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| 1 | Estrogen and insulin synergistically promote endometrial cancer progression via crosstalk between their receptor signaling pathways显示文摘Objective: Despite evidence that estrogens and insulin are involved in the development and progression of many cancers, their synergistic role in endometrial carcinoma(EC) has not been analyzed yet.Methods: Here, we investigated how estrogens act synergistically with insulin to promote EC progression. Cell growth in vitro and in vivo, effects of estradiol and insulin on apoptosis and cell cycle distribution, and expression and activation of estrogen receptor(ER), insulin receptor(InsR), and key proteins in the PI3K and MAPK pathways were examined after combined stimulation with estradiol and insulin.Results: Compared to EC cells treated with estradiol or insulin alone, those treated with both estradiol and insulin exhibited stronger stimulation. Estradiol significantly induced phosphorylation of InsR-β and IRS-1, whereas insulin significantly induced phosphorylation of ER-α. In addition, treatment with both insulin and estradiol together significantly increased the expression and phosphorylation of Akt, MAPK, and ERK. Notably, InsR-β inhibition had a limited effect on estradiol-dependent proliferation,cell cycle, and apoptosis, whereas ER-α inhibition had a limited insulin-dependent effect, in EC cell lines. Insulin and estradiol individually and synergistically promoted EC xenograft growth in mice.Conclusions: Estrogen and insulin play synergistic roles in EC carcinogenesis and progression by activating InsR-β and ER-α,promoting a crosstalk between them, and thereby resulting in the activation of downstream PI3K/Akt and MAPK/ERK signaling pathways. | Wenyan Tian Fei Teng Jinping Gao Chao Gao Guoyan Liu Yanfang Zhang Shizhu Yu Wei Zhang Yingmei Wang Fengxia Xue | 2019 | Cancer Biology & Medicine2019，16，1： | 6 |
| 2 | Characterization of uncultivated magnetotactic bacteria from the sediments of Yuehu Lake, China显示文摘Marine magnetotactic bacteria were collected from the intertidal sediments of Yuehu Lake(China), where their abundance reached 103–104 ind./cm3. Diverse morphotypes of magnetotactic bacteria were observed, including cocci and oval, vibrio-, spirillum-, rod-, elliptical-, handle- and bar-shaped forms. The magnetococci were the most abundant, and had flagella arranged in parallel within a bundle. The majority of magnetosomes were arranged in one, two or multiple chains, although irregular arrangements were also evident. All the results of high-resolution transmission electron microscopy(HRTEM) analysis show that magnetosome crystals were composed of Fe3O4, and their morphology was specific to particular cell morphotypes. By the 16 S r RNA gene sequence analysis, we found fourteen operational taxonomic units(OTUs) which were related to magnetotactic bacteria. Among these, thirteen belonged to the Alphaproteobacteria and one to the Gammaproteobacteria.Compared with known axenic and uncultured marine magnetotactic bacteria, the 16 S r RNA gene sequences of most magnetotactic bacteria collected from the Yuehu Lake exhibited sequence identities ranging from 90.1% to96.2%(<97%). The results indicate that microbial communities containing previously unidentified magnetotactic bacteria occur in the Yuehu Lake. | DU Haijian ZHANG Rui ZHANG Wenyan XU Cong CHEN Yiran PAN Hongmiao ZHOU Ke WU Long-fei XIAO Tian | 2017 | Acta Oceanologica Sinica2017，36，2： | 3 |
| 3 | Electrochemical performance of La_(2)NiO_(4+δ)-Ce_(0.55)La_(0.45)O_(2−δ)as a promising bifunctional oxygen electrode for reversible solid oxide cells显示文摘In this work,La_(2)NiO_(4+δ)-Ce_(0.55)La_(0.45)O_(2−δ)(denoted as LNO-xLDC)with various LDC contents(x=0,10,20,30,and 40 wt%)were prepared and evaluated as bifunctional oxygen electrodes for reversible solid oxide cells(RSOCs).Compared with the pure LNO,the optimum composition of LNO-30LDC exhibited the lowest polarization resistance(Rp)of 0.53 and 0.12Ω·cm^(2)in air at 650 and 750℃,respectively.The enhanced electrochemical performance of LNO-30LDC oxygen electrode was mainly attributed to the extended triple phase boundary and more oxygen ionic transfer channels.The hydrogen electrode supported single cell with LNO-30LDC oxygen electrode displayed peak power densities of 276,401,and 521 mW·cm^(−2)at 700,750,and 800℃,respectively.Moreover,the electrolysis current density of the single cell demonstrated 526.39 mA·cm^(−2) under 1.5 V at 800℃,and the corresponding hydrogen production rate was 220.03 mL·cm^(−2)·h^(−1).The encouraging results indicated that LNO-30LDC was a promising bifunctional oxygen electrode material for RSOCs. | Pengzhang Li Wei Yang Chuanjin Tian Wenyan Zhao Zhe Lü Zhipeng Xie Chang-An Wang | 2021 | Journal of Advanced Ceramics2021，10，2： | 2 |
| 4 | Bacterial and archaeal community structures in the Arctic deepsea sediment显示文摘Microbial community structures in the Arctic deep-sea sedimentary ecosystem are determined by organic matter input, energy availability, and other environmental factors. However, global warming and earlier ice-cover melting are affecting the microbial diversity. To characterize the Arctic deep-sea sediment microbial diversity and its relationship with environmental factors, we applied Roche 454 sequencing of 16 S r DNA amplicons from Arctic deep-sea sediment sample. Both bacterial and archaeal communities' richness, compositions and structures as well as taxonomic and phylogenetic affiliations of identified clades were characterized. Phylotypes relating to sulfur reduction and chemoorganotrophic lifestyle are major groups in the bacterial groups; while the archaeal community is dominated by phylotypes most closely related to the ammonia-oxidizing Thaumarchaeota(96.66%) and methanogenic Euryarchaeota(3.21%). This study describes the microbial diversity in the Arctic deep marine sediment(>3 500 m) near the North Pole and would lay foundation for future functional analysis on microbial metabolic processes and pathways predictions in similar environments. | LI Yan LIU Qun LI Chaolun DONG Yi ZHANG Wenyan ZHANG Wuchang XIAO Tian | 2015 | Acta Oceanologica Sinica2015，34，2： | 2 |
| 5 | Effects of electric-acoustic and acoustic-electric conversions of transducers on acoustic logging signal显示文摘Based on an acoustic logging transmission network and the engineering pattern of a sliding wave in acoustic logging, analysis and calculations have been performed in a study of the effects of the electric-acoustic and acoustic-electric conversions of the transducers on the acoustic logging signal. The results show that acoustic-electric conversion through the transducer can cause not only a serious disturbance in the signal amplitude, but also an apparent transmission delay. For engineering applications, the amplitude variation and transmission delay must be accounted for in a practical analysis of the acoustic logging signal in rocks. The results also show that with enhanced understanding and proper justification, the error caused by the acoustic-electric conversion can be significantly reduced in evaluation of the cement bond quality of a cased well, and the accuracy of rock porosity calculated using the measured acoustic velocity can be increased. | FA Lin XIE WenYan TIAN Yong ZHAO MeiShan MA Li DONG DaQun | 2012 | Chinese Science Bulletin2012，57，11： | 2 |
| 6 | Hyperactive PI3Kδ predisposes naive T cells to activation via aerobic glycolysis programs显示文摘Activated phosphoinositide 3-kinaseδsyndrome(APDS)is an autosomal-dominant combined immunodeficiency disorder resulting from pathogenic gain-of-function(GOF)mutations in the PIK3CD gene.Patients with APDS display abnormal T cell homeostasis.However,the mechanisms by which PIK3CD GOF contributes to this feature remain unknown.Here,with a cohort of children with PIK3CD GOF mutations from multiple regions of China and a corresponding CRISPR/Cas9 gene-edited mouse model,we reported that hyperactive PI3Kδdisrupted TNaive cell homeostasis in the periphery by intrinsically promoting the growth,proliferation,and activation of TNaive cells.Our results showed that PIK3CD GOF resulted in loss of the quiescence-associated gene expression profile in naive T cells and promoted naive T cells to overgrow,hyperproliferate and acquire an activated functional status.Naive PIK3CD GOF T cells exhibited an enhanced glycolytic capacity and reduced mitochondrial respiration in the resting or activated state.Blocking glycolysis abrogated the abnormal splenic T cell pool and reversed the overactivated phenotype induced by PIK3CD GOF in vivo and in vitro.These results suggest that enhanced aerobic glycolysis is required for PIK3CD GOF-induced overactivation of naive T cells and provide a potential therapeutic approach for targeting glycolysis to treat patients with APDS as well as other immune disorders. | Yanjun Jia Qiuyun Yang Yanping Wang Wenyan Li Xuemei Chen Tao Xu Zhirui Tian Minxuan Feng Liang Zhang Wenjing Tang Na Tian Lina Zhou Wenxia Song Xiaodong Zhao | 2021 | Cellular & Molecular Immunology2021，18，7： | 2 |
| 7 | Archaeal community structure in sediments from a seamount in the Mariana Volcanic Arc显示文摘Seamounts are subsurface mountains in the ocean. Examination of the abundance and distribution of Archaea in seamount ecosystems may provide a better understanding of their ecological functions. Most studies of marine archaeal assemblages in seamount area have focused on hydrothermal vents or ferromanganese crusts. We investigated the archaeal communities from a seamount of the Mariana Volcanic Arc, in the tropical western Pacific Ocean by using high-throughput sequencing. Thaumarchaeota was dominant in the sediments of all sample stations. Community diversity and species richness were greatest at stations near the top of the seamount, and lowest at the deepest station. One sample station on the steep southeast slope that faced the Yap-Mariana trench had a unique composition of Archaea. In summary, depth has an important influence on archaeal community structure, and the geographic properties and sediment characteristics may explain the unique distribution patterns of Archaea in this seamount. This study provides a foundation for future research on Archaea in seamounts. | ZHANG Wenyan LIU Jia DONG Yi LI Xuegang XU Cong XIAO Tian PAN Hongmiao WU Long-Fei | 2019 | Journal of Oceanology and Limnology2019，37，4： | 1 |
| 8 | Synthesis and Photocatalytic Activity of Fe-doped TiO_2 Supported on Hollow Glass Microbeads显示文摘In this paper, Fe-doped TiO_2 photocatalyst supported on hollow glass microbeads(Fe-TiO_2 /beads)is prepared by dip-coating method, which uses hollow glass microbeads as the carriers and tetrabutylorthotitanate [Ti(OC_4H_9)_4] as the raw material. The phase structure, ingredient, morphologies, particle size and shell thickness of the products are characterized by X-ray powder diffraction(XRD), energy-dispersive spectroscopy(EDS) and field emission scanning electron microscope(FESEM). The feasibility of photocatylic degradation of Rhodamine B(Rh B) under illumination of UV-vis light is studied. The results show that the core-shell structure catalyst is composed of Fe-doped anatase TiO_2 and hollow glass microbeads, and the catalytic activity of the TiO_2 is markedly enhanced by Fe ion doping. The optimum concentration of Fe ion is 0.1%(molecular fraction) in the precursor and the photocatalytic activity can be increased to 98% compared with that of the undoped one. The presence of ferrum elements neither influences the transformation of anatase to rutile, nor creates new crystal phases. The possible mechanism of photocatalytic oxidation is also discussed. | Wenyan Zhao Wuyou Fu Haibin Yang Chuanjin Tian Minghui Li Juan Ding Wei Zhang Xiaoming Zhou Hui Zhao Yixing Li | 2011 | Nano-Micro Letters2011，3，1： | 1 |
| 9 | Combined cell grafting and VPA administration facilitates neural repair through axonal regeneration and synaptogenesis in traumatic brain injury显示文摘Neuronal regeneration and functional recovery are severely compromised following traumatic brain injury(TBl).Treatment options,including cell transplantation and drug therapy,have been shown to benefit TBl,although the underlying mechanisms remain elusive.In this study,neural stem cells(NSCs)are transplanted into TBl-challenged mice,together with olfactory ensheathing cells(OECs)or followed by valproic acid(VPA)treatment.Both OEC grafting and VPA treatment facilitate the differentiation of NSCs into neurons(including endogenous and exogenous neurons)and significantly attenuate neurological functional defects in TBl mice.Combination of NSCs with OECs or VPA administration leads to overt improvement in axonal regeneration,synaptogenesis,and synaptic plasticity in the cerebral cortex in TBl-challenged mice,as shown by retrograde corticospinal tract tracing,electron microscopy,growth-associated protein 43(GAP43),and synaptophysin(SYN)analyses.However,these beneficial effects of VPA are reversed by local delivery of N-methyl-D-aspartate(NMDA)into tissues surrounding the injury epicenter in the cerebral cortex,accompanied by a pronounced drop in axons and synapses in the brain.Our findings reveal that increased axonal regeneration and synaptogenesis evoked by cell grafting and VPA fosters neural repair in a murine model of TBl.Moreover,VPA-induced neuroprotective roles are antagonized by exogenous NMDA administration and its concomitant decrease in the number of neurons of local brain,indicating that increased neurons induced by VPA treatment mediate axonal regeneration and synaptogenesis in mice after TBl operation.Collectively,this study provides new insights into NSC transplantation therapy for TBI. | Sujuan Liu Haili Tian Yanmei Niu Chunxia Yu Lingjian Xie Zhe Jin Wenyan Niu Jun Ren Li Fu Zhi Yao | 2022 | Acta Biochimica et Biophysica Sinica2022，54，9： | 1 |
| 10 | Botany, traditional uses, phytochemistry and pharmacology of Apocynum venetum L. (Luobuma): A review显示文摘 | Wenyan Xie Xiaoying Zhang Tian Wang Jianjun Hu | 2012 | Journal of Ethnopharmacology2012，，1： | 1 |
| 11 | IFN-β1b induces OAS3 to inhibit EV71 via IFN-β1b/JAK/STAT1 pathway显示文摘Enterovirus 71(EV71) caused hand, foot and mouth disease(HFMD) is a serious threat to the health of young children. Although type I interferon(IFN-I) has been proven to control EV71 replication, the key downstream IFNstimulated gene(ISG) remains to be clarified and investigated. Recently, we found that 2’-5’-oligoadenylate synthetases 3(OAS3), as one of ISG of IFN-β1b, was antagonized by EV71 3C protein. Here, we confirm that OAS3is the major determinant of IFN-β1b-mediated EV71 inhibition, which depends on the downstream constitutive RNase L activation. 2’-5’-oligoadenylate(2-5A) synthesis activity deficient mutations of OAS3 D816A, D818A,D888A, and K950A lost resistance to EV71 because they could not activate downstream RNase L. Further investigation proved that EV71 infection induced OAS3 but not RNase L expression by IFN pathway. Mechanically, EV71 or IFN-β1b-induced phosphorylation of STAT1, but not STAT3, initiated the transcription of OAS3 by directly binding to the OAS3 promoter. Our works elucidate the immune regulatory mechanism of the host OAS3/RNase L system against EV71 replication. | Baisong Zheng Xiaolei Zhou Li Tian Jian Wang Wenyan Zhang | 2022 | Virologica Sinica2022，37，5： | 1 |
| 12 | Cellular and molecular imaging for stem cell tracking in neurological diseases显示文摘Stem cells(SCs)are cells with strong proliferation ability,multilineage differentiation potential and self-renewal capacity.SC transplantation represents an important therapeutic advancement for the treatment strategy of neurological diseases,both in the preclinical experimental and clinical settings.Innovative and breakthrough SC labelling and tracking technologies are widely used to monitor the distribution and viability of transplanted cells non-invasively and longitudinally.Here we summarised the research progress of the main tracers,labelling methods and imaging technologies involved in current SC tracking technologies for various neurological diseases.Finally,the applications,challenges and unresolved problems of current SC tracing technologies were discussed. | Xiaoxia Yang De-Cai Tian Wenyan He Wei Lv Junwan Fan Haowen Li Wei-Na Jin Xia Meng | 2021 | Stroke & Vascular Neurology2021，6，1： | 1 |
| 13 | Programming of Regulatory T Cells In Situ for Nerve Regeneration and Long-Term Patency of Vascular Grafts显示文摘Rapid integration into the host tissue is critical for long-term patency after small diameter tissue engineering vascular grafts(sdTEVGs)transplantation.Neural recognition may be required for host integration and functionalization of the graft.However,immune rejection and inflammation hinder nerve regeneration of sdTEVGs.Here,a CRISPR/dCas9-nanocarrier was used for targeted programming of regulatory T cells(Treg cells)in situ to promote nerve regeneration of sdTEVGs by preventing excessive inflammation.Treg cells and(C-C chemokine receptor)CCR2+macrophage recruitment occurred after transplantation.The nanodelivery system upregulated ten eleven translocation(TET2)in Treg cells in vitro.Reprogrammed Treg cells upregulated anti-inflammatory cytokines and decreased the proportion of CCR2+macrophages.IL-6 concentrations decreased to the levels required for nerve regeneration.Implantation of CRISPR/dCas9 nanodelivery system-modified sdTEVGs in rats resulted in Treg cell editing,control of excessive inflammation,and promoted nerve regeneration.After 3 months,nerve regeneration was similar to that observed in normal blood vessels;good immune homeostasis,consistency of hemodynamics,and matrix regeneration were observed.Neural recognition promotes further integration of the graft into the host,with unobstructed blood vessels without intimal hyperplasia.Our findings provide new insights into vascular implant functionalization by the host. | Yanhong Wang Fangchao Xue Yanzhao Li Lin Lin Yeqin Wang Shanlan Zhao Xingli Zhao Yong Liu Ju Tan Gang Li Haoran Xiao Juan Yan Hao Tian Min Liu Qiao Zhang Zhaojing Ba Lang He Wenyan Zhao Chuhong Zhu Wen Zeng | 2022 | Research2022，，4： | 1 |
| 14 | The role of exercise in rehabilitation of discharged COVID-19 patients显示文摘Severe acute respiratory syndrome coronavirus 2(SARS-CoV-2)mainly caused pneumonia and pulmonary fibrosis through upper respiratory tract infection,which resulted in acute respiratory distress syndrome(ARDS)and multiorgan damage of cardiovascular,nervous,digestive,and genitourinary systems.Although the virus test turned negative after the patient recovered,the damage to multiorgan caused by SARS-CoV-2 may irreversible.Therefore,the health status of the recovered patients has gradually become the focus of people's attention.Whether coronavirus disease 2019(COVID-19)patients can receive exercise rehabilitation training after discharge?and what's the basis?We try to analyze and answer these questions,will provide some ideas about the patients to develop a reasonable and effective exercise rehabilitation program. | Wenyan Bo Yue Xi Zhenjun Tian | 2021 | Sports Medicine and Health Science2021，3，4： | 1 |
| 15 | EV713C protease cleaves host anti-viral factor OAS3 and enhances virus replication显示文摘The global spread of enteroviruses(EVs)has become more frequent,severe and life-threatening.Intereron(IFN)I has been proved to control EVs by regulating IFN-stimulated genes(ISG)expression.20-50-oligoadenylate synthetases 3(OAS3)is an important ISG in the OAS/RNase L antiviral system.The relationship between OAS3 and EVs is still unclear.Here,we reveal that OAS3,superior to OAS1 and OAS2,significantly inhibited EV71 replication in vitro.However,EV71 utilized autologous 3C protease(3C^(pro))to cleave intracellular OAS3 and enhance viral replication.Rupintrivir,a human rhinovirus 3C protease inhibitor,completely abolished the cleavage of EV713C^(pro)on OAS3.And the proteolytically deficient mutants H40G,E71A,and C147G of EV713C^(pro)also lost the ability of OAS3 cleavage.Mechanistically,the Q982-G983 motif in C-terminal of OAS3 was identified as a crucial 3C^(pro)cutting site.Further investigation indicated that OAS3 inhibited not only EV71 but also Coxsackievirus B3(CVB3),Coxsackievirus A16(CA16),Enterovirus D68(EVD68),and Coxsackievirus A6(CA6)subtypes.Notably,unlike other four subtypes,CA163C^(pro)could not cleave OAS3.Two key amino acids variation Ile36 and Val86 in CA163C^(pro)might result in weak and delayed virus replication of CA16 because of failure of OAS and 3AB cleavage.Our works elucidate the broad anti-EVs function of OAS3,and illuminate a novel mechanism by which EV71 use 3C^(pro)to escape the antiviral effect of OAS3.These findings can be an important entry point for developing novel therapeutic strategies for multiple EVs infection. | Xiaolei Zhou Li Tian Jian Wang Baisong Zheng Wenyan Zhang | 2022 | Virologica Sinica2022，37，3： | 1 |
| 16 | Up-regulation of Thioredoxin 1 by aerobic exercise training attenuates endoplasmic reticulum stress and cardiomyocyte apoptosis following myocardial infarction显示文摘Exercise training(ET)has been reported to reduce oxidative stress and endoplasmic reticulum(ER)stress in the heart following myocardial infarction(MI).Thioredoxin 1(Trx1)plays a protective role in the infarcted heart.However,whether Trx1 regulates ER stress of the infarcted heart and participates in ET-induced cardiac protective effects are still not well known.In this work,H9c2 cells were treated with hydrogen peroxide(H_(2)O_(2))and recombinant human Trx1 protein(TXN),meanwhile,adult male C57B6L mice were used to establish the MI model,and subjected to a six-week aerobic exercise training(AET)with or without the injection of Trx1 inhibitor,PX-12.Results showed that H_(2)O_(2)significantly increased reactive oxygen species(ROS)level and the expression of TXNIP,CHOP and cleaved caspase12,induced cell apoptosis;TXN intervention reduced ROS level and the expression of CHOP and cleaved caspase12,and inhibited cell apoptosis in H_(2)O_(2)-treated H9c2 cells.Furthermore,AET up-regulated endogenous Trx1 protein expression and down-regulated TXNIP expression,restored ROS level and the expression of ER stress-related proteins,inhibited cell apoptosis as well as improved cardiac fibrosis and heart function in mice after MI.PX-12 partly inhibited the AET-induced beneficial effects in the infarcted heart.This study demonstrates that Trx1 attenuates ER stress-induced cell apoptosis,and AET reduces MI-induced ROS overproduction,ER stress and cell apoptosis partly through up-regulating of Trx1 expression in mice with MI. | Mengxin Cai Zujie Xu Wenyan Bo Fangnan Wu Wenpu Qi Zhenjun Tian | 2020 | Sports Medicine and Health Science2020，2，3： | 1 |
| 17 | Distinct viral determinants for the packaging of human cytidine deaminases APOBEC3G and APOBEC3C显示文摘 | Tao Wang Wenyan Zhang Chunjuan Tian Bindong Liu Yunkai Yu Lingmei Ding Paul Spearman Xiao-Fang Yu | 2008 | Virology2008，，1： | 1 |
| 18 | Sustainable wood-based nanotechnologies for photocatalytic degradation of organic contaminants in aquatic environment显示文摘Wood-based nanotechnologies have received much attention in the area of photocatalytic degradation of organic contaminants in aquatic environment in recent years,because of the high abundance and renewability of wood as well as the high reaction activity and unique structural features of these materials.Herein,we present a comprehensive review of the current research activities centering on the development of wood-based nanocatalysts for photodegradation of organic pollutants.This review begins with a brief introduction of the development of photocatalysts and hierarchical structure of wood.The review then focuses on strategies of designing novel photocatalysts based on wood or its recombinants(such as 1D fiber,2D films and 3D porous gels)using advanced nanotechnology including sol-gel method,hydrothermal method,magnetron sputtering method,dipping method and so on.Next,we highlight typical approaches that improve the photocatalytic property,including metal element doping,morphology control and semiconductor coupling.Also,the structure-activity relationship of photocatalysts is emphasized.Finally,a brief summary and prospect of wood-derived photocatalysts is provided. | Xinyi Liu Caichao Wan Xianjun Li Song Wei Luyu Zhang Wenyan Tian Ken-Tye Yong Yiqiang Wu Jian Li | 2021 | Frontiers of Environmental Science & Engineering2021，15，4： | 0 |
| 19 | LaNiO3 modified with Ag nanoparticles as an efficient bifunctional electrocatalyst for rechargeable zinc-air batteries显示文摘No-precious bifunctional catalysts with high electrochemical activities and stability were crucial to properties of rechargeable zinc-air batteries.Herein,LaNiO3 modified with Ag nanoparticles(Ag/LaNiO3)was prepared by the co-synthesis method and evaluated as the bifunctional oxygen catalyst for oxygen reduction reaction(ORR)and oxygen evolution reaction(OER).Compared with LaNiO3,Ag/LaNiO3 demonstrated the enhanced catalytic activity towards ORR/OER as well as higher limited current density and lower onset potential.Moreover,the potential gap between ORR potential(at-3 mA cm^-2)and OER potential(at 5 mA cm^-2)was 1.16 V.The maximum power density of the primary zinc-air battery with Ag/LaNiO3 catalyst achieved 60 mW cm^-2.Furthermore,rechargeable zinc-air batteries operated reversible charge-discharge cycles for 150 cycles without noticeable performance deterioration,which showed its excellent bifunctional activity and cycling stability as oxygen electrocatalyst for rechargeable zinc-air batteries.These results indicated that Ag/LaNiO3 prepared by the co-synthesis method was a promising bifunctional catalyst for rechargeable zinc-air batteries. | Pengzhang Chuanjin TIAN Wei YANG Wenyan ZHAO Zhe LU | 2019 | Frontiers of Materials Science2019，13，3： | 0 |
| 20 | Hydrothermal growth of symmetrical ZnO nanorod arrays on nanosheets for gas sensing applications显示文摘 | Wenyan ZHAO Chuanjin TIAN Zhipeng XIE Changan WANG Wuyou FU Haibin YANG | 2017 | Frontiers of Materials Science2017，11，3： | 0 |
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