rest and during effort due to coronary spasm and myocardial bridging显示文摘We present a case of a 71-year-old male who hadchest symptoms at rest and during effort. He had felt chest oppression during effort for 1 year,and his chest symptoms had recently worsened. One month before admission he felt chest squeezing at rest in the early morning. He presented at our institution to evaluate his chest symptoms. Electrocardiography and echocardiography failed to show any specific changes. Because of the possibility that his chest symptoms were due to myocardial ischemia,he was admitted to our institution for coronary angiography(CAG). An initial CAG showed mild atherosclerotic changes in the proximal segment of the left anterior descending coronary artery(LAD) and mid-segment of the left circumflex coronary artery. Subsequent spasm provocation testing using acetylcholine revealed a bilateral coronary vasospasm,which was relieved after the intracoronary infusion of nitroglycerin. Finally,a CAG showed myocardial bridging(MB) of the mid-distal segments of the LAD. Fractional flow reserve using the intravenous administration of adenosine triphosphate was positive at 0.77,which jumped up to 0.90 through the myocardial bridging segments when the pressure wire was pulled back. Thus,coronary vasospasm and MB might have contributed to his chest symptoms at rest and during effort. Interventional cardiologists should consider the presence of MB as a potential cause of myocardial ischemia. | Hiroki Teragawa Yuichi Fujii Tomohiro Ueda Daiki Murata Shuichi Nomura | 2015 | World Journal of Cardiology2015，7，6： | 3 |
| 4 | 在有 vasospastic 咽峡炎的病人的冠的 microvascular 功能的评估显示文摘 AIM: To investigate endothelium-dependent and independent coronary microvascular functions in patients with vasospastic angina (VSA). METHODS: Thirty-six patients with VSA (30 men and 6 women; mean age, 58 years) were enrolled in this study. VSA was defined as ≥ 90% narrowing of the epicardial coronary arteries on angiography performed during a spasm provocation test, presence of chest pain, and/or ST-segment deviation on an electrocardiogram (ECG). Patients (n = 36) with negative spasm provocation test results and those matched for age and sex were enrolled as a control group (nonVSA group). Low-dose acetylcholine (ACh; 3 μg/min) was infused into the left coronary ostium for 2 min during the spasm provocation test. Following the spasm provocation test, nitroglycerin (0.2 mg) was administered intracoronally. Coronary blood flow (was calculated from quantitativeangiography and Doppler flow velocity measurements, and the coronary flow reserve was calculated as the ratio of coronary flow velocity after injection of adenosine triphosphate (20 μg) to the baseline value. Changes in the coronary artery diameter in response to ACh and nitroglycerin infusion were expressed as percentage changes from baseline measurements. RESULTS: Body mass index was significantly lower in the VSA group than in the nonVSA group. The fre- quency of conventional coronary risk factors and the rate of statin use were similar between the 2 groups. The left ventricular ejection fraction as evaluated by echocardiography was similar between the 2 groups. The duration of angina was 9 ± 2 mo. The results of blood chemistry analysis were similar between the 2 groups. Low-dose ACh did not cause coronary spasms. The change in coronary artery diameter in response to ACh was lower in the VSA group (-1.4% ± 9.3%) than in the nonVSA group (3.1% ± 6.5%, P < 0.05), whereas nitroglycerin-induced coronary artery dilatation and coronary blood flow increase in response to ACh or coronary flow reserve did not differ significantly between the 2 groups. CONCLUSION: These findings suggest that microvascular coronary function may be preserved despite endothelial dysfunction of the epicardial coronary arteries in patients with VSA. | Hiroki Teragawa Naoya Mitsuba Ken Ishibashi Kenji Nishioka Satoshi Kurisu Yasuki Kihara | 2013 | World Journal of Cardiology2013，5，1： | 2 |
| 5 | Impaired coronary microvascular endothelial function in men with metabolic syndrome显示文摘AIM: To assess coronary endothelial function of conduit and resistance vessels in patients with metabolic syndrome (MS). METHODS: Seventy-eight men (mean age, 57 years) with chest pain and angiographically normal coronary arteries were included in the study. Patients with coronary spastic angina were excluded. Changes in coronary artery diameter and coronary blood flow (CBF) in response to acetylcholine (ACh) were determined using quantitative coronary angiography and Doppler velocity measurements. Coronary flow reserve was calculated as the ratio of coronary blood velocity after adenosine triphosphate infusion relative to baseline values. Patients were divided into two groups based on the presence or absence of MS. RESULTS: There were 24 patients in the MS group (31%). The increase in CBF in response to ACh infusion was impaired in the MS group (P < 0.0001) compared to the non-MS group, whereas changes in coronary artery diameter in response to ACh infusion did not differ between the two groups. Multivariate regression analysis revealed that MS was a significant factor associated with the lesser change in CBF induced by ACh infusion at 30 μg/min (P < 0.0001, r 2 = 0.46). CONCLUSION: Coronary endothelial dysfunction was present at the level of resistance vessels but not conduit vessels in the MS patients included in our study. | Hiroki Teragawa Naoya Mitsuba Kenji Nishioka Kentaro Ueda Shingo Kono Yukihito Higashi Kazuaki Chayama Yasuki Kihara | 2010 | World Journal of Cardiology2010，2，7： | 2 |
| 6 | Vasomotility and Nitric Oxide Bioactivity of the Bridging Segments of the Left Anterior Descending Coronary Artery显示文摘 | Nobuo Shiode Masaya Kato Hiroki Teragawa Tadakatsu Yamada Hidekazu Hirao Katsuhiko Nomura Nobuo Sasaki Togo Yamagata Hideo Matsuura Goro Kajiyama | 1998 | The American Journal of Cardiology1998，，3： | 1 |
| 7 | The preventive effect of magnesium on coronary spasm in patients with vasospastic angina显示文摘 | Teragawa H Kato M Yamagata T | 2000 | Chest2000，118，6： | 1 |
| 8 | Relationship between endothelial function in the coronary and brachial arteries 显示文摘 | Teragawa H Ueda K Matsuda K | 2005 | Clin Cardiol2005，28，10： | 1 |
| 9 | Relation between C reactive protein concentrations and coronary microvascular function 显示文摘 | Teragawa H Fukuda Y Matsuda K | 2004 | Heart2004，90，7： | 1 |
| 10 | Mechanisms responsible for vasodilationupon magnesium infusion in vivo:clinical evidence显示文摘 | Teragawa H Matsuura H Chayama K | 2002 | Magnes Res2002，15，34： | 1 |
| 11 | Relation be- tween C-reactive protein concentrations and coronary micro- vascular endothelial function 显示文摘 | Teragawa H Fukuda Y Matsuda K | 2004 | Heart2004，90，7： | 1 |
| 12 | Tetrahdrobiopterin re- stores endothelial function of coronary arteries in patients with hy- perholes - terolaemia 显示文摘 | Fukuda Y Teragawa H Matsuda K | 2002 | Heart2002，87，： | 1 |
| 13 | Myocardial bridging increases the risk of coronary spasm显示文摘 | Teragawa H Fukuda Y Matsuda K Hirao H Higashi Y Yamagata T el al | 2003 | Clin Cardiol2003，26，： | 1 |
| 14 | Adverse effects of interferon on the cardiovascular system in patients with chronic hepatitis C 显示文摘 | Teragawa H Hondo T Amano H | 1996 | Jpn Heart J1996，37，6： | 1 |
| 15 | Usefulness of flow-mediated dilation of the brachial artery and/or the intima-media thickness of the carotid artery in predicting coronary narrowing in patients suspected of having coronary artery disease显示文摘 | Kato M Kurokawa J | 2001 | Am J Cardiol2001，88，10： | 1 |
| 16 | Relation between C reac- tive protein concentrations and coronary microvascular endothelial function显示文摘 | Teragawa H Fukuda Y Matsuda K | 2004 | Heart2004，90，7： | 1 |
| 17 | Leptin causes nitric-oxide independent coronary artery vasodilation in humans显示文摘 | Matsuda K Teragawa H Fukuda Y | 2003 | Hypertens Res2003，26，： | 1 |
| 18 | Magnesium causes nitric oxide independent coronary artery vasodilation in humans显示文摘 | Teragawa H Kato M Yamagata T | 2001 | Heart2001，86，2： | 1 |
| 19 | Adverse effects of interferon on the cardiovascular system in patients with chronic hepatitis C显示文摘 | Teragawa H Hondo T Amano H | 1996 | Jpn Heart J1996，37，6： | 1 |
| 20 | Usefulness of flowmediated dilation of the brachial artery and/or the intima media thickness of the carotid artery in predicting coronary narrowing in patients suspected of having coronary artery disease显示文摘 | Teragawa H Kato M Kurokawa J | | 0，，10： | 1 |
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