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6篇 您的检索式：作者名="Chisuwa"

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		题名	作者	年代	出处	被引量
	1	Living related liver transplantation in adults显示文摘	Kawasaki S Makuuchi M Matsunami H Hashikura Y Ikegami T. Nakazawa Y Chisuwa H. Terada M Miyagawa S		0，，：	1
	2	Living liver donation:preoperative assessment,anatomic considerations,and long-term outcome显示文摘	Chisuwa H Hashikura Y Mita A	2003	Transplantation2003，75，10：	1
	3	Living liver donation: preoperative assessment, anatomic considerations, and long-term outcome1显示文摘	Hisanao Chisuwa Yasuhiko Hashikura Atsuyoshi Mita Shin-ichi Miyagawa Masaru Terada Toshihiko Ikegami Yuichi Nakazawa Koichi Urata Shiro Ogino Seiji Kawasaki	2003	Transplantation2003，，10：	1
	4	Life-threat- ening veno-occlusive disease after living-related liver trans- plantation显示文摘	NAKAZAWA Y CHISUWA I-I MITA A	2003	Transplantation2003，75，5：	1
	5	Living Related Liver Transplantation in Adults显示文摘	Seiji Kawasaki Masatoshi Makuuchi Hidetoshi Matsunami Yasuhiko Hashikura Toshihiko Ikegami Yuichi Nakazawa Hisanao Chisuwa Masaru Terada Shin-ichi Miyagawa	1998	Annals of Surgery1998，，2：	1
	6	Relevance of ADAMTS13 to liver transplantation and surgery显示文摘A disintegrin-like and metalloproteinase with thrombospondin type-1 motifs 13(ADAMTS13) specifically cleaves unusually-large von Willebrand factor(VWF) multimers under high shear stress,and down-regulates VWF function to form platelet thrombi.Deficiency of plasma ADAMTS13 activity induces a life-threatening systemic disease,termed thrombotic microangiopathy(TMA) including thrombotic thrombocytopenic purpura(TTP).Children with advanced biliary cirrhosis due to congenital biliary atresia sometimes showed pathological features of TMA,with a concomitant decrease of plasma ADAMTS13 activity.Disappearance of their clinical findings of TTP after successful liver transplantation suggested that the liver is a major organ producing plasma ADAMTS13.In situ hybridization analysis showed that ADAMTS13 was produced by hepatic stellate cells.Subsequently,it was found that ADADTS13 was not merely responsible to development of TMA and TTP,but also related to some kinds of liver dysfunction after liver transplantation.Ischemia-reperfusion injury and acute rejection in liver transplant recipients were often associated with marked decrease of ADAMTS13 and concomitant formation of unusually large VWF multimers without findings of TMA/TTP.The similar phenomenon was observed also in patients who underwent hepatectomy for liver tumors.Imbalance between ADAMTS13 and VWF in the hepatic sinusoid might cause liver damage due to microcirculatory disturbance.It can be called as 'local TTP like mechanism' which plays a crucial role in liver dysfunction after liver transplantation and surgery.	Saiho Ko Hisanao Chisuwa Masanori Matsumoto Yoshihiro Fujimura Eiji Okano Yoshiyuki Nakajima	2015	World Journal of Hepatology2015，7，13：	0

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