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111篇 您的检索式:作者名="Williams TA"
    题名 作者 年代 出处 被引量
1初级保健中原发性醛固酮增多症的患病率和临床表现显示文摘原发性醛固酮增多症(primaryaldosteronism,PA)是一种异质性疾病,以高血压和相对自主于肾素血管紧张素系统产生过量醛固酮为特征。与心血管病风险相似的原发性高血压患者相比,PA患者发生心脑血管并发症的风险以及代谢综合征患病率增加,提示正确诊断PA的重要性。Monticone S Burrello J Tizzani D Bertello C Viola A Buffolo F Gabetti L Mengozzi G Williams TA Rabbia F Veglio F Mulatero P 练桂丽 叶鹏 2017中华高血压杂志2017,25,5:58
2Insights into gastric neuroendocrine tumors burden显示文摘Type 1 gastric neuroendocrine tumors(g NETs)are usually small lesions,restricted to mucosal and sub-mucosal layers of corpus and fundus,with low aggressive behavior,for the majority of cases.Nevertheless,some cases present aggressive behavior.The increasing incidence of g NETs brings together a new relevant problem:how to identify potentially aggressive type 1 g NETs.The challenging problem seems to be finding out signs or features able to predict potentially aggressive cases,allowing a tailored approach,since the involved societies dedicated to provide guidelines for management of these neoplasms apparently failed in producing staging systems able to accurately predict prognosis of these tumors.Additionally,it is also important to try to find out explanations for increasing incidence,as well as to identify potential targets aiming to reach better control of this neoplasia.Here,we discuss potential pathways implicated in aggressive behavior,as well as new strategies to improve clinical management of these tumors.Taíssa Maíra Thomaz Araújo Williams Fernandes Barra André Salim Khayat Paulo Pimentel de Assumpcao 2017Chinese Journal of Cancer Research2017,29,2:7
3三级高血压单位5100例患者低血钾症与原发性醛固酮增多症的发病率显示文摘常伴有低血钾症的原发性醛固酮增多症(原醛)被认为是一个少见病。广泛筛查高血压患者发现正常血钾的原醛发生率增加。许多研究报道原醛低血钾症的发生率,相反,低血钾症患者中原醛的发生率并不明确。在该回顾性观察研究,研究者探讨了高血压患者中低钾的发生率以及低血钾症合并高血压患者中原醛的发生率。赵狄(译) 刘莉(审校) Burrello J Monticone S Losano I Cavaglià G Buffolo F Tetti M Covella M Rabbia F Veglio F Pasini B Williams TA Mulatero P 2020中华高血压杂志2020,28,3:7
4Hereditary gastric cancer:Three rules to reduce missed diagnoses显示文摘Gastric cancer remains one of the most lethal cancers.The incidence and mortality rates are quite similar.The main reason for the high mortality is diagnosis at advanced stages of disease,when treatment options are poor.One of the supposed strategies to overcome late-stage diagnosis is identifying people at high risk with the aim of establishing rigorous clinical control,including routine endoscopy and biopsies.Hereditary gastric cancer(HGC)syndromes,though representing a sizeable group to monitor for prevention or,at least,for early diagnosis,are apparently extremely rare.The low rate of HGC diagnosis might be related to the low rates of suspicion,insufficient familiarity about clinical diagnosis criteria,and the supposed conditional necessity of a molecular diagnosis.In this review,we will discuss simple measures to increase HGC diagnosis by applying three rules that might provide an opportunity for precision care to benefit the families affected by this disease.Paula Assumpcao Taíssa Araújo André Khayat Geraldo Ishak Sidney Santos Williams Barra Joao Felipe Acioli Benedito Rossi Paulo Assumpcao 2020World Journal of Gastroenterology2020,26,13:3
5Trabeculecto- my with mitomycin C in fractory glaucoma associated with non- necrotizing anterior scleritis 显示文摘Williams TA Sii F Chaing M Murray PI Shah P 2009Ocal Immunol Inflamm2009,17,5:1
6Research Methodolo- gy in Management: Current Practices, Trends, and Implica- tions for Future Research显示文摘Scandura TA Williams EA 2000The Academy of Management Journal2000,43,6:1
7Validation of a quality assurance program for autologous cultured chondrocyte implantation 显示文摘Mayhew TA Williams GR Senica MA 1998Tissue Eng1998,4,3:1
8Mineralocorticoid receptor blockade in the protection of target organ damage显示文摘Mulatero P Milan A Williams TA 0,,01:1
9Hollow silica and silica-boron nano/microparticles for contrast-enhanced ultrasound to detect small tumors显示文摘Alexander Liberman H. Paul Martinez Casey N. Ta Christopher V. Barback Robert F. Mattrey Yuko Kono Sarah L. Blair William C. Trogler Andrew C. Kummel Zhe Wu 2012Biomaterials2012,,20:1
10Myocyte-enriched calcineurin-interacting protein,MCIPI,inhibits cardiac hypertrophy in vivo显示文摘Rothermel BA McKinsey TA Vega RB Nieol RL Mammen P Yang J Antos CL Shelton JM Bassel-Duby R Olson EN Williams RS 2001Proo Nail Aead Sei U S A2001,98,6:1
11Aquaretic Effect of Lixivaptan,an Oral,Non-Peptide,Selective V2 Receptor Vasopressin Antagonist,in New York Heart Associaion Functional Class Ⅱ and Ⅲ Chronic Heart Failure Patients显示文摘William TA Alireza AS Robert WS 2006Am Coll Cardiol2006,47,8:1
12Reversal of a paradoxical reaction to midazolam with flumazenil 显示文摘Thurston TA Williams CG Foshee SL 1996Anesth Analg1996,83,:1
13IL-10 regulates liver pathology in acute murine Schistosomiasis mansoni but is not required for immune down-modulation of chronic disease显示文摘Wynn TA Cheever AW Williams ME 1998J Immunol1998,160,9:1
14Regulation of Wnt/lS-catenin signaling within and from osteocytes 显示文摘Burgers TA Williams BO 2013Bone2013,54,:1
15A peptide representing the carboxyl-terminal tail of the met receptor inhibits kinase activity and invasive growth显示文摘Bardelli A Longati P Williams TA 1999[Journal Article]Journal of Biological Chemistry1999,274,29:1
16Regulation of Wnt/beta-catenin signaling within and from osteocytes显示文摘BURGERS TA WILLIAMS BO 2013Bone2013,54,2:1
17D-Ribose aids heart failure patients with preserved ejection fraction and diastolic dysfunction: a pilot study显示文摘Melike B St Cyr JA William TA 2015Ther Adv Cardiovasc Dis2015,9,3:1
18Subclavian flap aortoplasty: still a safe, reproducible, and effective treatment for infant coarctation 显示文摘Barreiro CJ Ellison TA Williams JA et ol 2007Eur J Cardiothorac Surg2007,31,4:1
19Ge- netic spectrum and clinical correlates of somatic mutations in aldosterone-producing adenoma 显示文摘Fernandes-Rosa FL Williams TA Riester A 2014Hypertension2014,64,:1
20Relationship between admission serum sodium concentration and clinical outcomes in patients hospitalized for heart failure: an analysis from the OPTIMIZE-HF registry 显示文摘Mihai G William TA Nancy MA 2007Eur Heart J2007,28,8:1
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